查询词典 pathway

At present, the late-flowering mutants are fallen into four pathways: photoperiod pathway, vernalization pathway, autonomous pathway and GA pathway according to factors described above.

目前，一般按照对以上因素的反应将晚花突变体归于四条开花调控途径：光周期途径、春化途径、自主途径和GA途径。

In Arabidopsis thaliana, flowering was regulated mainly by photoperiod pathway, vernalization pathway, autonomous pathway, and GA pathway.

在拟南芥中，植物开花途径主要有春化促进途径、光周期促进途径、自主开花途径及依赖于赤霉素的途径等。

When a pathway, such as the Embden-Meyerhof pathway or the TCA cycle are amphibolic pathway is referred to as an amphibolic pathway .

是专业知识，有些是术语，可以直接用英语表示，我把该翻译的英语尽力翻译了，希望对你有帮助哦

When a pathway, such as the Embden-Meyerhof pathway or the TCA cycle are amphibolic pathway is referred to as an amphibolic pathway .Pathways of glycolysis and the TCA cycle are amphibolic pathways because they provide ATP and chemical intermediates to build new cell material .The main metabolic pathways ,and their relationship to biosynthesis of cell material ,are shown in figure 21 below.

当一种方式，譬如Embden-Meyerhof方式或三辛胺周期方式，指一条醣解作用为方式路径，并且三辛胺周期是一种方式，因为它们提供ATP 和化工中间体建立新细胞物质主要新陈代谢的方式，与它们的关系构成细胞材料生物合成，下面介绍表21 。

RESULTS:Amo ng the 80 patients with conceal accessory pathway,no one had episodes of T wave change by RFCA. However 20(39%) patients appeared obvious T wave change among th e 52 patients with non-conceal assessory pathway after RFCA,including the 19 pa tients they passed through cmplete accessory pathway (QRS duration≥140 ms) and only one patient is incomplete accessory pathway (QRS duration ＜140 ms).

结果：80例隐匿性旁路在射频消融术后无1例出现心脏复极改变，52例显性旁路中有20例(占39 %)术后出现明显T波变化，其中完全性旁路(QRS≥0.14 s)占19例，而不完全性旁路(QRS＜0.14 s)仅占1例。

The fEPSP response evoked in one pathway was small, usually 20–30% of the maximu, whereas a large response, usually 80–90% of the maximum, was evoked in the strong pathway. After recording of the baseline fEPSP evoked at 0.033 Hz, LTP of the weak pathway could be associatively induced by paired stimulation of the weak and strong pathways 100 times at 6 sec intervals, with stimulation of the weak pathway preceded 3–10 msec.

在记录以0.033Hz所引发基础的兴奋性突触后电位之后，在弱的路径上可以因配合弱与强的刺激路径而引发出相关性长期增益（每六秒一次共100次，弱的刺激路径在强的刺激路径之前 3-10 ms），然而当在两个路径之间的配合时间差大於10 ms时即无法形成相关性长期增益，此类型长期增益的引发是需要依赖活化NMDA接受器的，因为给予50 mM的D，L-AP5可以阻断相关性长期增益的引发（兴奋性突触后电位为原来的105±6﹪；n=6）。

Results are as followed:1 Exposure of HELF cells to BP caused c-Jun activation,and increased the activity of MAPK,PI-3K,p53 and cyclin D1 pathway.2 BP-induced c-Jun activation was inhibited by dominant negative mutants of extracellular signal-regulated protein kinase or c-Jun NH_2-terminal kinase,but not by p38,impling that JNK and ERK pathways medicate c-Jun activation induced by BP.3 Overexpression of dominant-negative mutants PI-3K and Akt potently blocked phosphorylations of c-Jun and ERK,but not JNK in response to BP,suggesting that PI-3K/Akt pathway positively regulates BP-induced c-Jun activation through ERK.4 Inhibition of p53 by its chemical or molecular inhibitor markedly increased the phosphorylation levels of c-Jun,Akt and ERK upon BP stimulation,indicating that p53 negatively medicates BP-induced c-Jun activation through PI-3K/Akt/ERK pathway.5 The cell lines expressed TAM67 exhibits no significant affecting normal cell growth properties.6 TAM67 was able to significantly block G_1-S transition and subsequent cell proliferation,suggesting that c-Jun is essential for cell cycle alternations elicited by BP.7 Overexpression of TAM67 impaired BP-induced cyclin D1 activation,decreasing expression of E2F1 and pRb,indicating that c-Jun participates in the modulation of BP-induced activation of cyclin D1/pRb/E2F1 pathway.8 Stably expression of TAM67 led to the increases in the expression levels of p53 and p21,elevating phosphorylation level of p53,clearly indicating that c-Jun regulates p53/p21 pathway activation induced by BRCollectively,PI3K/Akt/ERK pathway mediated BP-induced c-Jun activation through p53-dependent mechanism.

