查询词典 neurons

Increased release of acetylcholine from the neurons, leading to heightened activity in cholinergic pathways throughout your brain.

从神经细胞里释放的乙酰胆碱增加，引发遍布你大脑的胆碱能通路活动的提高。

Objective To investigate the expression of muscarinic acetylcholine receptors and GABAB receptors in lamina Ⅱ neurons in spinal cord.

目的观察M胆碱能受体亚型M2和B型γ-氨基丁酸受体在脊髓Ⅱ板层神经元的表达情况。

It is also suggested that ACh plays an important role in the maintenance of excitability of cortical neurons.

这一作用可能是通过作用于M受体，影响突触前递质释放来实现的。

Evidences show that excitotoxcities of excited amino acid and free radical, Ca2+ overload, environmental alteration of neurons, mitochondrial dysfunction and activation of some apoptotic genes may be involved in it.

缺血性脑卒中确切的神经元凋亡机制尚不清楚，目前认为可能与兴奋性氨基酸、Ca2+超载、自由基的毒性作用、神经元生存环境的改变、线粒体功能障碍及凋亡调控基因的激活等因素有关。

This region remains highly active, which wears out neurons and alters their connections to each other.

这一区域一直保持高度活跃的状态，使神经细胞受到劳损，而且改变了神经细胞之间的连接线路。

The more active a region of the brain, the more active its neurons, and in turn, the more blood will travel to that region.

大脑中越是活跃的区域，它的神经元也就越活跃，为此，更多的血液会流经这一区域。

Thus, the membrane of the neurons in the respiratory center and CRCs may own TASK-1, which can sense the change of H+ or CO2 of the extracellular fluid and enhance the output of the respiration by increasing the excitability of the neurons.

因此，我们推测TASK-1可能存在于呼吸中枢和/或中枢化学感受器，当细胞外H+或CO2增加时，可通过作用于该通道而增加呼吸相关神经元的兴奋性，使呼吸活动增强。

Results Cerebroedema appeared in all animals and they have some behavioral alterations. The meninges tighten, the brain swollen overtly. The adventitia of the brain arteries and the perivascular Virchow－ Robin space dilated irregularly. In some neurons both the nucleus and cytoplasm condensed, chromatin gathered near the nuclear membrane. Some other neurons appeared degeneration or necrosis. There was intracelluar edema of the astrocytes.

结果所有动物均发生脑水肿，表现行为异常，脑膜及脑组织肿胀、充血，小血管壁外膜及Virchow－Robin间隙增宽，内含大量水肿液，毛细血管基底膜肥厚，神经元固缩、轮廓不清晰或肿胀、坏死，胶质细胞增生、肿胀，硬脑膜表面凹凸不平、水肿明显。

GHBA can protect hypoxia-reoxygenation neurons through upregulating GABA and GABARα1 positive neurons.

GHBA可通过上调GABA和GABARα1阳性神经元的表达水平对缺氧复氧皮层神经元发挥保护作用。

Further analysis of ICL-expressing neurons with residual GABAR-mediated inputs showed that the capacity of benzodiazepine to enhance GABAergic synaptic responses was reduced in ICL-expressing neurons, indicating that they were likely depleted of gamma2 subunit-containing GABAR.

苯二氮卓受体阻断药对EGFP-ICL转染神经元的GABA能诱发电流的增强作用也受到抑制，说明EGFP-ICL的转染是特异性的阻断gamma2亚基的转运上膜。

The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

这种做法不仅鼓励了更多的反应，而且减少跟风的可能性。