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plaques相关的网络例句

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与 plaques 相关的网络例句 [注:此内容来源于网络,仅供参考]

These include studies of enzymes that act on proteins to produce the plaques, and using antibodies against amyloid.

这包括对作用于蛋白质产生plaques的酶的研究和运抗体抵抗amyloid。

The major component of the neuritic plaques it was called as senile plaques in the pa...

AD的典型病理改变中的神经炎性斑和淀粉样血管病,其主要成分都是Aβ,Aβ是淀粉样蛋白前体通过某种降解途径产生的,由39-42个氨基酸残基组成,其活性片断主要在第25-35个氨基酸残基。

Plaques of these virus strains were obviously different in size and shape. The plaques of A/Vietnam/1194/2004 (H5N1) were mainly large in size and round in shape, except for a few smaller plaques appearing punctiform in shape. While A/Beijing/01/03(H5N1) were dominated by plaques of moderate size, also mixed with a few punctiform plaques.

结果 野生的人源H5N1禽流感病毒株在空斑形成上都存在不同程度的不均一性,空斑大小、形状差异明显:A/Vietnam/1194/2004(H5N1)以大圆形斑为主,夹杂少量小点状斑。A/Beijing/01/03(H5N1)大多数为中等大小空斑,并有少数针尖状斑。

(1)The levels of MCP-1 and VEGF'levels was significant increased in CHD patients,Compared with controls, It was implicated that they might take part in the actions of informing atherosderotic plaques;(2)The serum level of MCP-1 and VEGF were elavated step by stey among the group of controls、SAP、UAP、AMI, It was thought they might develop the matter on the stability of atherosderotic plaques;(3)The serum level of MCP-1 and VEGF were positive correlated with 7-TIMI risk score, It prompt that MCP-1 and VEGF was the reactiveness markers of coronary artery process ,and developed the risk to major acute cardiac events.

(1)血清MCP-1、VEGF水平在冠心病患者中显著升高,提示MCP-1、VEGF可能在促进冠状动脉粥样硬化斑块形成的过程中起重要作用;(2)血清MCP-1、VEGF水平在对照组、各冠心病亚组(SAP、UAP、AMI组)之间逐级升高,提示MCP-1、VEGF可能增加了冠状动脉粥样硬化斑块的不稳定性;(3)联合检测MCP-1、VEGF的灵敏度\特异度上升,证明临床上联合检测二者血清指标意义大;并且血清MCP-1、VEGF水平和7-TIMI积分呈正相关性,7-TIMI积分高的患者血清MCP-1、VEGF水平也高,说明它们能作为冠脉病变的活动性标志物,可能成为评价主要心血管恶性事件的危险性的相关指标。

Firstly , the V4 stock was purified three times by plaque formation. It's difficult to see clear plaques in CEF cells due to the avirulent characteristic , so we treated the V4 stock with trypsin and then allantoic Fluid and Mg2+ were added in the layer gel. In this case, we could see small plaques occasionally, but not stably.

本实验首先对实验室保存的V4株进行了三轮的蚀斑纯化,由于V4株属无毒株,其在CEF细胞中很难形成可见的蚀斑,因此我们采用了用胰酶处理V4病毒,并在上层胶中添加尿囊液及Mg2+,这样可以偶尔产生可见的蚀斑,但实验结果重复性很差。

Results (1)CIMT and FIMT in CHD group were significantly higer than those in non-CHD group(p<0.05), The detection rate of plaques in CHD group was significantly higer than those in non-CHD group(p<0.05);(2) The sensitivity of carotid and femoral atherosclerosis plaques in young CHD patients was 62% and 57%. The specificity was 90% and 86%. The positive predictive value of carotid and femoral atherosclerosis plaques in young CHD patients was 92% and 88%. The negative predictive value was 58% and 53%. The predictive value of combinant measurement of carotid and femoral atherosclerosis plaques was not improved.

