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muscle cell相关的网络例句

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与 muscle cell 相关的网络例句 [注:此内容来源于网络,仅供参考]

Its reasonable for us to propose the following hypothesis: in the early period CAN, MMPs/TIMPs system is a pivotal figure for mononuclear cell infiltration and vascular smooth muscle cell proliferation and migration through its effects on matrix remodeling, thusinfluences the late survival of allograft.

我们根据这些研究结果提出以下假说:MMPs/TIMPs系统在慢性移植肾病早期通过促进细胞外基质重新排列而促进单个核细胞浸润和血管平滑肌细胞移行,从而影响移植物的长期存活。

The result showed that there was no expression in the normal control group and an apparent expression of ERK1/2 and Elk in the asthma group. A rather dense expressing of ERK1/2 was at bronchiole and mucous membrane, sub mucous membrane, smooth muscle, cytoplasm and nuclei of the out layer of the smooth muscle cell and an expression of positive fiber at submucous membrane.

结果发现正常肺内没有发现ERK1/2和Elk的表达,而哮喘时肺内有明显的ERK1/2和Elk表达,ERK1/2较密集表达在小支气管和细支气管的粘膜层、粘膜下层、平滑肌层和平滑肌外层细胞的胞浆和胞核中,也可见粘膜下层有阳性纤维表达。

Using immunohistochemical S-P method, the expression of iNOS was found in the vascular smooth muscle cell and the endothelium cell of the lung in SAP group.

奥曲肽治疗组肺内NO水平显著降低,(P.05)肺组织病理学改变减轻。

Results: The tHey and blood fat level in blood plasma and expression of MCP-1mRNAof aorta on model group is obviously higher than that of control group (P<0.05);the result of immunohistochemistry shows that the expression of VCAM-1 and MMP-2enhanced, Bcl-2 is lower; aorta slice shows endarterium discontinuation, strippingpartly, smooth muscle cell arrange puffly and chaotic, infiltration of lipoid ofdifferent degree are observed in aorta wall, pathological changes suffusion, morexanthoma cell, plaque protrude lumens. The they and blood fat level in blood plasmaand expression of MCP-lmRNA, VCAM-1, MMP-2 of aorta on intervention group aremuch lower than that of model group (P<0.05); Bcl-2 of intervention group isstrengthened, and the pathomorphology and immunohistochemistry of aorta showpathological changes are slighter than model group.

结果:模型组,血浆中同型半胱氨酸浓度、血脂水平、主动脉MCP-1mRNA表达显著高于正常对照组(P<0.05),免疫组化示主动脉内皮细胞VCAM-1、MMP-2表达增强,Bcl-2表达降低;主动脉切片发现动脉内膜不连续,部分剥脱,平滑肌细胞排列疏松、紊乱,主动脉壁均见到程度不同的脂质浸润,其病变弥漫,泡沫细胞较多,可见斑块突出管腔;干预组,血浆中同型半胱氨酸含量、血脂水平显著低于对照组和模型组(P<0.05),主动脉MCP-1mRNA、VCAM-1、MMP-2表达显著弱于对照组和模型组(P<0.05),Bcl-2表达增强(P<0.05),主动脉病理形态学和免疫组织化学检测示病变轻于模型组。

130 rats were randomly divided into five groups: 5F electroacupuncture treatment group, 50Hz electroacupuncture treatment group,100Hz electroacupuncture treatment group, the model group and the control group. 4mm defect was produced in the left sciatic nerve of each rat and silicon nerve guards were used to bridge the nerve defect not including the rats of the control group. After 10 weeks electroacupuncture treatment, gross observation, histological examination of the sciatic nerve were carried with method of HE stainning and S-100 immunhistochemical staining, Sevier-Munger stainnning. The histomorphometric examination of the triceps muscles were observed with measuring width muscle cell and the wet weight, and measuring the content of the heparin and the lactic aid of the triceps muscle with biochemistry methods.

