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Excessive production of No is neurotoxic and is implicated in a variety of neurological disorders.

过多的NO则具有神经毒性并与许多神经系统疾病有关。

Neuronally derived NO participate in the neurotoxic mechanism of the rat model of PD

来源于神经元的NO参与了PD大鼠模型的神经损伤过程。

The level of NO in the brain tissue was determined by nitrate reductase method.

以硝酸还原酶法测定脑组织中的NO水平。

Sphaeroides (108 no./ml) were added in five different concentrations of Nitrous acid in water to study the removal of Nitrous acid after 24 hours.

ml於24小时后，水中亚硝酸盐去除率达100%，另外光合菌在五种不同铵氮浓度下，光合菌菌量於108 no。

They are to be stamped with Go/No-Go and size.

他们要被盖印着 'Go'和 'No Go' 和尺寸。

The changes of the content of NO and the activity of NOS in every group were observed in serum.

许多研究表明NO在哮喘的发病和治疗中起重要作用［1］。

The serum level of NO and NOS were determined before and after treatment.

然而辛伐他汀在调脂作用的同时对内源性NO的影响，目前国内研究较少。

Objective It is to investigate the NOS activity and NO content at different time points of myocardial ischemia reperfusion.

目的 研究心肌缺血再灌注过程中不同时点的NOS活性和NO产量。

It is suggested that NO and NOS may play an important role in the metabolism and progress of lung cancer.

提示NO、NOS在肺癌的代谢及病理过程中，可能起重要作用。

From the resnlts above, NYA can promote the nerve function resumed and mend the hematoma absorbed which has further prored its clinical curative effects that NYA can regulate plasma ET and NO levels, and improve the cerebrovascular function, which may be one of the protective mechanisms of NYA from ischemic injury after ICH.

综合以上结果，脑溢安不仅能促进神经功能恢复，加快血肿吸收，其临床疗效得到进一步证实，而且能有效调节ET和NO水平，改善脑血管内皮细胞舒缩因子的释放，可能是治疗急性脑出血，保护脑出血后缺血性损伤的机制之一。

This one mode pays close attention to network credence foundation of the businessman very much.

这一模式非常关注商人的网络信用基础。

Cell morphology of bacterial ghost of Pasteurella multocida was observed by scanning electron microscopy and inactivation ratio was estimated by CFU analysi.

扫描电镜观察多杀性巴氏杆菌细菌幽灵和菌落形成单位评价遗传灭活率。