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not detectable相关的网络例句

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与 not detectable 相关的网络例句 [注:此内容来源于网络,仅供参考]

The infestation of avocado root rot disease is not visually detectable.

鳄梨树根腐病的感染用视觉是无法探测出来的。

So in fact, if they senesce just a tiny tiny bit over time -- that is, if their half-life gets shorter, very very slowly -- we wouldn't actually be able to tell that this was happening in the time that we took our measurements, because the acceleration in the rate of atoms decaying (relative, of course, to the number that had not decayed already, which is always decreasing) would be too tiny to be statistically detectable.

所以在事实上,如果它们随时间衰老得只那么一点点,—就是说,如果它们的半衰期缩短得非常慢—那么我们实际上不可能知道,在我们测量期间发生过衰减,因为原子衰减的速率加速得太小,统计学上检测不出(当然,是相对于不曾衰减的数目而言,不曾衰减的原子数目总是在减少)。

Because a stimulus does not suddenly become clearly detectable at all times at specific intensity, the operational definition of absolute threshold is the stimulus level at which a stimulus is detected half the time.

因为刺激并不是突然间在某种强度变得能轻易被觉察到,所以关于绝对阈限的定义是--能在一半的次数中被被试所觉察到的刺激水平。

This is no feature of trisomy 21,though some Down syndrome fetuses may not have detectable abnormality on routine scam.

这不是21三体综合征的特点,尽管有些唐氏综合征胎儿可能不会对日常检出异常。

Results Expression of HGF and VEGF mRNA varied in normal mouse vibrissa follicles during hair growth cycles. HGF and VEGF mRNA were detectable in anagen follicles, but not in catagen/telogen follicles.

结果我们的研究发现正常毛囊生长周期中,生长期毛囊有明显的HGF和VEGFmRNA表达,而退行/休止期毛囊则很难检测出上述因子mRNA的表达。

The frequency of detectable viremia after year 4 was not associated with the magnitude of the CD4 cell count change.

但是,有些患者即使经过多年有效的抗病毒治疗,CD4 细胞计数仍不能完全恢复正常。

Compared to normal tissues,27 out of 40 colonic carcinoma did not show any detectable Syk expression.

目的:探索Syk基因的表达与大肠癌生成及转移的关系。

At the sixth day the expression of HGF and VEGF mRNA was still detectable for the group in which the follicles were cultured with Ligustrum lucidum, but not for control group.

与女贞子混合培养的毛囊,在培养的第6天仍有相当量的HGF和VEGFmRNA表达,而相应的对照组则未能测到这些因子的表达。

In S'pore, milk powder can not have detectable anti biotic and in M'sia, the range is 30 - 100 microgramme/kg depending on the type of anti biotic.

在新加坡,奶粉不能被发现含有抗生素,在马来西亚,根据抗生素种类的不同,范围可以在30~100mg/kg之间。

Majority of acute leukemias in infant, either acute lymphoblastic leukemia or acute myeloblastic leukemia, posses a chromosomal translocation affecting the 11q23 chromosome region which specifically inoles the mixed-lineage leukemia gene.1-3 Most pediatric leukemias with MLL rearrangement clearly hae a remarkably short latency.1,4 MLL gene rearrangement is also associated with secondary leukemias of patients preiously treated with the topoisomerase II inhibitors.4 The latency of these secondary leukemias is similarly ery short.4 Of note, the concordance rate of leukemia with MLL rearrangement in infant monozygotic twins approximates to 100%,1,4 and identical breakpoint in the MLL gene was shared in these pairs of identical twin infants with concordant ALL.1,4 Moreoer, the unique and clonotypic MLL fusion gene was detectable in neonatal blood spots for Guthrie cards from non-twined indiiduals who subsequently deeloped ALL.1,4 These obserations indicate not only that MLL fusion is generated in utero but also that MLL fusion proteins could be capable of inducing leukemic transformation with few, if any, secondary mutations.2,3,4 Greaes et al speculate that an MLL fusion protein somehow promotes rapid transition to full-blown disease in patients ia ery rapid clonal expansion, genetic instability, or inhibition of DNA damage repair.4 In general, for clonal expansion of malignancies, tumor cells often hae acquired strategies that escape immune sureillance of the hosts.5,6 Immune escape mechanisms also contribute to the failure of graft-ersus-leukemia effect after allogeneic hematopoietic stem cell transplantation.7 Therefore, leukemia cells could acquire some immune escape mechanisms during leukemogenesis.

绪论 绝大多数的婴儿白血病,不管是急性淋巴性白血病或是急性骨髓性白血病,在染色体11q23部位有染色体易位的情况;这个部位的染色体易位牵连了混合谱系白血病基因。大多数具有MLL基因重排的儿童白血病潜伏期明显短很多。MLL基因重排也和经拓扑异构酶II抑制剂治疗后的继发性白血病有关。这些继发性白血病的潜伏期类似地都非常的短。很重要的是,单卵双胞胎婴儿同时患有或同时免于MLL基因重排阳性的白血病的一致性接近100%;并且同样患有ALL的同卵双胞胎的MLL基因的断裂点是一致的。而且,这种独特的克隆特异性的MLL融合基因能够从那些得ALL的非双生个体出生时的血斑标本中检测到。这些发现表明MLL融合基因产生在胎儿还在子宫的是后,而且MLL融合蛋白能过和其他的基因突变一起诱导白血病的产生。Greaes 等推测MLL融合蛋白在某种情况下同过快速克隆增殖,遗传的不稳定性或是DNA损伤修复的抑制促使疾病迅速地全面爆发。恶性肿瘤细胞的克隆增殖通常已经获得了逃避机体免疫监视的能力。免疫逃避机制也归因于异体外周血干细胞移植后移植物抗白血病作用的失效。所以,白血病细胞在白血病的产生过程中可能获得了某些免疫逃脱机制。

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