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myasthenic相关的网络例句

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ABSTRACT : Objective The phenotypes of the T、B lymphocyte subpopulation in myasthenic thymus were studied and the correlation of the peripheral blood and thymus were analyzed.

目的 研究重症肌无力(myasthenia gravis,MG)患者胸腺的T、B淋巴细胞亚群表型,并分析与其外周血的相关性。

Results:There were 12 cases of peripheral sensory neuropathy,12 cases of myasthenic syndrome,6 cases of spinal cord compression,4 cases of brain stem encephalitis,3 cases of subacute cerebellar degeneration.3 cases were diagnosed in 1 week after appearing neurological symptoms,and 3 cases were diagnosed in 1 month,the other 16 cases were diagnosed after many months or years while the neuropathy was existing.

结果:癌性感觉性周围神经病12例,肌无力综合征8例,脊髓压迫症6例,脑干脑炎4例,亚急性小脑变性3例。3例在出现神经系统症状后1周内确诊,3例在1月内确诊,其余16例均在神经病变持续存在数月至数年后经多次检查确诊为肿瘤。

The congenital myasthenic syndromes are a heterogeneous group of hereditary disorders affecting neuromuscular transmission and most present at birth or soon after.

先天性肌无力综合症不同于遗传性肌病,其影响神经肌肉传递并且常常在出生后或出生后不久发病。

These up-regulations were observed in immune response effector cell s, namely, lymph node cell s, and in the target organ of the autoimmune attack, the muscle of myasthenic rats, and were significantly reduced after suppression of EAMG by mucosal tolerance induction with an AChR fragment.

这些因子的上调见于免疫效应细胞和免疫攻击的靶器官,但在乙酰胆碱受体片段粘膜耐受的EAMG小鼠中却表达下降。

These up-regulations were observed in immune response effector cells, namely, lymph node cells, and in the target organ of the autoimmune attack, the muscle of myasthenic rats, and were significantly reduced after suppression of EAMG by mucosal tolerance induction with an AChR fragment.

这些因子的上调见于免疫效应细胞和免疫攻击的靶器官,但在乙酰胆碱受体片段粘膜耐受的EAMG小鼠中却表达下降。

There are abnormal phenotypes of AChR-specific-responding T、B cell subpopulation in the myasthenic thymus, especially the activated Th cells. In addition,CD5- B cells might be related to the pathogenesis of the MG.

MG患者胸腺存在着异常的AChR特异应答性T、B淋巴细胞亚群表型,尤其以活化的Th细胞为著;CD5-B细胞的产生可能与MG密切相关。

These up-regulations were obsered in immune response effector cells, namely, lymph node cells, and in the target organ of the autoimmune attack, the muscle of myasthenic rats, and were significantly reduced after suppression of EAMG by mucosal tolerance induction with an AChR fragment.

这些因子的上调见于免疫效应细胞和免疫攻击的靶器官,但在乙酰胆碱受体片段粘膜耐受的EAMG小鼠中却表达下降。

The IgG purified from MG serum was administrated to the rats cerebroventricular system to establish the myasthenic model of CNS dysfunction prior to detection of level of TNF-alpha in brain, thymus and blood by radioimmunoassay .

将MG患者血中提取的IgG注入大鼠脑室系统,建立CNS损害的MG大鼠模型,然后用放免法测定其脑、胸腺、周围血中TNF-α的水平及其变化。

In the article, we took part sample out of 158 patients to exam the expression level of Bcl-2 and Fas in myasthenic thymus tissue, and to find the role of which in whole pathogenesis. we also try to find the role of neopterin and sIL-2R through examing the concentration of neopterin and sIL-2R in peripheral blood of 26 MG patients. Material and methods1.select of case: to choose 158 patients who wear followed up from 1988 to 2002 after operation. All patients were diagnosed as MG, and treated with same medical cure during period of preoperation and post operation. 2.Collectionofdata:.To collect all data through designing a uniform follow-up table for thymectomized patients ,and to analysis the effect of factors including age, gender, disease duration of preoperation, clinic types and pathological types to prognosis at three follow-up period?

本文拟在对全组病例的远期预后进行流行病学统计分析的基础上,通过抽取一定量样本,研究重症肌无力患者胸腺组织中伽 12、Fas的异常表达及生发中心的形成在重症肌无力发病机制中可能的作用,以及它们对胸腺切除术后远期预后的影响;与之相似,通过检测部分样本外周血清中新碟吟和可溶性IL-2受体的浓度及其与正常对照组的差异,以期推测两者在重症肌无力起病及病情进展中的作用和意义,并分析其对胸腺切除术后远期预后的影响。

All patients were treated by anticholinesterase,some patients were also treated by corticosteroid therapy,immunosuppressant or both.After24months follow up,all cases were relieved,and no myasthenic crisis occured.

全部病例用胆碱酯酶抑制剂治疗,部分加用肾上腺糖皮质激素或免疫抑制剂,病情均得到缓解,随诊至今无危象发生。

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