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necrosis相关的网络例句

查询词典 necrosis

与 necrosis 相关的网络例句 [注:此内容来源于网络,仅供参考]

RESULTS: A total of 372 patients treated with anti-tumor necrosis factor therapies met the inclusion criteria.

结果:共有372位接受抗肿瘤坏死因子治疗的患者满足入选标准。

The experiment provides a basis for treatment of the necrosis of the femoral head.

为股骨头坏死这种新的治疗方法提供实验依据。

Objective: To explore the clinical significance of tumor necrosis factor α and C-reactive protein in patients with unstable angina.

目的:探讨检测不稳定型心绞痛患者血清肿瘤坏死因子和C反应蛋白的临床意义。

AIM: To investigate the effective diagnosis and treatment of acute retinal necrosis.

目的:探讨急性视网膜坏死综合征临床诊断、治疗的有效方法。

Methods: One hundred and twenty-six patients with UAP were randomly divided into two groups. The control group was treated with Xueshuantong injection, and the treatment group was treated with propylgallate. The clinical therapeutic effects were observed and the blood parameters such as C reactive protein, interleukin-6(IL-6) and tumor necrosis factor-α were assayed by rate nephelometry and enzyme linked immunoadsorbent assay.

选择126临床确诊为UAP的患者,随机分为血栓通注射液对照组和注射用棓丙酯治疗组,进行临床疗效观察,并分别采用速率散射比浊法及酶联免疫双抗夹心法检测C反应蛋白、白细胞介素-6(IL-6)及肿瘤坏死因子-α水平,观察其治疗前后的变化情况。

It was considered an effective and ideal model of PM.2 uPA,PAI-1 and TNF-αwere all involved in the pathogenesis of PM and participated in the process of the degeneration,apoptosis and necrosis of the diseased muscle.3 MMP-9 and TIMP-2 immunoreactivity were positive in the capillary,venous wall,arteriola endotheliocyte and neurilemma in the normal group skeletal muscle,but more intensive in EAM.They may aggravate the pathological changes of the muscle in human PM.

结论1兔纯化的肌球蛋白加含卡介苗的完全弗氏佐剂可仿制出人类多发性肌炎的理想动物模型。2 UPA、PAI-1和TNF-α参与多发性肌炎的肌纤维坏变过程。3 MMP-9和TIMP-2在正常肌肉组织中的毛细血管、静脉管壁、动脉肉皮细胞和神经鞘膜表达,在多发性肌炎中,参与加重肌纤维的坏变过程。

Results:It was found that in experimental group,retinal neurofibril were disorder,the ganglion cell,the cells in inner and outer nuclear layer presented nibble like lysis,necrosis and lose.gradually at 1,3,5 day after moc...

结果:接种后1、3、5天,对照组未见异常改变,绿脓杆菌接种组视网膜神经纤维排列紊乱、消失,神经节细胞、内核层细胞及外核层细胞从内向外呈蚕蚀样溶解或坏死脱落。

Necrosis was observed in some of the 30 Gy-3-month-period group. Different levels of morphological changes in blood vessels,neuroglia cells and neurons were found in rats of all other groups.

在30 Gy照射后3个月组中有坏死灶的存在,而其他组别中可以看到血管、胶质细胞和神经元等组织的形态学异常。

The results indicated that EMP can promote necrosis and apoptosis of hippocampal neurons,induce morphological changes in blood vessels, neuroglia cells and neurons at early stage, which may be the possible injury mechanism in rat brain induced by EMP.

表明大鼠大脑受电磁脉冲辐照后早期海马区可发生神经细胞坏死和凋亡,以及各组织成分的病理形态学改变,上述变化可能与电磁脉冲致细胞DNA损伤有关。

Seven days after operation, some pathological changes were found in the pigs of treatment group: mild congestion in hepatic tissue, with inflammatory cell infiltrated around blood vessels, liver cells showed spotty necrosis, and neutrophile infiltration.

术后第7天,处理组肝组织轻度淤血,血管周围有少量炎性细胞浸润,肝细胞点状坏死,中性粒细胞浸润。

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