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Based on the quasi-dimension combustion model, the emission model is set up in this dissertation. The NO emissions in different conditions of LPG engine can be simulated and calculated by the model. The calculated results show that the NO emissions are larger influenced by ignition timing and less influenced by compression ratio, finally, choosing a right coefficient of excess air will have great effect to reduce NO emissions.

本文在建立的准维燃烧模型基础上建立了排放生成模型,利用该模型对液化石油气发动机在变参数情况下NO的排放生成情况进行了模拟计算,计算结果表明点火提前角对NO的生成影响较大,压缩比对NO的生成影响较小,而选择合适的过量空气系数更是对降低NO的生成起着关键作用。

At low temperature (700℃), NO��� emission increases considerably with excess air coefficient, at 800℃ NO��� emission is almost independent of excess air coefficient, and at 900℃ NO��� emission is zero indicating that this temperature is the optimal operating temperature.

稀相区出口浓度为零,密相区内NO和NO〓浓度随氧量增加而减少,说明在900℃下,氧量增加促进NH〓(i=0,1,2)对NO〓还原。

Result revealed :Long time resistance training could increase the level of NO, iNOS and TNF-a in serum of rat, which had correctitude correlations in diversification . The level of NO, iNOS and TNF-a in serum of untrained rat had a markedly raise after one time exhausting exercise, which was likely to have relationto the answer of organism to exercise. During exercise, the raise of level of NO, iNOS and TNF-a in serum of untrained rat could promote blood circle ,transportation of substance and energy, level of immunity and was seasoned with other level of cytokines .

结果显示,大鼠长期有氧训练能够提高其血清中NO、iNOS和TNF一a水平,三者变化呈现正相关;未训练大鼠在一次性运动至力竭时,NO、NOS和TNF一a水平显著上升,这可能与一次性运动相适应机体生理应激有关,TN下一a、NO、iNOS含量/活性水平显著提高,促进血液循环,提高物质能量的运输,优化免疫水平以及与机体内其他细胞因子水平相适应。

Hoisting beam of the No.1 crane was replaced with that of No.4 crane and the brake assembly of the hoisting motor was replaced with a ship's spare, which had been sustained damage in the incident of the No.1 crane ar Port of XXX, by the crew so that the No.1 crane could be continued in normal working.

为了不影响卸货,船方将No.1起重机吊钩梁与No.4的进行调换,并由船员利用船上的备件对在XXX港No.1起重机损坏案相关的起货马达刹车部件进行了更换,使No.1

Three kind of bond energies of the Co-NO bonds in series of cobalt nitrosyl tetraphenylporphyrins have been determined by thermodynamic cycles combined with calorimetric measurements and relevant electrochemical data.

本文以金属卟啉模拟体内重要的NO靶分子金属蛋白,通过确立新的适于量热和电化学测定的Y/〓成Y-NO键反应体系,利用合适的热力学循环,建立了新的适于金属卟啉体系的NO亲合势的测定方法,得到了苯腈中系列亚硝基四苯基钴卟啉Co-NO键的键能,即钴卟啉NO亲合势的定量标度,以表征钴卟啉的NO结合能力。

The concentrations of TNF-αand IL-10 in peripheral blood increased significantly after CVVH.The concentration of NO decreased significantly after CVVH.There was no TNF-αin ultrafiltrate;IL-10 was detected little and NO was detected much in ultrafiltate.Compared with prefilter,the concentration of TNF-αin postfilter blood decreased significantly at 1h of CVVH and increased at the end of CVVH. Compared with prefilter,the concentration of NO in postfilter blood decreased significantly at 1h ,12h and the end of CVVH.

CVVH治疗后患者血清TNF-α、IL-10浓度与治疗前相比明显升高,而NO浓度与治疗前相比显著下降,超滤液中未能检测出TNF-α,仅检测到少量IL-10,但NO在超滤液中浓度较高;CVVH治疗1h时,静脉端血清TNF-α浓度明显低于动脉端,CVVH治疗结束时,静脉端血清TNF-α浓度明显高于动脉端;CVVH治疗1h,12h和结束时,静脉端血清NO浓度均明显低于动脉端。

After being treated with Nephritis NO.1 Prescription, NO and the ET-1 contents in Model group recovered(P〈0.05)2、Nephridial tissue examination indicated that there were lessened microscopic structural pathological changes ,cellular proliferation and widening mesangial region in mesangial cells through treatment compared to Model Control , Conclusions :1 For Mesangial Proliferative Nephritis , Manshen NO.1Prescription has satisfactory therapeutic effects, It can improve symptoms, restrain accrementition in intercapillary cells and mesangial region, thus prevent nephric tubule's indurascent and prevent renal failure.2、Manshen NO.1 Prescription can increase NO contents; reduce ET-1 contents in Model control.

结果:1、与正常对照组相比,模型组血清NO含量减低(P〈0.05),而ET-1的含量升高P〈0.05〉,给予慢肾1号方治疗后,可明显提高系膜增生性肾炎模型组血清NO(P〈0.05〉,减低血清ET-1的含量P〈0.05〉,2、肾组织学检查显示:经慢肾1号方治疗后,与模型组相比,肾小球系膜细胞增生及系膜区增宽等超微结构病变均有所减轻。

Result:① In NC group,the levels of plasmic and lymphocytic NO were higher in the morning than those in the afternoon.② The plasmic and lymphocytic NO were significantly decreased in CAD patients then those in NC group (P<0.01).③The physiological NO curve disappeard in CAD patients.The concentrations were no obvious changes and maintained lower levels in 24 hours.Conclusion:As similar as other endocrinic hormones,the NO secretion was not equal in 24 hours.

结果:①正常人24 h血浆和淋巴细胞中NO分泌呈上午高、下午低的趋势,晨6时最高,下午6时呈低谷,晚10时起又渐升高至早晨;②CAD心绞痛患者血浆和淋巴细胞中NO含量明显低于正常人,其差异具有统计学意义(P<0.01);③CAD患者失去了正常的NO生理性分泌曲线,血浆和淋巴细胞中24 h均在较低水平,波动范围不大。

Additionally, analysis of G-banding patterns for macrochromosomes of them indicated that there was great difference between NO.1 and NO.2 chromosome, which can be explained by the pericentric inversions.

NO.l染色体,家鸡为m型,而鹌鹑为sm型;NO.4、NO.6、NO.8和Z染色体,家鸡分别为sm、sm、m、m型,而鹌鹑全为t型。G带分析结果显示,家鸡和鹌鹑的G带差异主要反映在NO.1、NO.2染色体上,这可能是由於臂间倒位所致。

The enhancement of NO oxidation to NO��� and the suppressing of propene combustion are beneficial to NO selective reduction.

在NO-SCR催化剂研究中,抑制烃类的完全燃烧和提高NO氧化为NO〓的能力,是提高NO选择还原性能的有效途径。

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