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praxiology相关的网络例句

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与 praxiology 相关的网络例句 [注:此内容来源于网络,仅供参考]

The experiment research indicates: contrasting with thecontrol groups, Bushen Huoxue Huatan Prescription has the following functions: 1. improving the praxiology function of model rats, 2. improving the activity in cholineacetyl-transferase in hippocampus tissue, 3. increasing the expression of NOS in hippocampus region of cortex, 4. promoting the expression of the synapse P3, in hippocampus region of cortex, 5. reducing obviously the apoptosis number of brain nerve cell .

实验研究表明:与对照组比较,补肾活血化痰方具有:1、改善动物行为学功能;2、提高海马组织胆碱乙酰转移酶活性;3、增加皮质及海马区一氧化氮合酶的表达;4、促进皮质海马区突触素蛋白P_(38的表达;5、显著降低脑神经细胞的凋亡数目。

The Effects of Analog 165 of APP 5-mer Peptide on Praxiology, Expression of PSD95 and Shank 1 in Alzheimer Disease Rats

APP5肽类似物165对老年性痴呆模型大鼠行为学及PSD95和Shank 1表达的影响,陆珊;雷亚平;王蓬文;王蓉;盛树力;田金洲-66文免费论文库,大学生论文库

Results:① large dose of piracetam(≧600mg/kg) significantly increased scores of praxiology(p.01)、decreased water content(p.05) and the expression of aquaporin( aqp4) in mcao.②all dose of piracetam decreased volume of ischemic (p.01) and remained relatively intact ultramicrostructure.

结果:吡拉西坦注射液剂量依赖性地降低侧大脑半球的含水量、改善其神经功能评分、下调水通道蛋白4(aop4)的表达、降低动物的脑梗死体积、改善缺血脑组织神经元细胞的超微结构。

Objectives In order to investigate how does King's Brain pill work or mechanism of King's Brain pill effective in treating AD clinically, we observed effects of extract of King's Brain on neuronal and vascular lesions induced by Aβ combining with praxiology and neuropathology.

目的 运用现代分子神经病理学和行为学技术,围绕炎症假说、淀粉样蛋白瀑布流假说,以神经元和脑血管为保护目标,着眼于AD的关键病理机制Aβ(β淀粉样蛋白沉积和淀粉样血管病变)及其代谢,开展中药补肾活血化痰复方制剂金思维对损伤的神经元和血管的保护作用机理研究,从而探讨其防治老年性痴呆的机理。

Methods OA was injected into hippocampal CA1 region of rats. We observed rat praxiology changes by Morris water maze and the senile plaques and neurobrillary tangles of hippocampus by Bielschowsky stain and the expression of β-amyloid protein and neuronal nitric oxide synthase of the rats hippocampus by immunohistochemistry.

在大鼠海马CA1区多次微量注射OA,水迷宫实验观测大鼠行为学改变;Bielschowsky染色观察海马CA1区神经原纤维缠结和老年斑等特征性病理变化;免疫组化方法观察海马CA1区Aβ1-40和nNOS的表达。

Objective To study the statement of immunoreaction of c Jun and functional recovery of spinal cord injury in rats after fetal spinal cord transplantation.Methods Experimental rats were divided into two groups:spinal cord hemisection injury combination with transplants of FSC to injured site group,and spinal cord injury that received gelfoam pledgets group.On the lst,3th,7th,14th and 28th day after operation,6 animals in each group at each time phase,were observed for their function recovery with the methods of praxiology and electro physiological examination,the animals were then sacrificed for the observation of the statement of c Jun reaction by immnochemistry method and computer image analysis system was used for quantitative analysis.

目的:研究大鼠胚胎脊髓移植后能否影响,c-Hun免疫反应的表达和脊髓损伤后大鼠功能的恢复,方法;将动物分为脊髓半切洞损伤加胚胎脊髓移植组和单纯脊髓板切洞损伤加明胶海绵填塞组,每个时相点每组6只动物,术只1,3,7,14和28天,应用行为学和电生理检查观察大鼠功能恢复情况,应用免疫细胞化学方法,c-Jun免疫反应的表达,采用计算机图像分析技术,进行定量分析。

Object: The project is on the basis of chronic alcoholic myopathy model of rat, to dynamically observe the change of NF-κB and MMP-2, MMP-9 in muscle cell, observe the function in the mechanism of alcohol myopathy, move forward a single step to explain the pathogenesy of this disease from gene and protein leve. We also provide a helpful platform for other metabolic myopathy. Methods:(1) 80 health, male, Sprague-Dawley rats(weight 180~200g), were chosen to sep up the CAM rat myopathy model ,to observe the rat bodyweight, the change of praxiology and the patho-change of muscle tissue.

目的:本实验旨在建立大鼠慢性酒精中毒性肌病模型的基础上,动态观察NF-κB及MMP-2、MMP-9在酒精中毒性肌病发展中的表达、变化情况,探讨其在慢性酒精中毒性肌病发病过程中的作用,从基因和蛋白水平进一步阐述该病发病机制,为慢性酒精中毒性肌病的临床防治提供理论依据,同时对于其他代谢性肌病的研究也提供一个平台。

Open-field test,step-down avoidance test and immunohistochemstry staining were employedto study the effects of the extract of the fruit of Hyperieum perforatum on praxiology and cerebral pathology in the rat models.

制作大鼠慢性应激抑郁模型,应用敞箱法、跳台法、免疫组织化学染色研究贯叶连翘提取物对抑郁大鼠行为学、脑病理学的影响。

Guinea pig sensitied by ovalbumin for model made and challenged with substance P were second group,and the last two groups were with steroid treatment and contral. Praxiology markers、 IgE、 histamine and SP were detected .Gene chip of rat were used in observing genes expression change.

再经卵蛋白致敏制作变应性鼻炎豚鼠模型,并按模型组,P物质干预组及类固醇激素治疗组及对照组分为四组,观察行为学指标,血IgE,组胺及SP含量的变化,通过鼠芯片观察基因表达谱及其变化。

The aim is to use rat model of formalin result to pain to observe rat praxiology responses after giving ASA , The results found that: The rats showed spontaneous pain-related responses after formalin test. After being injected formalin into one hindpaw, the rats of F group immediately showed intense pain-related response: flinching / shaking and licking / biting injected paws.

在福尔马林炎性痛动物模型测定给药后大鼠的痛行为反应,发现ASA可以明显抑制F引起的双相痛反应,故从行为学的水平确定了ASA有显著的镇痛效应。

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