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protein shock相关的网络例句

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与 protein shock 相关的网络例句 [注:此内容来源于网络,仅供参考]

The content of soluble protein rapidly increased with the embryo development after pollination for 16 days, while BiP markedly increased from 22nd day after pollination. The BiP failed to respond to heat shock. Immunohistochemical localization showed that BiP localized in plumule, primary vascular tissue and aleurone layer during embryo development.

组织化学免疫定位表明,在玉米胚发育的不同时期,BiP主要定位在胚芽端、初生维管组织和糊粉层中,提示胚在构建器官的同时,也为其功能执行准备了条件;热激不影响其定位。

One hand mechanical obstruct led to the increase of veinous resistance and the obstacle of microcirculation, the other hand the adhesive PMN was activated in excess, the white blood cells released a lot of enzymes, in which PMN-elastase can decompose the components of cell and many albumens, inclusive of immunoglobulin、alexin and fibrication. These components induced the injury of the pancreatic capillary vessels and cell and lysosome enzy made the tissue protein hydrolyze and produced unsaturated fatty acids, which destroyed the structure and function of cellar membrane. The inflammatory cellar factors activate other immunocytes to produce the injury and necrosis of tissue, which aggravated the pathological injury and led to shock、pyaemia and MODS. So ICAM-1 and LFA-1 played an important role in SAP. Frossard found that the expression of ICAM-1 in the rat model, especially in serum、pancreas and lung. All these showed ICAM-1 is an important factor in AP and concomitant lung injury.

胰腺小叶组织局部血管EC首先被激活,ICAM-1表达升高,与被激活的PMN表面表达的LFA-1相结合,"PMN-EC"相互作用加剧,一方面机械性阻塞毛细血管导致静脉阻力增加、微循环障碍;另一方面粘附的PMN过度吞噬或激活,当白细胞吞噬的颗粒不能被封闭隔离,连同细胞内的酶被释放出来,其中的PMN-elastase能够降解细胞基质中各种成分,水解多种蛋白,加重胰腺的毛细血管内皮细胞和腺泡的损伤;释放的溶酶体酶使组织蛋白水解,产生的不饱和脂肪酸引发脂质过氧化方应,破坏细胞膜的结构和功能;释放的炎性细胞因子,激发其他的免疫细胞的功能,导致进一步的组织损伤和坏死,加重SAP的病理损伤,最终导致休克、脓毒血症及多器官功能障碍等严重后果。

Cloning, expression and antigenic analysis of heat shock protein A gene of human Helicobacter pylori.

人幽门螺杆菌热休克蛋白A编码基因的克隆、表达及抗原性研究。

METHODS: The skinned first class arborization of superior mesenteric artery from HS rats were adopted to observe the influence of inhibitor of ZIPK on the effects of PKCα and PKCε agonists on calcium sensitivity after shock via measuring the contraction initiated by Ca2+ with isolated organ perfusion system, hypoxic vascualr smooth muscle cells were adopted to measure the protein expression and activity of ZIPK after applying PKCα and PKCε agonists following hypoxia via Western blotting.

采用离体微血管环张力测定技术,观察褪膜后抑制ZIPK对PKCα、PKCε激动剂增高失血性休克肠系膜上动脉一级分支微血管钙敏感性作用的影响;同时观察缺氧血管平滑肌细胞在PKCα、PKCε激动剂作用下ZIPK蛋白表达、活性的变化。

Alcohol ; astrocyte ; heat shock protein 70

酒精;星型胶质细胞;热休克蛋白70

Cold adaptation and cold shock protein can be determined by radiolabelling and autoradiography.

对该菌冷激适应性生理特性的研究中,发现了一个分子量约为7000Da的冷激蛋白。

HSP90 as a important member of HSP protein family, are usually constitutively expressed in most mammalian cell types and can be further induced by heat shock and other stresses.

HSP90是热激蛋白家族的主要成员之一,它在细胞中呈高组成性表达,热激和其它应激条件可进一步诱导其表达。

HSP90 is an important member of HSP protein family. HSP90 usually constitutively expressed in most mammalian cell types and can be further induced by heat shock and other stresses.

HSP90是热激蛋白家族的主要成员之一,它在细胞中呈高组成性表达,热激和其他应激条件可以进一步诱导其表达。

The results showed that under the cold stimulation, the types of blood protein both changeed, cold shock proteins was generated. Since the different genes which control CSPs need different intensity and degree of stimulation, the same specie of Loaches had the differences in the time and new protein generation under the different temperature treatment. Since two types of Loaches have different adaptability, the time and type of CSPs generation are also different under the same temperature treatment.

结果表明,泥鳅和大鳞副泥鳅的血液蛋白质发生了变化,即产生了冷激蛋白;同种泥鳅,控制产生冷激蛋白的特定基因在一定的刺激强度和刺激程度下,产生冷激蛋白的时间和种类不同;不同种泥鳅,在相同低温处理条件下,产生冷激蛋白的时间和种类也不一样。

BACKGROUND: It is proved that protein of heat shock protein 70 family has protective effects on cells.

背景:研究证实,热休克蛋白70家族的蛋白质对细胞有保护作用。

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