结果显示：1BP刺激细胞可促进c-Jun活化，并伴随着MAPK、PI-3K、p53和cyclinD1通路各组成成分的活性增强。2利用MAPK通路的显性失活突变体分别阻断细胞外信号调节激酶和c-Jun氨基末端激酶活性，均可明显抑制BP诱导的c-Jun活化，但阻断p38活性对BP引起的c-Jun活化无明显影响，提示JNK和ERK通路参与调控BP诱导的c-Jun活化。3过表达PI-3K和Akt的显性失活突变体也可显著抑制BP诱导的c-Jun活化，并降低磷酸化ERK的表达水平，但对磷酸化JNK的表达水平无明显影响，说明PI-3K/Akt通路通过ERK正性调控了BP诱导的c-Jun活化。4p53的化学/分子抑制剂能使BP作用的细胞内c-Jun活性明显增加，并同时诱导Akt和ERK的磷酸化水平的升高，表明p53可通过PI-3K/Akt/ERK通路对BP诱导的c-Jun活化进行负性调控。5随后观察转染细胞的生长情况，发现TAM67对细胞正常生长和形态无明显影响。6稳定表达TAM67可有效抑制BP诱导的S期细胞数的增加，提示c-Jun在BP致细胞周期改变的过程中发挥了重要作用。7TAM67过表达能够抑制BP诱导的cyclin D1活化，降低磷酸化Rb以及E2F1蛋白表达水平，表明c-Jun参与调控BP诱导的cyclin D1/Rb/E2F1通路的活化。8过表达TAM67可使BP刺激的细胞中p53、p21总蛋白以及p53磷酸化的表达水平明显升高，可见c-Jun也参与调控BP诱导的p53/p21通路活化。

Tissue factor pathway inhibitor is the majo rinhibitor of the extrinsic pathway of the blood coagulation cascade.

组织因子途径抑制因子(tissue factor pathway in hibitor， TFPI)是组织因子凝血途径的主要抑制因子，具有抑制组织因子、凝血因子VIIa、和Xa的功能。

Taken together, our data showed that both the CSF-box and the octamer are important for the LPS-induced G-CSF promoter activity, moreover, the activation of MEK/ERK signaling pathway is indispensible for LPS-induced G-CSF expression in RAW264.7 macrophage.. It seems that LPS-induced activation of MEK/ERK pathway does not directly regulates NF-κB transactivity, but is essential for LPS-induced Oct-2 binding to the octmaer within G-CSF promoter and transcription activation of the G-CSF expression.

透过这些结果，我们认为MEK/ERK pathway的确参与LPS活化G-CSF表现的过程，虽然MEK/ERK pathway都影响了CSF-box以及octamer对G-CSF启动子所贡献的活性，但似乎MEK/ERK pathway的下游并非直接影响NF-κB调控基因转录的活性，而主要是影响了Oct-2对G-CSF启动子的调控。

Background: New studies showed that changes in the balance between haemostatic and antithrombotic processes, including a prethrombotic state might contribute to the development of atherosclerotic lesions which occured in hypertensive vascular complications. This may be one of the causes that satisfactory blood pressure reduction with antihypertensive drugs does not lead to an equal reduction in heart attacks and strokes. With the development of traditional presentation of blood coagulation, the role of tissue factor pathway has been emphasized.

中文题名原发性高血压患者组织因子途径变化与血栓前状态关系的临床与实验研究副题名外文题名 Clinical and experimental study of relation between changes of tissue factor pathway and prethrombotic state in hypertensive patients 论文作者张卫茹导师孙明教授学科专业内科学研究领域\研究方向学位级别博士学位授予单位中南大学学位授予日期2002 论文页码总数70页关键词血栓组织因子高血压馆藏号BSLW /2003 /R544 /3 背景：新近的研究表明，凝血与抗凝系统失衡所导致的血栓前状态与高血压并发症的发生关系密切，这可能是高血压病人虽经有效的降压治疗但发生心、脑、肾、血管并发症的危险仍很高的主要原因之一。

The absorption and distribution of chromium were studied in ryeusing nutrient culture technique and pot experiment.

采用不同浓度K2CrO4(0，0.4，0.8和1.2 mmol/L)的Hoagland营养液处理黑麦幼苗，测定铬在黑麦体内的亚细胞分布、铬化学形态及不同部位的积累。

By analyzing theory foundation of mathematical morphology in the digital image processing, researching morphology arithmetic of the binary Image, discussing two basic forms for the least structure element: dilation and erosion.

通过分析数学形态学在图像中的理论基础，研究二值图像的形态分析算法，探讨最小结构元素的两种基本形态：膨胀和腐蚀；分析了数学形态学复杂算法的基本原理，把数学形态学的部分并行处理理念引入到家实际应用中。