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necrosis相关的网络例句

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与 necrosis 相关的网络例句 [注:此内容来源于网络,仅供参考]

Expresion of FasL was found mainly in the cytoplasm of liver-infiltrating mononuclear cells,these positive cellswere found particularly at the advancing edges of piecemeal necrosis,bridge necrosis and in the intralobular.

以免疫组化法研究Fas配体在丙型肝炎病人肝组织内的表达情况,发现在碎屑样坏死、桥样坏死及灶性坏死区浸润的单个核细胞有明显的FasL表达,阳性率为41.2%。

Labeled perforin cDNA probe in liver biopsy tissues. RESULTS: Perforin positive cells were seen in the whole intralobular areas, especially in inflammatory areas. The amount of positive cells was different, 71.9% in spotty necrosis areas, 31.0% in focal necrosis areas, 9.2% in portal areas and only 4.8% in non????

结果: Perforin阳性细胞在整个肝小叶内均可检见,有炎性细胞浸润区均有perforin阳性细胞分布,浸润阳性细胞数量因病变部位的不同而有较大差异,慢性丙肝点状坏死区阳性率最高达71.9%,其次是片状坏死区(31.0%),门管区阳性率为9.2%,小叶内非坏死区仅为4.8%。

RESULTS: Perforin positive cells were seen in the whole intralobular areas, especially in inflammatory areas. The amount of positive cells was different, 71.9% in spotty necrosis areas, 31.0% in focal necrosis areas, 9.2% in portal areas and only 4.8% in non necrotic areas. The average amount of perforin cells in the liver lobules of 18 patients had no linear correlation with the ALT level or Knodell's histology activity index.

结果: Perforin阳性细胞在整个肝小叶内均可检见,有炎性细胞浸润区均有perforin阳性细胞分布,浸润阳性细胞数量因病变部位的不同而有较大差异,慢性丙肝点状坏死区阳性率最高达71.9%,其次是片状坏死区(31.0%),门管区阳性率为9.2%,小叶内非坏死区仅为4.8%。

2Compared with PN group,following microcirculatory changes were observed in EEN group in vivo status:interlobular artery and vein structures disappeared,the vessel course was distorted and deformed;intralobular vessels were twisted into nidus,there was patchy bleeding around the vessel combined with necrosis;microcirculatory structures of pancreatic island disappeared and pancreatic tissue necrotized;capillary network had bleeding and necrosis.

2活体状态下观测大鼠胰腺微循环情况显示与PN组相比,接受EEN治疗大鼠急性胰腺炎微循环小叶间动、静脉伴行结构消失,血管走行扭曲变形更加明显;小叶内血管扭曲变形成团,血管周围出现斑片状出血,并伴有坏死;胰岛微循环结构消失,组织坏死;毛细血管网大片状出血、坏死。

In group B, a large amount of inflammatory cell infiltration in liver tissues were noted and hepatic cells showed spotty necrosis and bleeding under the light microscope; the electron microscope showed a significant decrease of mitochondria cristae, the broadness of endoplasmic reticula, hepatic cell necrosis and karyopyknosis.

B组光镜下可见肝组织大量炎性细胞浸润,肝细胞点状坏死及出血;电镜下可见线粒体嵴明显减少,内质网扩张,出现凋亡小体及核固缩。C组上述病理变化明显减轻。

Arsenic poisoning have magma role to paralysis small blood vessels, mercapto Jimei inh ibit the activity of the liver and fat necrosis change Hepatic Lobules Centre, heart, liver, kidney, intestine congestion, epithelial cell necrosis, telangiectasia.

砒霜有原浆毒作用,能麻痹细血管,抑制巯基梅的活性,并使肝脏脂变肝小叶中心坏死,心、肝、肾、肠充血,上皮细胞坏死,毛细血管扩张。

AIM To study the long-term toxicity of modified recombinant human tumor necrosis factor rhTNF-NC in Macaca mulatta compared with recombinant human tumor necrosis factor rhTNF.

目的 研究新型重组人肿瘤坏死因子rhTNF-NC iv对恒河猴的长期毒性,并与重组人肿瘤坏死因子rhTNF作比较。

The main pathology histology change included liver denaturalization, necrosis, spleen giant cell hyperplasia, lymphocyte necrosis, lung and kidney microthrombus form, small intestine edema, microvascular hyperemia, cerebrum neuronophagia and satellitosis.

主要病理组织学变化为:肝变性、坏死,脾巨细胞增生、部分淋巴细胞坏死,肺充血,肾间质血管微血栓形成,小肠绒毛结构被破坏,固有层水肿,间质微血管充血,大脑噬神经现象和卫星化现象等。

The first, many tuberculosises in glandular stomach were found besides liver cell degeneration, pneumoncongestion, epithelium cell degeneration of tubuli tenales, necrosis and kidney hemorrhage, cardiac muscle celldegeneration, heart hemorrhage, cerebrum hemorrhage, satellitosis and neuronophagia, cerebellum purkinje cell necrosis.

发现病例1,肝细胞变性,肺淤血,肾小管上皮细胞变性、坏死,间质出血,心肌纤维变性,间质出血,大脑出血,&卫星化&与&噬神经&现象,小脑浦肯野氏细胞坏死,还可见腺胃有大量的结核结节。

The ani-mal amount of happening hepatic cell necrosis changes in2.00g/kg group was lowered,the degree of pathobiological changes was lightened,and the cell necrosis-changed evaluation had dramatical differences compared with the CCl 4 control group.

结果 0.67g/kg和2.00g/kg剂量组的血清谷丙转氨酶、谷草转氨酶水平降低,与CCl 4 对照组比较(P.05);2.00g/kg剂量组发生肝细胞坏死的动物例数明显减少,病变程度减轻,坏死评分低于CCl 4 对照组(P.01)。

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