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necrosis相关的网络例句

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与 necrosis 相关的网络例句 [注:此内容来源于网络,仅供参考]

Cancer cells showed various levels of focal necrosis in the experimental groups and the cell nuclei underwent swelling, cavitations,and karyopyknosis.

三苯氧胺组和联合组在G0期左侧出现凋亡峰,凋亡率分别为2.3%和13.1%,统计学上有差异(P〈0.05);病理观察对照组肿瘤细胞生长活跃。

ResultsThe glandular cell in the endometrial implant after therapy with middle or high-dose of (10,15 gkg-1d-1) XiaoChaiHu Decoction showed characteristic features of apoptosis which displayed by the cell decreased in size, karyopyknosis , cytoplasmic and nuclear chromatin condensation , density increased and a lot of apoptotic bodies among cell. Whereas some stromal cell displayed degeneration and necrosis. The protein expression of Fas and Caspase-3 in endometriotic tissue of XiaoChaiHu Decoction group was higher than that in the endometrium.

结果中、高剂量小柴胡汤(10,15 gkg-1d-1)治疗后,异位内膜有较多腺上皮细胞出现凋亡的特征,表现为细胞体积变小,核固缩,胞浆和核染色质凝集,密度增高,细胞间凋亡小体,同时间质细胞中可见一些坏死细胞;异位内膜Fas蛋白、Caspase-3蛋白的表达水平明显高于其在位内膜。

Results HE staining showed that sulindac could inhibit the growth of tumor cells, with the morphological presentations as apoptosis and necrosis including pyknosis, karyorrhexis and chromatin crescent formation, etc. By electron microscopy, cells also had apoptotic characteristics including chromatin crescent formation and apoptotic body. Immunohistochemistry and Western-blot assay results showed that 48 hours after sulindac administration, the expression of NF-κB, bcl-2 and bax gradually decreased, while the level of bax protein gradually increased, in a dose dependence manner.

结果 苏木精-伊红染色检测结果显示舒林酸抑制肿瘤细胞生长,肿瘤细胞出现核固缩、核碎裂、染色质边集等凋亡及坏死的形态学表现;电镜下可看到染色质边集、凋亡小体;免疫组化、Western-blot结果显示舒林酸作用48 h后人胃癌SGC-7901细胞NF-κB、bcl-2表达逐渐下降,bax表达逐渐上升,具有剂量依赖性。

Recent research shows hip arthroscopy can provide early diagnosis and treatment hip unexplained pain, such as femoroacetabular impingement,labral tears, chondral lesions, furthmore hip arthroscopy shows good prospects in the treatment avascular necrosis of femoral head, synovial abnormalities, post hip arthroplasty discomfort.

研究表明,髋关节镜对于既往不明原因的髋痛如髋关节撞击征、髋臼盂唇、髋臼软骨损伤可以早期诊断和治疗。此外,髋关节镜在股骨头坏死、股骨头圆韧带损伤、髋关节置换后的应用也展现出极大的应用前景。

Recent research shows hip arthroscopy can provide early diagnosis and treatment hip unexplained pain, such as femoroacetabular impingement, labral team, chondral lesions, furthmore hip arthroscopy shows good prospects in the treatment avascular necrosis of femoral head, synovial abnormalities, post hip arthroplasty discomfort.

研究表明,镜关节镜对于既往不明原因的髋痛如髋关节撞击征、髋臼盂唇、髓臼软骨损伤可以早期诊断和治疗。此外,髋关节镜在股骨头坏死、股骨头圆韧带损伤、髋关节置换后的应用也展现出极大的应用前景。

Methods 89 cases of acute necrosis pancreatitis were retrospectively analyzed and compared with laparotomy.

对89例经手术病理证实或CT复查确诊的急性坏死性胰腺炎进行回顾性分析。

Results According the MRI characteristric of hypoxic\|ischemic encephalopath sequela:(1)The most common findings were white matter injury in 47 cases:①Periventricular leukomalacia in 23 cases;②Selective cerebral neuronal necrosis of basilar node lentiform nucleus and optic colliculus in 9 case...

结果 根据儿童缺血缺氧性脑病后遗症的MRI影像特点,将其分为:(1)脑白质损伤为主:47 例;①脑室周围脑白质脱髓鞘、软化:23 例;②基底节豆状核、视丘神经元选择性损害:9 例;③胼胝体受损变薄:15 例;(2)脑灰质损害为主:18 例;(3)局部或多处脑软化:9 例;(4)广泛脑损害:5 例。

Methods Models of leucocytopenia induced by irradiation were prepared by 60Coγ-ray and by cyclophosphamide injection. The shengbaiyin was administrated orally. The white blood cell counts, reticulocyte count, the number of nucleated bone marrow cells,T cell transformation indices, activity of tunmor necrosis factor and interleukin-6(IL-6) in blood of leukopenia mice on the 5th, 10th, 15st day after chymisty therapeutics were checked.

用60Coγ照射和注射给予环磷酰胺的方法形成骨髓抑制和免疫低下小鼠模型,中药生白饮合剂给予口服,以外周血白细胞、骨髓有核细胞、淋转指数以及细胞肿瘤因子、白细胞介素-6(IL-6)活性为观察指标。

OBJECTIVE:To explore the pathogenesis of IR , we established a rat model of insulin resistance by feeding high-fat diet for 8 weeks and measured serum insulin, leptin, tumor necrosis factor- a concentrations and the expression of the lipogenic transcription factor, sterol regulatory element binding protein-Ic in liver of rats.

目的: 建立胰岛素抵抗的动物模型,进行胰岛素、瘦素、肿瘤坏死因子-α和固醇调控元件结合蛋白-1c等相关激素、基因的测定与分析,探讨IR的发生机制;对产生IR的动物进行药物干预,筛选出有效的中药制剂;进一步比较治疗前后相关激素、基因的变化情况,揭示干预治疗的作用机理。

Results:The etiologic mechanism of steroid-induced necrosis of femoral head is:Disorder of lipometabolism;increasing of intraosseous pressure;Osteoporosis and poisonous action;vasculitis,coagulation before steroid administration and intrava...

结果 :激素性股骨头缺血坏死存在如下发病机理:脂肪代谢紊乱;骨内压增加;骨质疏松及激素的毒性作用;血管炎、前凝血状况及血管内凝血。

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