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myocardium相关的网络例句

查询词典 myocardium

与 myocardium 相关的网络例句 [注:此内容来源于网络,仅供参考]

Atrophy of myocardium, expansion of mitochondria,destruction of myofibril and interstitial fibrosis are the main morphological changes of diabetic cardiomyopathy, and lipid peroxidation and NO may be involved in it.

糖尿病大鼠心肌病变主要为心肌萎缩、线粒体扩张及肌原纤维破坏,间质纤维增生,脂质过氧化作用及NO所致的损伤可能参与其中。

At ED10.5 tiny blood vessels in the myocardium first appeared and the myofibril, which was weakly stained compared to the cardiac muscle cell, first appeared in the ventricle.

ED12.5时心肌小梁结构第一次在心室出现,ED14.5时增厚,而在心房少见心肌小梁。

Cardiac myosin is the essential material for the structure and function of myocardium, it is also the major factor which determine the condition of cardiac function.

心脏肌球蛋白是心肌结构和功能的主要物质基础,是决定心脏功能状态的重要因素。

Nighty rats were randomly divided into the groups of control, myocardial infarction and doxycycline, which were given orally 48 hours before and 48 hours after MI with 30mg/Kg per day. Protein and mRNA extraction was done on left ventricular samples containing scar and myocardium together. Samples were assayed from 1 day to 4 weeks post-Mi. The activity of MMPs was measured by zymography and collagen amount by the method of chloramine T, the ratio of II III collagen was assessed by immunohistochemical stain. Protein and mRNA of MMP-2, 9, TIMP-1 were determined by Western-Blot and RT-PCR.

分为对照组、心梗组和治疗组(D组,术前两天至术后两天,口服Doxycycline,30mg/kg/天),分别于术后第一天、术后一周、术后两周、术后四周取心肌组织,采用免疫组化测定胶原含量和Ⅰ/Ⅲ胶原比例,酶谱法测定心梗后MMP-2,9活性蛋白的表达规律,Western Blotting进一步确定酶谱法中所消化条带蛋白的属性,逆转录-聚合链反应法测定心梗后MMP-2,9和TIMP-1的mRNA的变化规律,运用Acuson Sequoia 512超声心动图机分别测定心梗组、治疗组在两周和四周时左室前壁、后壁厚度以及左心室舒张末内径和左室射血分数。

The MMP-1 expression was increased in the myocardium of systolic HF patients compared with diastolic HF patients and normotensive subjects.

这个MMP-1表达增加了心肌收缩性心力衰竭患者的左室舒张相比心力衰竭患者的正常血压和主题。

Rats with CHF were divided randomly into two groups, one group was injected dexamethasone(1mg /kg) intramuscularly at the first and forth day respectively,the other group was injected equi-volume saline likewise, and observing the changes of symptoms of rats with CHF; measuring rats hemodynamics index including blood pressure, heart rate and left ventricular end-diastolic pressure 4 days later. Part 2: To measure every group rats myocardialα1、β1、β2、β3-AR density by immunohistochemical method ,and to understand the AR density changes .Taking the first part rats'myocardium tissues for testing myocardialα1、β1、β2、β3-AR density and 5 normal rats for normo-contrast group and taking gray scale of slice as AR density. Part 3: The study enrolled 35 patients with DCHF. Patients were included if they had orthopnea and refractory edema due to acute DCHF that was severe and had taken more 1 week hospitalization and intravenous therapy in addition to diuretics.

观察心衰大鼠的症状,4天后对两组大鼠进行血流动力学检测,检测指标包括血压、心率和左室舒张末压;第二部分:取第一部分实验动物大鼠的心肌组织,并取5只正常大鼠的心肌组织作为正常对照,用免疫组化法检测大鼠心衰前后及糖皮质激素治疗前后大鼠心肌组织肾上腺素能受体α1、β1、β2、β3受体的变化,取镜下切片的灰度值反应受体的密度;第三部分:研究对象为临床难治性心衰病人35例,病人的选择标准包括由于急性失代偿的心力衰竭出现的端坐呼吸,难治性水肿,患者病情严重,住院超过1周,包括利尿剂之外药物的静脉治疗。

Pathological examination early after the ablation procedure showed coagulatie necrosis of both the myocardium and the septal perforator arteries.

修改:消融术后早期进行的病理检查发现心肌和冠状动脉的室间隔支都有凝固性坏死。

Damage to the heart more common cardiac damage, can occur pericardium, myocardium or cardiac conduction system disease.

心脏损害心脏损害较为常见,可以出现心包、心肌或心脏传导系统的病变。

The concentration of plasma renin active, angiotensin Ⅱ and NO were measured before operation and after operation 1,4,7,10,15,21 days. The histopathological changes of pericardium, myocardium, lung and liver were observed in the adopted specimens.

分别于术前和术后 1、4、7、10、15、2 1天采血测定血浆肾素活性、血管紧张素和 NO的浓度,观察心包、心肌、肺和肝的病理改变。

Results Cvb-D obviously lightened isoprenaline-induced myocardial pathological changes such as turbulence of myocardial fiber, plasmolysis of necrotic cell, inflammatory cell infiltration of necrotic interstitial region. Cvb-D remarkably inhibited myocardium cell apoptosis of myocardial ischemia rats caused by isoprenaline.

结果 Cvb-D可显著减轻Iso诱发的心肌缺血致心肌肌纤维排列紊乱,坏死细胞胞泉溶解,坏死间质区炎性细胞浸润等病理改变;Cvb-D可显著抑制Iso诱发的心肌缺血性模型大鼠心肌细胞凋亡。

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