查询词典 neurogenic

Exccesive synthetise and release of calcitonin gene-related peptide in trigeminal nerve may promote release of substance P, cause the intense paroxysmal pain and form a local neurogenic inflammation.

我们认为：三叉神经痛的痛支神经过度合成和释放降钙素基因相关肽可能促进了P物质的释放，导致阵发性剧烈疼痛，并在局部形成神经源性炎症。

Exccesive synthetise and release of calcitonin gene-related peptide in trigeminal nerve may promote release of substance P, cause the intense paroxysmal pain and form a local neurogenic inflammation.

我们认为：叉神经痛的痛支神经过度合成和释放降钙素基因相关肽可能促进了P物质的释放，导致阵发性剧烈疼痛，并在局部形成神经源性炎症。

Exccesive synthetise and release of calcitonin gene-related peptide in trigeminal nerve may promote release of SP, cause the intense paroxysmal pain and form a local neurogenic inflammation.

我们认为：三叉神经痛的痛支神经过度合成和释放降钙素基因相关肽可能促进了SP的释放，导致阵发性剧烈疼痛，并在局部形成神经源性炎症。

Conclusion摘要: Exccesive synthetise and release of calcitonin gene-related peptide in trigeminal nerve may promote release of substance P, cause the intense paroxysmal pain and form a local neurogenic inflammation.

结论摘要：我们认为摘要：三叉神经痛的痛支神经过度合成和释放降钙素基因相关肽可能促进了P物质的释放，导致阵发性剧烈疼痛，并在局部形成神经源性炎症。

Exccesive synthetise and release of calcitonin gene-related peptide in trigeminal nerve may promote release of su tance P, cause the inte e paroxysmal pain and form a local neurogenic inflammation.

我们认为：三叉神经痛的痛支神经过度合成和释放降钙素基因相关肽可能促进了P物质的释放，导致阵发性剧烈疼痛，并在局部形成神经源性炎症。

All fatal cases died of serious complications such as neurogenic pulmonary oedema due to EV-71 infection.

所有死亡病例均是由于严重并发症，例如EV-71感染引起的神经源性肺水肿。

The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

这种做法不仅鼓励了更多的反应，而且减少跟风的可能性。