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protein shock相关的网络例句

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与 protein shock 相关的网络例句 [注:此内容来源于网络,仅供参考]

Objective To study the effects and significance of Bax protein and heat shock protein 70 (HSP70) in prevention of insulitis and diabetes by complete Freund's adjuvant in female NOD mice.

目的 研究Bax蛋白和热休克蛋白70在完全弗氏佐剂预防NOD鼠胰岛炎和糖尿病中作用及意义。

The cold shock protein thermophile Bacillus caldolyticus and its homologous protein mesophile Bacillus subtilis are very similar in sequence and structure but they have great difference on thermostability.

本论文主要对冷休克蛋白Bc-Csp的热稳定性进行了研究。

Objective To study the effects of heat-shock protein fusion protein on the differentiation and maturation of human monocyte derived dendritic cells.

目的:研究热休克蛋白融合蛋白对人树突状细胞分化成熟的刺激作用。

Heat shock protein 70 (HSP70) is a sort of unspecific protein which can be induced by acupuncture and moxibustion.

热休克蛋白 70 (HSP70)是一种非特异性细胞保护蛋白,针灸可诱导其产生。

Twenty-seven differential proteins were tested by MALDI-TOF-MS, and 16 were identified. Of the 16 proteins, 13 were up-regulated in patients with HCC, including heat-shock protein 27, member RAS oncogene family (RAB14), actin, alpha 1 antitrypsin, RNA-binding protein regulatory subunit and so on. By contrast, the levels of proteins such as PDX6, HSPA8 and MnSOD were significantly reduced in HCC patients.

对27个差异蛋白质点进行了质谱分析,鉴定出16个蛋白质,在HBV相关性HCC患者PBMC中表达明显增强的有热休克蛋白质27、ras癌基因家族(RAB14)、肌动蛋白、α1-抗胰蛋白及RNA结合蛋白调节亚基等13个蛋白点;而PDX6、HSPA8及锰超氧化物歧化酶在HCC患者PBMC中低表达。

This suggests that antibody or inhibition of cGMP-dependent protein kinase may prevent vascular hyperpermeability of severity burn shock and septic shock,and fundamentally take on a new look in treatment of shocks instead of traditional and passive clysis.

提示运用PKG抗体或抑制剂有可能防治大面积烧伤和中毒性休克时的血管通透性增加导致的血容量减少的发生,从根本上改观传统的、被动的烧伤补液抗休克方法。

Recent study indicates that heat shock protein, a protective protein, could also be used as the marker of overtraining.

最新研究显示,热休克蛋白作为一种应激保护性蛋白,也是监控过度训练的有效指标。

Heat shock factor 1; Heat shock protein ; Gene knockout ; Mouse ; Reproduction

热休克因子1;热休克蛋白;基因剔除;小鼠;生殖

Methods: Twenty-four male SD rats were randomly divided into normal control (NC, n=6), protein C activator (PCA, n=6), lipopolysaccharide (LPS, n=6) and LPS+PCA group (n=6). NC group were given normal saline and PCA group were injected protein C activator (0.4 mg/kg) through vein of the tail. LPS group were administered lipopolysaccharide (10 mg/kg, 4h) by intraperitoneal injection method to make the model of septic shock rats. After the animal model was made, LPS+PCA group were injected protein C activator (0.4 mg/kg) through vein of the tail. 30 minutes after the injection, each rat was tested by Medlab biosignal analyzing system for observing mean arterial pressure and left ventricle function, the activity of LDH and iNOS in myocardium and the cuttings of myocardial tissues.

取SD雄性大鼠24只随机分成对照组、PCA组、LPS组和LPS+PCA组四组,对照组尾静脉注射生理盐水,PCA组尾静脉注射蝮蛇毒蛋白C激活物,LPS组腹腔注射LPS (10 mg/kg, 4h)的方法建立败血症休克的实验动物模型,在建立败血症的实验动物模型的基础上,尾静脉注射PCA (0.4 mg/kg)为LPS+PCA组,每只大鼠均于药后30 min时运用Medlab生物信号采集处理系统记录平均动脉压和左心室功能指标,并测定心肌中乳酸脱氢酶活性、诱导性一氧化氮合酶活性及心肌病理切片。

Aim To assess the effect of heat shock factor-1 (HSF1) and heat shock protein 70 (HSP70) to tachyphylaxis by bradykinin in the opening blood-tumor barrier.

目的 探讨缓激肽(bradykinin, BK)开放血瘤屏障过程中,HSF1和HSP70对BK诱发快速耐受性的影响。

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