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no-win相关的网络例句

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与 no-win 相关的网络例句 [注:此内容来源于网络,仅供参考]

N L-Leucine Producer,Strain No .145,was obtained from mutants resistant to α-amino-β-hydroxyl valeric acid and rifampin by mutagenizing Brevibacterium Tianjinese T6-13 with ul-traviolet rays,It produced L-Leucine up to 32mg/mL on shaker and 21.8mg/mL of L-Leucine was produced in 16 liter fermentor in 60 hours.

以天津短杆菌T6~13为出发菌株,采用紫外线进行诱变处理,筛选α-氨基-β-羟基戊酸和利福平抗性突变株,获得一株L-亮氨酸产生菌No.145,L-亮氨酸摇瓶产量可以达32.1mg/mL。在16L发酵罐中培养60h,L-亮氨酸产量可以达21.8mg/mL葡萄糖转化率可以达20%。

In the group of valvular heart disease, NO level increases 18 hours after stopping using L-arginine.

在瓣膜性心脏病引起的肺动脉高压患者中,停止用药18小时后仍能监测到血清NO浓度的升高。

And under the presence of high concentrated ammonium sulfate aerosol seed,the concentration of aerosol does not have significant effects on NO_x,NO and O_3 varia...

在高浓度的硫酸铵气溶胶种子条件下,其初始浓度对反应过程中NOx、NO和O3的浓度变化没有明显的影响,但对二次有机气溶胶(secondary organicaerosol,SOA)的生成有显著影响。

Overproduction of NO by iNOS is thought to be the principal factor of hypotension and hyporeactivity to vasoconstrictor agents observed in endotoxic shock.

特别是内毒素所引起之败血性休克时,iNOS产生过量的NO被认为是导致低血压,低收缩反应性血管及组织氧化性伤害的主要原因之一。

Simavastatin has comparative beneficial effects on improving the impaired endothelium-dependent vasodilatation and increasing serum NO, and this may be one of mechanisms for the beneficial effects of simavastatin in prevention and treatment of CAD.

辛伐他汀通过增加血清NO浓度改善冠心病患者的血管内皮功能,该作用可能是他汀类调脂药防治冠心病的机制之一。

The data here indicate that in patients with CHD, endothelium vasodilation is impaired and serum NO decrease significantly.

冠心病患者肱动脉内皮依赖性血管舒张功能明显减退,血清NO浓度降低。

Therefore, NO may be a potential selective pulmonary vasodilator.

因此,NO可能为一种有很大发展潜力的选择性肺血管扩张剂。

ET is a peptide with vasoconstrictive and vasodilator effect produced by endothelial cells,and NO as a powerful ...

实验证实ET和NO与肝硬化的发生密切相关。

Notifying Compound preventing and treating menopause coronary heart disease may be concerned with regulating NO and ET balance, and improving vasomotor function.

结论]补肾复方防治绝经后冠心病的机理可能与调节NO和ET两者平衡,改善血管舒缩功能有关。

Conclusion] The mechanism of Kidneynotifying Compound preventing and treating menopause coronary heart disease may be concerned with regulating NO and ET balance, and improving vasomotor function.

结论]补肾复方防治绝经后冠心病的机理可能与调节NO和ET两者平衡,改善血管舒缩功能有关。

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