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oxidative相关的网络例句

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The effect of oxidative accelerant NaNO_2 and La(NO_3)_3 on potential vs time relationships was studied. The growth process of phosphate coating was also observed.

研究了氧化剂NaNO2和稀土促进剂La(NO3)3对磷化过程的φ-t曲线的影响,及磷化各阶段的磷化膜生长情况。

Moreover, the magnesium salt of ferulic acid has been proved to have more efficient anti-oxidative activity.

另外,通过对自己合成的阿魏酸镁与〓之间电子转移反应的瞬态研究,发现它具有比阿魏酸强得多的抗氧化效果。

Reactive oxygen species causing DNA oxidative damage comes from two kinds of ways:one is from cellular normal physiological metabolism;the other is from outer environment.Redox-sensitive green fluorescent protein was expressed in Saccharomyces cerevisiae.Recombinant cells were evaluated in monitoring the changes in the redox state of living cells when challenged with toxicologically relevant metal ions NaAsO_2 or Pb(NO_3)_2 by measuring emission intensity at 510 nm with a Hitachi F6500 fluorescence spectrophotometer,roGFP expressed in yeast responded not only to typical membrane-permeant oxidants H_2O_2 and reductants DTT,but also to toxicological metal ion-induced intracellular redox changes in a dose-dependent manner.Moreover,exposure of yeast cells to NaAsO_2 or Pb(NO_3)_2 at concentrations that induced redox changes reported by roGFP caused up to 2~3 fold increases in DNA mutation frequency.This mutagenic effect was largely caused by oxidative stress since blocking the production of hydryl radicals with thiourea significantly reduced the mutation rate as well as delayed the cell death.

本文将对氧化还原状态变化敏感的绿色荧光蛋白roGFP1-R12,在酵母细胞中实现了多拷贝强表达;荧光扫描经强氧化剂H_2O_2和还原剂DTT以及环境中重金属NaAsO_2或Pb(NO_3)_2处理后的酵母细胞悬液,测定510 nm处的荧光发射强度结果显示,表达的绿色荧光蛋白对氧化还原水平敏感,且在510 nm处的荧光强度与一定的重金属浓度呈正相关,即roGFP1-R12在510nm处的荧光发射值随重金属浓度的增高而增强,从而说明重金属对细胞的毒性在一定程度上很可能是通过破坏细胞内的氧化还原平衡发生作用;同时通过该绿色荧光蛋白对胞内氧化还原状态变化的响应情况可以来实时检测环境中的重金属;遗传学的点突变频率及致死率实验数据表明,重金属能导致菌体的点突变频率和致死率升高,且活性氧的清除剂巯基脲能明显降低这种点突变和致死率,说明由重金属引发的这种点突变和致死效应在很大程度上是依赖于重金属对细胞诱导产生的氧化胁迫。

Molybdophopsphoric Acid Catalysis in the Oxidative Cleavage of Olefins by the Hydrogen Peroxide Oxidative cleavage of the double bond could be catalyzed by H_3PMo_(12)O_(40) using H_2O_2 as oxidant, various trisubstituted olefins were oxidized to aldehydes or ketones in high yields. The system was not satisfied for electron-deficient olefins such as trans-(2-nitrovinyl) benzol or chalcone. The reaction of aliphatic olefins did not occur under the same conditions.

二、磷钼酸催化过氧化氢对烯烃的氧化断裂反应用过氧化氢(30%)作为氧化剂,磷钼酸可以催化氧化碳碳双键的断裂反应,三取代的烯烃被氧化生成醛或酮,产率较高;此体系对于缺电子烯烃如查尔酮,不饱和硝基化合物的断裂反应效果较差,脂肪族烯烃在此体系下的氧化并不发生。

Terminal oxidative inhibitors, such as KCN, Salicylhydroxamic acid , antimycin A and NaN〓, can cut off electron transport chains of cytochrome pathway and alternative pathway, marked decreased BADH protein levels. Oxidative phosphorylation uncoupler 2, 4-dinitrophenol had a similar negative effect on BADH gene expression, and exogenous ATP and ADP increased the BADH levels. These results, in conjunction with the terminal oxidation results, indicated that ATP could be a critical component in BADH gene expression.

2.2 末端氧化抑制剂KCN、SHAM、抗霉素A和NaN〓能打断呼吸链,阻断电子传递,使BADH蛋白的表达量明显降低,说明末端氧化同样参与BADH基因表达的调节:末端氧化影响基因表达的实质就是ATP的影响,线粒体磷酸化解偶联剂DNP明显降低BADH蛋白的含量,而外源的ATP和ADP对BADH基因表达的正效应作用最大。

From the experiment, we can conclude that formaldehyde can decrease anti-oxidative ability of mice and damage freebase or active oxygen clearing system , which can cause oxidative injury of lung tissue in mice.

本研究结果表明,通过亚慢性毒性实验,吸入一定浓度的甲醛,可明显降低肺组织的抗氧化能力,损伤自由基和活性氧的清除系统,导致肺部的氧化性损伤,引起肺组织病变。

The result indicated BaP and the subsequent oxidative stress disturbed the expression of mitochondrion-encoded genes, and this was potential biomarker for oxidative stress following xenobiotic exposure.

研究表明,土壤中苯并[a]芘污染改变了蚯蚓线粒体基因组的表达水平,线粒体基因组表达异常是多环芳烃污染胁迫的重要分子生物标记物。

Increased levels of RONS and oxidative damage are initiators of a specific adaptive response, such as the stimulation of the activation of antioxidant enzymes, thiols, and enhanced oxidative damage repair.

规律性的运动能加强机体对氧化应激的适应性和系统自我修复的能力,这能降低氧化损伤的程度和提高机体对氧化氧化应激产生时的抵抗性。1: Exerc Immunol Rev。

The results show that the degradation effectiveness of PVA was optimum when potassium ferrate and sodium hypochlorite were jointly used as the oxidative agent. The removal rate of PVA and COD was more than 98% and 40% under the conditions that the concentration of PVA was 3.0 g/L and the pH value was 7.2, the dosage of potassium ferrate and sodium hypo chlorite were 0.32 g/L and 3.92 g/L, and the reaction time was 50 min. In the process of united oxidative degradation, the chain of PVA was cut off and many low molecular weight compounds were formed. The main degradation products were carboxyl compounds.

结果表明,用高铁酸钾与次氯酸钠联合氧化PVA时,降解效果最佳;在3.0 g/L,pH值为7.2的PVA溶液中,当高铁酸钾与次氯酸钠的投加量分别为0.32 g/L、3.92 g/L,反应时间为50 min时,PVA的去除率大于98%,COD去除率大于40%;在联合氧化降解过程中,PVA断链成小分子物质,最终降解产物主要为羧基化合物。

Influence of the antioxidants vitamin E and idebenone on retinal cell injury mediated by chemical ischemia, hypoglycemia, or oxidative stressRego A, SantosM, Oliveira C.. Free Radic. Biol. Med, 1999, 26: 1405 - 1417Abstract:A role for the antioxidants vitamin E and idebenone in decreasing retinal cell injury, after metabolic inhibition induced by chemical ischemia and hypoglycemia, was investigated and compared with oxidative stress conditions.

对由于化学贫血与低血糖所引起的代谢抑制后,抗氧化剂维生素E与艾地苯醌视网膜细胞受损所引起的作用进行了研究,同时并与氧化应激情况进行了对比,对氧化物维生素E(20 microM)和艾地苯醌(10 microM)进行先期培养可以在氧化应激或低血糖后防止视网膜细胞受损,而对上述二种抗氧化剂给予预先培养后带来的保护作用则有所减低。

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