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necrosis相关的网络例句

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与 necrosis 相关的网络例句 [注:此内容来源于网络,仅供参考]

The influencing factors of femoral necrosis included the degree of healing activity, the dosage summation of corticosteroids, and length of ictus therapy. The influencing factors of bone density included age, dosage summation, and length of ictus therapy. The influencing factors of the bone fabric and flexibility included the use and length of ictus therapy. Statistics showed that serum IgG was not related with avascular necrosis of femoral head and osteoporosis.

股骨头缺血性坏死的影响因素有后期活动量、糖皮质激素总量和糖皮质激素冲击疗法的持续天数;骨密度的影响因素有年龄、冲击疗法和糖皮质激素总量;骨结构和骨弹性的影响因素有冲击疗法、冲击疗法持续的天数;血清抗体IgG与患者康复期股骨头缺血性坏死、骨质疏松在统计学上无联系。

Pathology and ultrastructure detection revealed cell necrosis and collapse, sever nuclear damage was observed in the death cells. The early characteristics of necrosis such as margination of heterochromatin was also found in some tumor cells. Besides, well differentiated tumor cells, degenerative tumor cells and some lymphocytes were seen.

直形针给药组肿瘤组织早期表现为大片的组织坏死,晚期表现为存活区域的增加,坏死区域与存活区域界限分明,两者间多有移行带;弧形针分段扇形注药者早期、晚期组织均表现为大片坏死,肿瘤间质内可见淋巴细胞浸润和纤维结缔组织增生;各时间段的检测标本中都可见一定数量的凋亡细胞,但比例远远小于坏死的肿瘤细胞。

Tumor necrosis factorαis a pleiotropic factor involved in diverse cellular responses, including inflammation,apoptosis and necrosis.

肿瘤坏死因子TNFα是一个多效性细胞因子,参与调节炎症反应、细胞凋亡和坏死等。

Main clinical symptom of poult gooses were depressed, retardation in reaction, food appetite decreased, alo laxata; and the average death time was 62.6 h Mark processes in dissection were that spleen was intumescenced, gray necrosis appeared; haemorrhage in pancreas, gray necrosis appeared; haemorrhage in intestinal tract, anabrotic; the HA titer of the virus isolated from these samples could reached as high as 2^8, and had the chicken red blood cell agglutination and lys agglutination ability.

雏鹅平均死亡时间为62.6 h。特征性剖检病变为脾脏肿大,有灰白色坏死点;胰脏出血,有灰白色坏死点;肠道出血,溃疡;病料分离培养的病毒液HA价高达28,能凝集鸡红细胞,并出现解凝现象。

BACKGROUND: According to traditional theory of the recovery after acutetubular necrosis, reconstruction of renal tubule is dependent on residual epithelialcells next to the necrosis region. Undergoing the process of dedifferentiation,proliferation and redifferentiation, these cells can reestablish the injured tubule and itsfunction.

背景:传统的急性肾小管坏死修复理论认为,肾小管的重建依赖于坏死区周围存活的肾小管上皮细胞,这些细胞经过去分化、增殖和分化成熟等过程修复坏死的肾小管,重建其组织结构和功能。

Day puppies were inoculated by abdominal cavity and nasal cavity with CDV,canine distemper patho-sample of duplicating meet with success and through differentdrug of treatment, specific antibody, dexamethasone, causing death by letting. Theresults of pathohistology observation on 14 PID: Control group, the hearts showedcardiac muscle hemorrhage and necrosis, the livers showed a large area of hepatictissue hyperemia, hemorrhage and necrosis, the spleens showed the quantity oflymphocyte grow downwards, the blood vessels of lung congested, congestion andhaemorrhage were serous fluid exudation, the kidneys showed heavy haemorrhageand hyperemia, the cerebrum showed the number of microglial cells of cerebrumincreased.

通过特异性抗体和地塞米松、特异性抗体、地塞米松三种不同的方法治疗后,放血致死,取材作病理切片,观察各组病理组织学变化,结果为:感染后14d阳性对照组的心脏表现为心肌出血、坏死;肝脏表现为肝组织大面积充血、出血坏死;脾脏表现淋巴细胞数量大量减少;肺脏表现为肺泡结构破坏,有坏死灶;肾脏表现为弥漫性的充血出血;大脑表现为胶质细胞增多。

OBJECTIVE: To evaluate the therapeutic effect of liquid-electric extracorporeal shock wave on traumatic avascular necrosis of talus, and to explore new treatments of traumatic avascular necrosis of talus.

目的:应用液电式体外冲击波治疗创伤性距骨缺血性坏死并分析其治疗效果,探索创伤性距骨坏死新的治疗方法。

Tobacco disease caused by PVY is known as yellow spot necrosis, vein strip disease or vein necrosis.

如何防治与解决PVY病毒病已经是当前烟草生产面临的重要问题。

N perifocal tissue intracerebral hemorrhage there were rarefaction neuron,cell spaces augmentation,cell diminution,distinct demarcation of cell membrane and surrounding,and we discovered a lot of degeneration and necrosis nerve cells,cell body collapsed,pycnosis anachromasis,nucleoli disappeared. In EPO group we discovered that center area of hemorrhage shinked,nerve cells of degeneration and necrosis decreased in perifocal tissue,majority cells morphous were normal and pathological changes were light.

CH对照组在术后皮层出血中心无神经元,仅见少量胶质细胞,细胞间质空泡样改变;出血边缘区神经元稀疏,细胞间隙增大,细胞缩小,胞膜与周围分界清楚,并可见大量变性及坏死的神经细胞,表现为胞体皱缩,核固缩深染,核仁消失。rhEPO治疗组出血中心区变小,边缘区神经细胞变性坏死减少,多数存活细胞形态相对正常,病变较轻。

Results Denatured necrosis occurred on the surface of hypothalamic periosteum with no cells and there was a large amount of starchy bodies and hydrops in foci with angiectasis of choroid plexus and necrosis and calcification in tunicaintima of vesicles,and mitochondria in the nerve synapse disappeared and part of anteroposterior membranes ...

结果 慢性SZ患者下丘脑部室管膜表面变性坏死、无细胞,病灶中有大量淀粉样小体和水肿,脉络丛血管扩张,管壁内膜有坏死和钙化;神经突触内大部分突触小泡和线粒体消失,部分前后膜模糊不清。

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