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No atrophy was found in the surviving motoneurons, and the expression of NOS was inhibited. Conclusion SDNF can prevent the motoneurons from death following spinal root avulsion. NO may play a role in the injury-induced motoneuron death.

SDNF对脊髓前角运动神经元死亡有明显的保护作用，NO在臂丛神经根性撕脱所致运动神经元死亡中起一定作用。

In addition, Prescription for flourishing stomach could relieve the inflammatory reaction induced by NO, and prevent the gastric mucosa from damaging by oxidative nitrous acid, benefiting for preventing canceration.

欣胃方有降低NO的作用，可减少NO诱发的炎症反应，阻断过氧化亚硝酸对粘膜的进一步损害，对预防癌变有积极的作用。

The possible mechanisms may be inhibition of overproduction of NO, and reduction the cytotoxicity as free radicals of NO.

其作用机制可能与抑制NO的过量产生，减轻其作为自由基的细胞毒性有关。

In plants , NO has recently been demonstrated to be involved in the regulation of growth and development in the response to abiotic and biotic stresses and it is a key signal messenger in disease resistance in plants. More important , it has been found that NOs function in plant is similar to the traditional phytohormone , so it is forecasted to be a new type of hormone in plant.

近年来的研究发现，它不仅在血管松弛、神经传导以及先天性免疫反应等动物生理代谢过程中是一种关键的第二信使，而且在植物中也起着非常重要的作用，是植物生长和发育的调节分子，能够对生物性和非生物性的胁迫作出反应，并且在植物体的抗病防御反应中也是一种重要的信号物质；更重要的是，近年来发现NO与各种植物激素的功能都有着重叠，因此有人预测NO可能是植物体内的一种新型激素。

Vesicatoria and compatible bacterial phytopathogen Xoo with Nipponbare were used to investigate the production of NO, the activities of iNOS and the role of NO in hypersensitive response.

NO在过敏性反应中的作用。研究结果表明，1。

Methods The rabbit model with hyperhomocysteinemia was established by L-methionine subcutaneouse injection. The changes of white microthrombus and microvascular diameter in pia mater microvessel were observed by using cranial window method and closed microcirculation detection system, The concentration of Hcy was detected by high performance liquid, The level of von Willebrand factor, endothelin-1 (ET-1) were assayed by ELISA. The content of NO was measured by nitrate reduction method. Meanwhile, the relationship between the changes of microvascular diameter and blood concentration ET-1, NO was analyzed.

采用L-蛋氨酸皮下注射法建立HHcy家兔模型，应用"颅骨窗口法"和微循环闭路检测系统观察其软脑膜微血管中白微栓数量及微血管口径变化；采用柱前衍生反相高效液相色谱－荧光法检测同型半胱氨酸浓度；平行观测血管内皮功能指标血管性血友病因子抗原（vWF，用酶联免疫吸附法）、血浆内皮素－1ET-1，用ELISA）、血清一氧化氮水平；并对HHcy所致ET-1、NO血液浓度变化和微血管口径变化进行相关分析。

HA increased level of NO by activating NOS after combining with H〓 and H〓 receptors; and on the one hand 5-HT caused brain microvessel endothelial cells constracting strongly, made the tight joint opening, increased the permeability of BBB, and on the other hand 5-HT increased the quantity of pinocytosis vesicle in endothelial cells and enhance the permeability of BBB after comining with 5-HT〓 receptor.

HA与H〓和H〓受体结合，激活NO合成酶，使NO含量增多而诱发BBB通透性增加；5-HT与5-HT〓受体结合，一方面引起脑微血管内皮细胞强烈收缩，使内皮细胞间的紧密连接开放，BBB通透性增高，另一方面可引起内皮细胞转运小泡增加，BBB通透性增高。

To observe the effect of bifidobacterium on plasma endotoxin NO and hemorheological characters in cirrhotic rats,we have detected plasma levels of endotoxin and No and index of hemorheology at the end 11th week after CCl4 administraation in preclusive groups and later treatment groups microecological preparation- bifidobacterium administraation.

结果显示，预防组血浆内毒素、NO水平及血液流变学多项参数明显低于肝硬化模型组（P＜0.05～0.of），与健康对照组相比，差异无显著性（P＞0.05），而晚期治疗组介于模型组与健康对照组之间。

The ET and NO produced locally in renal play more important role in the development of proteinuria.

在蛋白尿发生发展过程中血浆和肾皮质ET、NO均有显著改变，而肾皮质ET、NO含量的改变在蛋白尿的发生发展中起重要作用。

Because of its redox properties, the role of NO in tumor biology has gained much attention in recent years.

由于NO具有氧化还原特性，近年来NO在肿瘤生物学中的作用受到了极大关注。

The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

这种做法不仅鼓励了更多的反应，而且减少跟风的可能性。