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NO.1 chromosome was submetacentric,NO.2 and Z chromosome were metacentric,and the others were telocentric,which was slightly different from the results of others. Additionally,analysis of G-banding patterns for macrochromosomes indicated that they were divided into 27 zones and 134 bands.

染色体形态分别为：NO.1染色体为sm型，NO.2、Z染色体为m型，其余染色体均为t型，这与前人研究结果存在一定差异。G带分析结果显示，鹌鹑的前9对大染色体及Z、W染色体G带可分为27个区，134带。

NO could be synthezied for maintaining the resting cerebrovascular tonus in normoal condition and was involved in the vasodilatation mechanism under hypoxia. In these 2 aspects, the NO effects could be inhibited by L-NNA.

在静息状态时，NO有维持离体基底动脉血管条基态张力的作用；低氧时，NO在脑血管的舒张反应中也占有重要地位，两种作用都可以被L-NNA抑制。

The results show that after lone- term load swimming training, the CEC was increased, the plasma ET content was increased and serum NO content was desreased; endothelium ultrastructure was damaged; tragacanth makes the CEC was decreased After large load exercise, the plasma ET content was decreased and serum NO was content was insreased; endothelium ultrastructure was improved.

结果表明：①大运动量运动训练后，大鼠CEC增多，血桨ET含量上升，血清NO含量下降；内皮细胞结构受损。②黄芪使大鼠大运动量训练后CEC减少，血浆ET下降，血清NO含量升高；内皮细胞超微结构改善。

The reason Manshen NO.1 prescription can restrain accrementition in intercapillary cells and mesangial region may be due to its adjustment of NO and ET-1's contents in vivo. It is worth of doing further researchs in the future.

1、慢肾1号方对系膜增生性肾炎有良好的治疗作用，改善症状，抑制系膜细胞及基质区的增生，从而阻止肾小管硬化的发展，防止肾功能衰竭的发生，2、肾慢1号方能够提高模型组小鼠血清NO的含量，降低ET-1的含量，使之恢复正常。3、慢肾1号方可能是通过调节体内细胞因子(NO及ET-1)的含量，从而达到抑制系膜细胞及基质增生的目的，值得深入研究。

NO concentration did not change significantly in the groups with M or S-PH and mild PH before and after CPB.NO level in the group with non-PH 30 min after CPB decreased significantly as compared with that after induction of anes thesia.

伴肺高压两组病人CPB前后NO浓度无明显变化，no-PH组CPB后30分钟时较麻醉诱导后明显降低(P＜0.05)。

The results showed as followed:low level NO could keep Sertoli Cells' microtubule and cell viability normally,and high level NO could increase the activity of p-38MAPK of cells, decrease antioxygen ability and cell secretion and destroy the structure and distribution of microtubule.

结果表明，低浓度NO能维持支持细胞正常的骨架和活力，而高浓度NO能提高SC内p38MAPK的活性，降低抗氧化酶的活性和破坏细胞内微管的结构和分布，甚至细胞的分泌功能。

Results: Compared to healthy subjects, the levels of plasma ET, NO, ET/NO, AMY and uric AMY in patients with SAP increased significantly. All determined indexes, catabatic time of bellyache and time of hospitalization in treated group decreased significantly compared to control group.

结果 SAP组较正常对照组ET、NO、ET/NO及血、尿淀粉酶显著增高；治疗后治疗组较对照组各值明显降低，腹痛缓解及住院时间明显缩短。

Study the activity of inducible nitric oxide synthase in the liver of rabbits infected with Eimeria stiedai, the concentration of nitric oxide in the serum、the coccidian oosysts per gram feces ,Score the pathologies according to anatomic structures of rabiits'livers and micoscopic pathological changes. So as to study the effect of exogenous NO and NOS of the rabbits infected with E.stiedai. The results showed that when the rabbits were infected with E.stiedai, the activity of iNOS in the liver and the concentration of NO in the serum gradually increased and they both reached the peak after 12 days in the control group , GTN and GSNO groups.

为了探讨外源性一氧化氮和一氧化氮合酶在家兔球虫感染时的作用，本试验以腹腔注射的途径给予感染斯氏艾美耳球虫的家兔NO供体硝酸甘油和亚硝基谷胱甘肽，对球虫感染家兔诱导型一氧化氮合酶活性、血清中NO的浓度、每克粪便的球虫卵囊数、肝的剖检和镜下病变及其病变计分进行了研究。

Results: Remarkable pathological changes were found in h ippocampus: Nissl bodies decreased or disappeared, cyton swelled. The water cont ents and the level of LDH and NO were increased in scalded rats. CNTF could rema rkably improve the pathological changes and reduce the increase of LDH, NO a nd the water contents.

结果：大鼠严重烫伤后，脑组织含水量增加，海马出现明显的病理变化，尼氏小体减少或消失，胞体肿胀，LDH和NO的含量增加；给予CNTF后，脑水肿、海马的病理变化有明显改善，并能降低 LDH和NO的含量。

Results: Remarkable pathological changes were found in h ippocampus: Nissl bodies decreased or disappeared, cyton swelled. The water cont ents and the level of LDH and NO were increased in scalded rats. CNTF could rema rkably improve the pathological changes and reduce the increase of LDH, NO a nd the water contents.

结果：大鼠严重烫伤后，脑组织含水量增加，海马出现怎么明显的病理变化，尼氏小体减少或消失，胞体肿胀，LDH和NO的含量增加；给予CNTF后，脑水肿、海马的病理变化有明显改善，并能降低 LDH和NO的含量。

Do you know, i need you to come back

你知道吗，我需要你回来

Yang yinshu、Wang xiangsheng、Li decang,The first discovery of haemaphysalis conicinna.

1〕 杨银书，王祥生，李德昌。安徽省首次发现嗜群血蜱。