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proliferation相关的网络例句

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与 proliferation 相关的网络例句 [注:此内容来源于网络,仅供参考]

Objective To evaluate the relationship between growth rate and cell proliferation index of acoustic neuroma.

听神经瘤是Schwann细胞沿前庭神经过度增殖的结果,其生长速率差别很大。

Point reactor dynamic equation is a simple equation that not considered reactor space factor, it was used to calculate the dynamic proliferation of reactors because there is no space variable , otherwise it can be used to calculate coupled with the thermal feedback to get the number of neutron density changes situation.

点堆动力学方程是不考虑反应堆空间特性的简单方程,由于没有空间变量所以它通常被用来计算反应堆的动态增殖特性,而且可以用来与热工耦合计算有反馈的中子数密度变化情况。

Objective To observe the effects of nicotine on the proliferation of microglia.

目的 观察尼古丁对小胶质细胞增殖的影响。

Selling human waste products as fertilizers became much less common after World War II, both for sanitary reasons and because of the proliferation of chemical fertilizers, and less than 1% is now used for night soil fertilization.

二战以后,将人类的排泄物作为肥料售卖变得很少见,出于卫生的考虑及化肥的普及,直至现在,只有不到1%的人仍在用农家肥。

Results (1)ATP could inhibit the proliferation of U937 cells with inhibition rate over 43% after 48hour ATP treatment;(2)ATP treated U937 cells numbers increased obviously in G1 phase,and apoptosis peak appeared before G1 phase,apoptotic cell number was 3.4% for 24hour,and was 22.7% for 48hour of ATP treated U937 cells;(3) Nuclear chromatin condensed into sperical masses bound cytoplasma membrane formed apoptotic bodies which is shed from membrane surface into intercellular medium;(4) Apoptotic bodies were nigrosine staining negtive.

结果 (1)ATP对U937细胞的增殖有按摩明显的阻抑作用,加药48h后增殖的抑制率可达43%以上;(2)ATP处理的U937细胞周期发生改变,G1期细胞数按摩明显增多,ATP作用24h时G1期前出现亚二倍体峰——凋亡峰,凋亡细胞数为3.4%,作用48h时凋亡细胞数增多为22.7%;(3)ATP处理的U937细胞首先在核内染色质浓缩成半月形贴近核膜,逐渐向核膜外移动,进入胞浆内再移向质膜内外面,紧贴质膜外面再逐步脱离细胞体,进入细胞基质中,成为游离的凋亡小体。

Methods Cell culture,flow cytometry,HE staining,Nigrosine staining and electron microscopy were used. Results (1)ATP could inhibit the proliferation of U937 cells with inhibition rate over 43% after 48hour ATP treatment;(2)ATP treated U937 cells numbers increased obviously in G1 phase,and apoptosis peak appeared before G1 phase,apoptotic cell number was 3.4% for 24hour,and was 22.7% for 48hour of ATP treated U937 cells;(3) Nuclear chromatin condensed into sperical masses bound cytoplasma membrane formed apoptotic bodies which is shed from membrane surface into intercellular medium;(4) Apoptotic bodies were nigrosine staining negtive.

结果 (1)ATP对U937细胞的增殖有明显的阻抑作用,加药48h后增殖的抑制率可达43%以上;(2)ATP处理的U937细胞周期发生改变,G1期细胞数明显增多,ATP作用24h时G1期前出现亚二倍体峰——凋亡峰,凋亡细胞数为3.4%,作用48h时凋亡细胞数增多为22.7%;(3)ATP处理的U937细胞首先在核内染色质浓缩成半月形贴近核膜,逐渐向核膜外移动,进入胞浆内再移向质膜内外面,紧贴质膜外面再逐步脱离细胞体,进入细胞基质中,成为游离的凋亡小体。

Methods: Cell proliferation was measured by crystal voilet staining assay. The activity of lactic acid dehydrogenase in the upper layer of culture solution was tested to examine the damage to the cell membrane.3H-TdR was used to study the effect of porphyrin nitrogen mustard on the synthesis of DNA.

用结晶紫染色法测定卟啉氮芥对人肝癌细胞SMMC7721的光动力抑制效应,用测定培养上清乳酸脱氢酶活性方法研究光动力作用对人肝癌细胞SMMC7721细胞膜的作用,用3H-TdR掺入法研究光动力作用对人肝癌细胞SMMC7721DNA合成的影响。

Objective:This work is to investigate the effects of the proliferation and apoptosis of K562 cell affected by Dendrobium nobile polysaccharidesand and explore the possible mechanisms,in order to provide experimental evidence to further research pharmacological activity of Dendrobium nobile polysaccharides.

中文摘要目的:研究金钗石斛多糖对慢性粒细胞白血病K562细胞增殖和凋亡的影响及其可能的机制,为金钗石斛多糖药理作用的进一步研究提供实验依据。

2Its one of cytology mechanisms of Qiang-gu-bao decoction on preventing and curing diabetes osteoporosis were related with promoting for osteoblastic cell proliferation 、 differentition and mineralization of formation,moreover inhibiting nonenzymic glycation reaction and reducing the formation of AGEs on the early stage, inhibiting the secretion of osseous absorptive cytokine, Consequently, Qiang-gu-bao decoction could inhibited bone resorption and promote for bone formation as well as regulated the bone metabolization.

2强骨宝方直接提取液及糖尿病模型大鼠载药血清均能促进 OB 增殖、分化及矿化成骨作用,并抑制早期糖基化反应,减少 AGEs 的形成,抑制 OB 分泌骨吸收因子,从而达到抑制骨吸收、促进骨形成、调节骨代谢,这可能是强骨宝方防治糖尿病性骨质疏松的细胞学机制之一。

Results In the very elderly, most of the DCs were nonviable at the 7th day after cultured, and no DCs were detected in 8 subjects.The capacity to stimulate proliferation of allogeneic T lymphocytes was also lower.On the other hand, in the elderly,the DCs grew prosperity in all the subjects. At the 7th day after cultured, DCs could be seen in the every subjects.

结果 ≥80岁组DCs培养过程中细胞逐渐死亡,培养至第7天大部分细胞死亡,获得的DCs数量较少,其中有8例未能培养出DCs;较60~70岁组对同种T淋巴细胞的刺激能力也显著下降,而60~70岁组培养细胞的活力保持较好,至第7天均可获得数量较多的DCs。

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