结果 (1)冠心病组CIMT、FIMT与非冠心病组比较,前者均高于后者(p<0.05);冠心病组颈动脉、股动脉粥样硬化斑块检出率均明显高于非冠心病组(p<0.05);(2)颈动脉、股动脉粥样硬化斑块预测年轻冠心病的敏感性分别为62%、57%,特异性为90%、86%,阳性预测值分别为92%、88%,阴性预测值为58%、53%;颈动脉、股动脉联合检测未见明显提高预测值。

The sprouts from endplate and a few contact sites of NCAM with AChR can be seen.C: Untreated group on the 20th day after L5 nerve root compression. There are some AChR plaques of morphological abnormality, and no contact sites of NCAM with AChR although the intensity and scope of NCAM staining is increased.D: Yiqi Huayu Recipetreated group on the 20th day after L5 nerve root compression. All the AChR sites are reoccupied by NCAM and the morphology of AChR are restored. E: Untreated group on the 30th day after lumbar nerve root compression. AChR plaques display irregularity in shape, with a few round or spindle shaped cupulae. Meanwhile, some NCAM stainings are still shown to be around the AChR. F: Yiqi Huayu Recipetreated group on the 30th day after L5 nerve root compression.

随着时间的推移,AChR斑的形态渐趋规则,NCAM反应也逐步增强,至造模后20 d达到最高峰,但益气化瘀方组仍然明显优于模型组,其不仅在数值上超过了胚胎期大鼠,且与AChR全部重叠;而模型组仍可见到不少重叠不全且形态异常的AChR斑。L5神经根受压后30 d,NCAM的表达水平开始下降,基本局限于终板区内,其下降速度和范围在益气化瘀方组明显大于模型组;模型组不仅NCAM的表达明显高于益气化瘀方组,且仍能见到一些成熟不全的AChR斑;而益气化瘀方组AChR斑形态已接近于正常。

There are some AChR plaques of morphological abnormality, and no contact sites of N-CAM with AChR although the intensity and scope of N-CAM staining is increased.D: Yiqi Huayu Recipe-treated group on the 20th day after L5 nerve root compression. All the AChR sites are reoccupied by N-CAM and the morphology of AChR are restored. E: Untreated group on the 30th day after lumbar nerve root compression. AChR plaques display irregularity in shape, with a few round or spindle shaped cupulae. Meanwhile, some N-CAM stainings are still shown to be around the AChR. F: Yiqi Huayu Recipe-treated group on the 30th day after L5 nerve root compression.

随着时间的推移,AChR斑的形态渐趋规则,N-CAM反应也逐步增强,至造模后20 d达到最高峰,但益气化瘀方组仍然明显优于模型组,其不仅在数值上超过了胚胎期大鼠,且与AChR全部重叠;而模型组仍可见到不少重叠不全且形态异常的AChR斑。L5神经根受压后30 d,N-CAM的表达水平开始下降,基本局限于终板区内,其下降速度和范围在益气化瘀方组明显大于模型组;模型组不仅N-CAM的表达明显高于益气化瘀方组,且仍能见到一些成熟不全的AChR斑;而益气化瘀方组AChR斑形态已接近于正常。

There are some AChR plaques of morphological abnormality, and no contact sites of NCAM with AChR although the intensity and scope of NCAM staining is increased.D: Yiqi Huayu Recipetreated group on the 20th day after L5 nerve root compression. All the AChR sites are reoccupied by NCAM and the morphology of AChR are restored. E: Untreated group on the 30th day after lumbar nerve root compression. AChR plaques display irregularity in shape, with a few round or spindle shaped cupulae. Meanwhile, some NCAM stainings are still shown to be around the AChR. F: Yiqi Huayu Recipetreated group on the 30th day after L5 nerve root compression.

随着时间的推移,AChR斑的形态渐趋规则,NCAM反应也逐步增强,至造模后20 d达到最高峰,但益气化瘀方组仍然明显优于模型组,其不仅在数值上超过了胚胎期大鼠,且与AChR全部重叠;而模型组仍可见到不少重叠不全且形态异常的AChR斑。L5神经根受压后30 d,NCAM的表达水平开始下降,基本局限于终板区内,其下降速度和范围在益气化瘀方组明显大于模型组;模型组不仅NCAM的表达明显高于益气化瘀方组,且仍能见到一些成熟不全的AChR斑;而益气化瘀方组AChR斑形态已接近于正常。

Some say the plaques could be an effect of the disease, not the cause.

一些人认为plaques可能是病症的结果不是原因。

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Do you know, i need you to come back

你知道吗,我需要你回来

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