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necrosis相关的网络例句

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与 necrosis 相关的网络例句 [注:此内容来源于网络,仅供参考]

There are many high risk factors of recurrence and death after resection of stage B large intestinal cancer patients.The main factors influencing the long-term survival of stage B large intestinal cancer patients are bowel obstruction,tumor necrosis,and perineurial invasion.

B期大肠癌切除术后复发和死亡的高危因素较多,影响其长期生存的因素主要是肠梗阻、肿瘤坏死和神经囊膜侵润,而与其它因素关系不大。

Results After a median follow-up period of 8.5 years,bowel obstruction was a significant factor related to poor prognosis (log-rank test,P=0.03).Extensive necrosis(P=0.01) and perineurial invasion (P=0.03) were also associated with decreased survival.

结果 平均随访8.5年,肠梗阻与不良的预后有关(圆木行列试验,P=0.03),肿瘤广泛坏死(P=0.01)和神经囊膜浸润(P=0.03)也是降低生存期的因素。

Objective To investigate the value of peripancreatic fluid cytology,serum tumor necrosis factor-alpha and nitric oxide in the diagnosis of acute rejection in pigs pancreas.

目的 研究胰周渗液细胞学检查、血清肿瘤坏死因子和一氧化氮在猪胰腺移植急性排斥反应中的价值。

Objective The later complications of pancreatic and peripancreatic necrosis or infection are very dangerous for patients with severe acute pancreatitis.

目的 重症急性胰腺炎后期出现胰腺及胰周组织坏死、感染,临床上已是非常危重。

Conventional CT showed 44 cases of edematous type, dynamic contrast enhanced CT found 28 cases had different degree of necrosis of the pancreatic tissue, 6 cases were found to have focal hemorrhage, peripancreatic fluid accumulation were found in 32 cases, pseudocyst were found in 3 cases, abscess in 2 cases.

常规平扫显示胰腺水肿44例,动态增强扫描显示28例出现不同程度的胰腺组织坏死,6例检出局灶性出血;胰周液体聚集32例;假性囊肿3例,脓肿2例;伴胆石症15例,胆囊肿大或胆囊壁增厚22例;胆急管扩张12例,胰管扩张5例;胸腔积液11例。

Methods DCs were pre pared from peripherad blood mononuclear induce d with gra-nulocyt e/macrophage colony-stimulating factor and interleukin-4.Apoptosis of he patochlangioma cells were induced with γ-radiation.We design these experiment groups including(1)coculture of DCs and apoptotic cancer cells and T cells,(2)co culture of DCs and necrosis cancer cells and T cells,(3)coculture of DCs and cul tured cancer cell and T cells,(4)cocultu re of DCs and T cell,(5)cocuture of DCs and cultured cancer cell.

用粒-巨噬细胞集落刺激因子加白介素-4(IL-4)从人外周血分化、诱导DCs、γ-射线在体外诱导培养的人胆管癌细胞凋亡,将DCs、T淋巴细胞和凋亡胆管癌细胞共培养,同时设计不同类型肿瘤细胞(坏死胆管癌细胞及培养胆管癌细胞)作对照,7d后,分离、富集DCs、T 淋巴细胞进行免疫应答及肿瘤细胞杀伤试验。

Results After ANIT intoxication,the necrosis of hepatic cell and neutrophil infiltration im periportal region of the liver were observed in liver setions.

结果 S-D大鼠经ANIT染毒后,病理切片显示肝损伤明显,汇管区有大量中性粒细胞浸润。

The liver specimens were immunohistostained by SP method (1)Fas antigen and FasLs specific positive signals were found mainly on the surface or/ and in the cytoplasm of the hepatocytes, these positive hepatocytes were scattered nearby periportal region and hepatic lobule with piecemeal necrosis, the infiltrating lymphocytes were mainly FasL-positive cells.

1Fas、FasL在肝组织肝细胞膜及胞浆均有不同程度表达,其表达阳性细胞主要位于汇管区小叶内及周边碎屑状坏死区,呈弥漫分布,在其周围浸润的淋巴细胞上多为FasL阳性细胞,亦可见到Fas阳性细胞。

For raising the accuracy of the diagnasis ancl treatinemt of ALH We realised of 86 patients with diagnosis of Autoimmune liver disease, strictly defined by the criteria for diagnosis of autoimmune hepatitis or the results of liver histology, and tested its biochemical, serological, histological results. AIH patients were main women female 75 cases, male 11 cases, main in 40 - 60 years old ,AIH characterized by a flare of ALT, AST, elevated immunoglobulin G,γ-immunoglobulin: ALP, IgG, and presence of antinuclear antibody (81.5%); moderate or severe periportal or periseptal lymphocytic piecemeal necrosis.

为提高对自身免疫性肝炎的诊治水平,采用国际自身免疫性肝炎小组新修订的描述性诊断标准和计分系统,对符合诊断的患者进行临床、生化、病理特点分析。86例AIH患者,女75例,男11例,高发年龄为40~60岁,其中Ⅰ型80例(93.020k),Ⅱ型3例(3.49%);Ⅲ型3例(3.49%);肝功能生化检查表现为肝炎样改变;实验室检查主要表现为高γ球蛋白血症,高IgG、ALP、γ-GT及甘油三酯增高,81.5%患者抗核抗体阳性:病理改变以界板性肝炎,浆细胞浸润为主,78%的患者对免疫抑制剂治疗效佳。

objective to search the factors of liver injury via the anit-induced liver injury in rats.methods male sprague-dawley rats,were divided into 4 groups,data were analyzed using a completely randmized factorial anova and linear correlation.results after anit intoxication,the necrosis of hepatic cell and neutrophil infiltration im periportal region of the liver were observed in liver setions.jinshisan can reduce the alt,the tbil.pla2 correlated positively with alt、akp and tbil.conclusion the severity of anit-induced liver injury correlated positively with the pla2.jinshisan can protect against the rat anit-induced liver injury.

目的 拟通过对α-萘异硫氰酸酯诱导大鼠肝损伤发病机理的探讨,对引起肝损伤的因素进行系统的研究。方法选择s-d大鼠,雄性,随机分4组,检测数据以平均值±标准差表示,采用方差分析,相关分析进行统计学处理。结果s-d大鼠经anit染毒后,病理切片显示肝损伤明显,汇管区有大量中性粒细胞浸润。经金石散及地塞米松治疗后,生化指标均有所下降,中药金石散优于地塞米松,血生化指标与pla2活性相关分析显示,呈正相关。结论 anit诱导的肝毒性损伤程度与肝组织匀浆pla2活力呈正相关,应用抑制剂降低pla2活力,肝损伤程度也减轻,中药金石散具有降低pla2活性,减轻肝毒性损伤。

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