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necrosis相关的网络例句

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与 necrosis 相关的网络例句 [注:此内容来源于网络,仅供参考]

It was found that in thalidomide treated patients serum tumor necrosis factor a and interferon-γ gradually decreased with increasing IFNγ/IL-4 and IFNγ/IL-10 ratios when compared with control group. Meanwhile the clinical symptoms in thalidomide group improved faster, and the improvement of liver function was comparable with control group.

结果发现GHB患者血清TNFα、IFNγ基础值显著高于健康人水平,经沙利度胺治疗后血清TNFα和IFNγ逐渐下降,IFNγ/IL-4及IFNγ/IL-10比值有升高倾向,而对照组变化不大;沙利度胺组临床症状改善较快,退黄时间和转氨酶下降速度与对照组相近。

Methods Mimicry and speculation on possible displaced amino acids in proper positions of peptide increased the biological effects of TNFR blocking peptide by compote modeling. The original peptide and its mutant were synthesized. The competitively inhibitory effects on green fluorescence protein-tumor necrosis factor fusion protein with TNFR were compared between original peptide and its mutant (receptor competitively inhibitory binding assays). The inhibitory effects on cytotoxicity were compared between original peptide and its mutant.

通过计算机模建,模拟并推测提高TNFR封闭肽生物学效应需置换的位点和氨基酸;合成原封闭肽和突变肽;受体竞争抑制结合实验比较原肽与突变肽对绿色荧光蛋白-肿瘤坏死因子融合蛋白结合TNFR的竞争抑制效应;细胞毒作用抑制实验比较原肽与突变肽对TNFR的封闭效应。

The study of the histopathology showed that the haemolymph in the disease crab decreased sharply, and was replaced by large amount of parasites, which was observed in muscle, heart and hepatopancreas from the disease crab. The pathological characteristics mainly appeared as cells swelling, necrosis, nucleus condensation and collaspe.

组织病理学观察发现,病蟹血淋巴急剧下降,代之以大量的寄生原虫,该寄生虫在病蟹的肝胰腺、鳃、心脏、肌肉等部位大量侵袭,并引起这些组织发生以坏死为主的变质性病变,其病理特征主要表现为:细胞肿胀、变性、坏死,某些细胞的细胞核固缩、碎裂或崩解。

A remarkable histologic observation is the presence of tumour necrosis, which was seen as both single cells and large confluent areas.

一项显著的组织观察证据是肿瘤细胞坏死,包括单个细胞和大的 confluent 区域。

The upregulation of HO1 expression induced by CoPP can inhibit the necrosis of the myocardial cells and subsequently alleviate the myocardial ischemia/reperfusion injury. One of its main mechanisms may be related with antioxygen free radicals.

CoPP诱导的HO1过表达可以抑制心肌缺血-再灌注损伤后的细胞坏死,从而减轻心肌的再灌注损伤,其主要机制与抗氧自由基有关。

The pathological changes of the soft tissues around coxa are the early dangerous signals of femoral head necrosis, thus should be paid sufficient attention.

髋周软组织病理改变是股骨头坏死的早期危险信号,应引起足够重视。

Results:The avascular necrosis of the femoral head was related with the following effects:case history including fracture of the lower limb,coxitis,rheumatoid arthritis,hyperlipidemia and diabetes

果危险度分析表明,可能与股骨头坏死危险性有关的因素(OR及9%CI)病史有下肢骨折、髋关节炎和类风湿关节炎、高脂血症、糖尿病史

In some cases there is severe angitis and fibrinoid necrosis of the vascular wall with perivascular cuffing by lymphocytes and plasma cells. Trophozoites and cysts were found within the perivascular space.

有的病例可见显著的血管炎,病变血管壁可见纤维素样坏死,血管周围可见淋巴细胞和浆细胞呈套袖样浸润及棘阿米巴滋养体和包囊。

Objective to investigate the effect of mifepristone on the expression of tumor necrosis factor-α and transforming growth factor-βin decidua of early pregnancy.

目的 探讨米非司酮对胚胎生长及蜕膜肿瘤坏死因子-α和转化生长因子-β表达的影响。

Results Punctiform erosion, hemorrhage were observed by nake eyes in each groups. Epithelium cellular necrosis, defluxion, neutrophil being dominant in propria lamina and intact glandular were observed with microscopy. The erosion became severe with the successive stimulate of ethanol. There is no difference between distillate spirit and sodium chloride rats in Guth in 7th day, but Guth in distillate spirit rats was higher than that in sodium chloride rats in 30th day. PGE2, TGF-α contents in gastric mucosa of treated group increased significangly compared with control group. PGE2 showed no difference between each group in 7th and 30th day; TGF-α in gastric mucosa of distillate spirit and sodium chloride rats decreased after 1 month.

结果 茅台组、乙醇组、生理盐水组胃黏膜均肉眼可见程度不同的点状糜烂、出血;光镜下黏膜部分上皮坏死脱落,固有层以中性粒细胞为主炎性细胞浸润,腺体结构基本完整,病变随时间延长有所加重,Guth积分1周茅台组与生理盐水组差异无统计学意义,1月两者相比差异有统计学意义;茅台组、乙醇组胃黏膜组织内PGE2水平与生理盐水组相比明显增高,同时TGF-α表达增强;但PGE2水平各组间1周与1月相比差异无统计学意义;TGF-α表达强度在茅台组、生理盐水组1月后逐渐降低,差异有统计学意义。

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