查询词典 neurotrophic

Neuroectoderm; neural crest; neural plate; floor plate; neural tube; neuraxis; neurulation; neuroblasts; stem cells; differentiation; ontogeny; morphogenesis; histogenesis; organogenesis; synaptogenesis; gangliogenesis; embryogenesis; axonogenesis; retinogenesis; gliogenesis; glial progenitor cells; oligodendrocyte progenitor cells; retinal progenitor cells; nerve growth factor; neurotrophic factors; trophic factors; growth factors; neural tube defects; anencephaly; spina bifida; neuroblastoma cells; cell migration; neurogenesis; development; developmental stages

神经外胚层；神经脊；神经板；底板；神经管；轴索；神经胚形成；成神经细胞；干细胞；分化；个体发生；形态发生；组织发生；器官发生；突触发生；gangliogenesis；胚胎发生；axonogenesis；retinogenesis；gliogenesis；神经胶质祖细胞；少突神经胶质细胞祖细胞；视网膜祖细胞；神经生长因子；神经营养因子；营养因子；生长因素；神经管故障；无脑；脊柱裂；成神经细胞瘤细胞；细胞迁移；神经发生；发展；发展进程首页上一页下一页末页共有 22 条记录，本页从第 11 到第 20 条。

Neurotrophic factors play an important role in the culture, proliferation and differentiation of stem cells, and regeneration and remyelination of neurite.

神经营养因子在干细胞的培养、增殖、分化以及轴索的再生和再髓鞘化等过程中发挥着重要作用。

Among the involving factors, neurotrophic factors in the microenvironment adjoining to perikaryon and neurites play crucial roles in maintaining survival, promoting orientional regrowth of axons and re-expression of functional phenotype of neurons.

近年来大量神经生物学研究表明：在影响损伤后神经修复或再生的诸多因素中，神经元胞体及轴突周围微环境产生的一些神经营养因子发挥着维持受损神经元生存、促进神经再生、恢复神经功能表型等重要作用。

CONCLIUSION: Human myoblast could be induced to neuron-like cells treated with ciliary neurotrophic factor and Salvia miltiorrhiza injection. The p44/p42 kinase participates in redifferentiation of myoblasts by phosphorylation.

经睫状神经因子预诱导后逐渐去分化的成肌细胞，在丹参注射液的诱导下能够成功分化为表达神经元特异蛋白标记物的神经元样细胞，p44/p42激酶通过磷酸化参与了成肌细胞的再分化过程。

The neurotrophic and myotrophic effect of CNTF ma y play an important role in the treatment of nerve-muscle injury and retrogress ion.

CNTF的神经和肌肉双重营养性能使其可能在神经肌肉损伤和退行性病变的治疗上发挥重要作用。

Brain-derived neurotrophic factor was shown to induce antiapoptotic mechanisms after stroke and to reduce infarct size and secondary neuronal cell death.

脑源性神经营养因子被显示了卒中后可诱导抗凋亡机制并减少梗塞面积以及继发神经细胞死亡。

Results①Make clear of the general principal during the morphologicaldevelopment of the C57BL/6 mouse inner ear and reconstructed the 3D modelof E13 inner ear;②The mature time of many histological structuresassociated closely with the hearing setout of C57BL/6 mouse were madeclear as basilar membrane in D10, scala vestibular and tympanicD4, Cortis tunnel D12, inner sulcus D10, tectorial membrane D10, haircells D12, spiral ganglion neurons D12 and neurofilament in cochlear axisD12-D14.③Outlined the time table and the patterns of the expressionsof those proteins associated with the inner ear development which includedproneural committed proteins, neuronal differential proteins,neurotrophic factors and neuron special proteins.

结果①HE常规染色大体观上了解了C57BL／6小鼠内耳形态发育E8-Da期间的一般规律并且获得了E13的三维立体图像；②获得了各种听功能密切相关的结构的成熟时间点，基底膜(D10)、鼓阶前庭阶(D4)、螺旋器隧道(D12)、内螺旋沟(D10)、盖膜(D10)、毛细胞(D12)、螺旋神经元(D12)和蜗轴纤维(D12-D14)；③完成了前神经决定蛋白、神经分化蛋白、神经营养因子和神经元特异性蛋白等内耳发育相关的蛋白的表达时限表并详细观察了其在内耳各个部位表达随发育变化的特点：④Calbindin在E16以后的螺旋神经元没有表达，但是在scarpa神经元持续表达。

Results①Make clear of the general principal during the morphologicaldevelopment of the C57BL/6 mouse inner ear and reconstructed the 3D modelof E13 inner ear;②The mature time of many histological structuresassociated closely with the hearing setout of C57BL/6 mouse were madeclear as basilar membrane in D10, scala vestibular and tympanicD4, Corti\'s tunnel D12, inner sulcus D10, tectorial membrane D10, haircells D12, spiral ganglion neurons D12 and neurofilament in cochlear axisD12-D14.③Outlined the time table and the patterns of the expressionsof those proteins associated with the inner ear development which includedproneural committed proteins, neuronal differential proteins,neurotrophic factors and neuron special proteins.

结果①HE常规染色大体观上了解了C57BL／6小鼠内耳形态发育E8-Da期间的一般规律并且获得了E13的三维立体图像；②获得了各种听功能密切相关的结构的成熟时间点，基底膜(D10)、鼓阶前庭阶(D4)、螺旋器隧道(D12)、内螺旋沟(D10)、盖膜(D10)、毛细胞(D12)、螺旋神经元(D12)和蜗轴纤维(D12-D14)；③完成了前神经决定蛋白、神经分化蛋白、神经营养因子和神经元特异性蛋白等内耳发育相关的蛋白的表达时限表并详细观察了其在内耳各个部位表达随发育变化的特点：④Calbindin在E16以后的螺旋神经元没有表达，但是在scarpa神经元持续表达。

Objective:To investigate the effects of marrow stroma cells on the expression of nerve growth factor and brain-derived neurotrophic factor after traumatic rat brain injury.

目的：研究骨髓基质细胞治疗脑创伤大鼠后内源性的神经生长因子和脑源性神经营养因子的影响。

Neurogenesis in DG could be induced to increase at a certain time window after global ischemia reperfusion. The accommodational mechanisms of neurogenesis were possibly related to the changes of hormones, neurotransmitters, neurotrophic factors and the micro-circumstances of the dentate gyrus after ischemia.

结论缺血性脑损伤可诱导海马齿状回神经元发生水平在一定时间窗内上调，其机制可能与缺血引起脑组织激素水平、神经递质、神经营养因子浓度发生变化以及齿状回局部微环境改变有关。

The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

这种做法不仅鼓励了更多的反应，而且减少跟风的可能性。