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normotensive相关的网络例句

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与 normotensive 相关的网络例句 [注:此内容来源于网络,仅供参考]

In this context, we have reported recently that the volume of myocardial tissue occupied by perimysial and endomysial collagen was lower in hypertensive patients with HF and depressed ejection fraction, than in hypertensive patients with HF and preserved ejection fraction and normotensive subjects.56 Of interest, the interstitial and perivascular accumulation of collagen fibres was abnormally high in the two groups of patients.

在这种情况下,我们已经说了占用体积perimysial心肌组织人肌内膜胶原水平低的高血压患者用高频和沮丧的射血分数在高血压患者的心力衰竭和保存被罚出场分数与正常血压subjects.56舒张压的利息、间质和血管堆积过高的胶原纤维是在两组病人。

On the basis of the above studies, the effects and mechanisms of microinjection of glucocorticoid or mineralocorticoid into the rVLM on the arterial blood pressure, HR, pressor and tachycardiac responses produced by stimulation of the dorsal periaquaductal gray in the midbrain were further observed and analysed in the acute experiments on normotensive rats.

在此基础上,进一步在急性动物实验中观察分析肾上腺糖、盐皮质激素在rVLM区对血压、心率、电刺激防御反应区诱发的防御性升压和加快心率反应的作用及其机制。

Microinjection of glucocorticoid or mineralocorticoid (40 or 10 ng/0.1 ul/site) into the rVLM bilaterally caused an increase in BP, HR and the pressor response induced by the dPAG stimulation in the normotensive rat, respectively. Dose-dependence of both drugs was observed on the above effects in three directions. Nevertheless, there was no effect of both drugs on the tachycardiac response induced by the dPAG stimulation.

二、分别微量注射糖、盐皮质激素(40或10ng/0.1μl/site)至双侧rVLM,可引起正常血压大鼠的BP升高,HR加快,以及电刺激中脑中央周围灰质背侧区诱发的防御性升压反应增强,并均呈现出剂量-效应依赖性关系,而对电刺激dPAG诱发的加快心率反应无明显改变作用。

After microinjection of the antagonists of glucocorticoid RU 38486 or mineralocorticoid spironolactone (40 ng/0.1 ul/site, respectively) into the rVLM bilaterally, a decrease in BP, HR and the pressor response induced by the dPAG stimulation was observed in the normotensive rat, and there was no change on the tachycardiac response induced by the dPAG stimulation.

分别微量注射糖、盐皮质激素的拮抗剂RU 38486(40ng/0.1μl/site)或螺旋内酯(40ng/0.1μl/site)至双侧rVLM,则引起正常血压大鼠的BP下降、HR减慢,以及电刺激dPAG诱发的防御性升压反应减弱,对电刺激dPAG诱发的加快心率反应无明显改变作用。

On the normotensive rat, bilateral microinjection of L-arginine (16 n mol/0.1 ul/site), a precursor of nitric oxide into the rVLM, caused a decrease in BP, HR and the pressor response induced by the dPAG stimulation, while microinjection of NG-methyl-Larginine (L-NMMA, 16 n mol/0.1 ul/site), a blockade for NO to form, into the rVLM bilaterally, caused an increase in BP, HR and the pressor response induced by the dPAG stimulation. There was no effect of both drugs mentioned above on the tachycardiac response induced by the dPAG stimulation.

五、在正常血压大鼠,于双侧rVLM微量注射一氧化氮前体(16nmol/0.1μl/site)可引起BP降低、HR减慢、以及电刺激dPAG诱发的防御性升压反应减弱;而于同区微量注射生成一氧化氮的拮抗剂N〓-甲基-L-精氨酸(16nmol/0.1μl/site)则可引起BP升高、HR加快、以及电刺激dPAG诱发的防御性升压反应增强;而上述两药对电刺激dPAG诱发的防御性加快心率反应均无明显作用。

Methods: ELISA was used to determine the serum of TPA and sVCAM-1 in pre-eclamptic and normotensive pregnant women, and the correlation of pre-eclampsia and neonatal outcomes was analyzed by rank correlation.

采用酶联免疫吸附法(enzyme linked immunosorbent assay,ELISA)测定子痫前期患者和正常晚孕妇女外周血中TPA、sVCAM-1的浓度,并与子痫前期患者病情及新生儿体重进行相关分析。

We need to complete preventive and therapeutic clinical trials in the near future if we are to identify certain anti-hypertensive drugs with anti beta-amyloid anti-oligomeric activities, which will need to be prescribed at dosages that do not interfere with blood pressure in normotensive Alzheimer's disease patients.

& &我们需要完整的预防和治疗的临床试验在不久的将来,如果我们的目的是查明某些抗高血压药物,抗β-淀粉样蛋白反低聚活动,这部分须在处方用量不干扰血液压力血压正常的老人痴呆症患者。

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