实验方法:将130只Wistar大鼠随机分为五组:正常组、造模组、电针5Hz组、电针50Hz组、电针100Hz组,除正常组外,其余各组手术切取坐骨神经4mm后,做坐骨神经硅胶管桥接术,造成周围神经损伤模型,取患侧足三里、三阴交、环跳穴,术后第四天开始电针,每两天一次,每次10分钟,治疗10周后处死动物,取出硅胶管内新生的坐骨神经,纵切片,HE染色、S100免疫组化染色、Sevier-Munger银染,观察坐骨神经纤维再生情况,以健侧同段坐骨神经做对照;取患侧脖肠肌称取湿重,HE染色,观察腓肠肌肌细胞直径,生化测量腓肠肌肌糖元、乳酸。

The proliferation of thyroarytenoid muscle and lateral cricoarytenoid muscle cell of dog remained at high level in 5 months after denervation and then slowed down and atrophy of muscles occurred 6 months after denervation.

甲杓肌及环杓侧肌失神经支配后5个月内保持高增殖水平,以后均降低,失神经支配6个月后肌萎缩开始明显。

Object: The project is on the basis of chronic alcoholic myopathy model of rat, to dynamically observe the change of NF-κB and MMP-2, MMP-9 in muscle cell, observe the function in the mechanism of alcohol myopathy, move forward a single step to explain the pathogenesy of this disease from gene and protein leve. We also provide a helpful platform for other metabolic myopathy. Methods:(1) 80 health, male, Sprague-Dawley rats(weight 180~200g), were chosen to sep up the CAM rat myopathy model ,to observe the rat bodyweight, the change of praxiology and the patho-change of muscle tissue.

目的:本实验旨在建立大鼠慢性酒精中毒性肌病模型的基础上,动态观察NF-κB及MMP-2、MMP-9在酒精中毒性肌病发展中的表达、变化情况,探讨其在慢性酒精中毒性肌病发病过程中的作用,从基因和蛋白水平进一步阐述该病发病机制,为慢性酒精中毒性肌病的临床防治提供理论依据,同时对于其他代谢性肌病的研究也提供一个平台。

The second, many coccus in kidney, heart and pancreas were found besides liver cell degeneration, congestion of liver, epithelium cell degeneration, necrosis of tubuli tenales, kidney hemorrhage, cardiac muscle cell degeneration, heart hemorrhage.

病例2,肝细胞变性,肺淤血,肾小管上皮细胞变性、坏死,间质出血,心肌纤维变性,间质出血,肾、心、胰脏均可见到散在分布的球菌。

Some cell dropped into the cavity and became free. Thrombosis or part organization could be seen. The internal elastic layer became thin, disappear or broken. In internal and middle layer existed fibroblasts, fibrocytes and collagen. Some of the wall indicated hyaline change, soomth muscle cell decreased greatly. The massive inflammatory cells invaded the middle and external layer. There were many foam cells in the capsule tissue. Cytoplasm was filled with fatty tissue and cholesterol. some cavities were full of thrombosis. Some thrombosis was fibrosis, the bottom was organization. The surface of the thrombosis existed red blood cell and librae.(2)Elatic fibrila staining: the internal elastic menbrane almost completely disappeared, the intact internal elastic menbran could be seen in the new small vessels.

动脉瘤管壁厚薄明显不均,全层或局部区域显著变薄向外膨出,内皮细胞空泡变性或坏死脱落,部分内皮细胞剥离并突入管腔成游离状,可见血栓形成及部分血栓机化;内弹力板变薄、消失或突然中断;在内膜及中膜部位主要为纤维母细胞、纤维细胞和大片胶原;部分动脉瘤壁呈均质状玻璃样变,平滑肌细胞明显减少;中膜和外膜可见大量的炎性细胞浸润;瘤壁组织有纤维母细胞、纤维细胞、大片胶原成分及较多泡沫细胞,胞浆内充满脂类物质及胆固醇结晶;部分动脉瘤腔内充满血栓,有的血拴已经纤维化,血栓基部机化,血栓表面有红细胞和纤维素。

Smooth Muscle Cell Medium is a complete medium designed for optimal growth of normal human smooth muscle cells in vitro .

平滑肌细胞培养基是专门为正常人类平滑肌细胞体外培养设计的最适于其生长的培养基。

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