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muscle cell相关的网络例句

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与 muscle cell 相关的网络例句 [注:此内容来源于网络,仅供参考]

Between P14 and P21,CIAPIN1 immunoreaction in the brain,heart and liverbecame much lower. However,between P21 and P28,CIAPIN1 immunoreactionin the heart,brain,liver and skeletal muscle became much lower,while with thekidney development,CIAPIN1 immunoreaction in the kidney became higher. Invarious tissues from adult mouse,CIAPIN1 immunoreaction could be seen incardiac muscle cell,brain,hepatocyte,epithelium of renal tubule,skeletal muscle,lung tissue,gastric mucosa and gland,acinus lienalis.2. Distribution of CIAPIN1 in normal fetal and adult human tissuesTo reveal the possible physiological role of CIAPIN1,we examined theexpression and distribution of CIAPIN1 in fetal and adult human tissues usingimmunohistochemistry. We found that CIAPIN1 was ubiquitously distributed infetal and adult tissues,and was localized in both the cytoplasm and the nucleus.

然而,在3个月大的成年鼠中,CIAPIN1阳性反应物在心、脑、肝和肾小管中的表达强度要低于P28小鼠;但CIAPIN1阳性反应物在成年鼠骨骼肌中较P28小鼠高。2、CIAPIN1蛋白在人5个月胚胎及成人多器官组织内的表达在人5月胚胎多器官组织中,CIAPIN1阳性反应物见于心脏、胆囊单层柱状上皮和粘膜、结肠粘膜、小肠粘膜和绒毛、肝脏、直肠腺体、胃粘膜、肾上腺束状带、甲状腺滤泡、脾索、胸腺小叶间隔、皮肤真皮层和汗腺、睾丸白膜和间质、脑组织内神经元和神经胶质、肺小支气管和肺泡、骨骼肌、肾脏皮髓质和肾小管、子宫内膜、胰腺腺泡和胰岛、卵巢、输卵管粘膜等绝大多数组织细胞。

Aim To explore the effect of auricularia auricula polysaccharide on the proliferation of vascular smooth muscle cells in the formation of atherosclerosis.

在动脉粥样硬化(atherosclerosis,As)发生和发展过程中,血管平滑肌细胞(smooth muscle cell,SMC)的增殖和向内膜下迁移起重要作用。

The recombinant eukaryotic expression vector pcDNA3.1-Myc-His B/tPA can stable express tPAcDNA in transfected hUVEC cell strains and can secret compt tPA protein. Experiment III The Effects of Transfected tPA Gene on Vascular Smooth Muscle Cells Proliferation and Migration in VitroObjective To investigate the effects of transfected tPA gene on vascular smooth muscle cell proliferation and migration in vitro and to provide a basis for further study on preventing vein graft restenosis.

结论构建的pcDNA3.1-Myc-HisB/tPA基因新型真核表达载体,能在HUVEC中表达,其表达具有高拷贝、稳定性好的特点,获得的表达蛋白活性较高,提示本载体和稳定表达tPA基因的人脐静脉内皮株能够作为基因治疗工具用于哺乳动物的实验研究。

FCM outcome show that osteosarcoma cell DNA become heteroploid and DNA content is abnormal. Compare with normal muscle cell, the G1 phase cell number of osteosarcoma decrease, however the S phase cell number increase significantly.

流式细胞术检测结果显示:骨肉瘤细胞DNA均呈异倍体改变,DNA含量异常,与正常肌肉细胞相比,骨肉瘤G1期细胞数减少,而S期细胞数显著增多。

Coenzyme Q10 widely exists in each kind of histiocyte in particular in mitochondrion of the cardic muscle cell, is one kind of important active enzyme in cardic muscle cell, react as electron transfer system in breath-chain of mitochondrion, and is a important part in breath-chain, participates in ATP synthesis.

辅酶Q_(10)广泛存在于各种组织细胞尤其是心肌细胞中的线粒体中,是组织细胞内具有重要作用的活性酶,在细胞线粒体呼吸链即电子传递系统中起递氢体作用,是细胞呼吸链的重要组成部分,参与ATP的合成。

Transplanting cells include skeletal muscle celK smooth muscle cell n cardiac muscle cell and others cell.

此类细胞包括骨骼肌成肌细胞、平滑肌细胞、心肌细胞和干细胞。

Wenxin capsule can promote endolethial cells releasing CO and synthesing 〓, enhance activity of HO, induce angiectasis through activating guanylic cyclase and adenglate cyclate of intracellar of vessel smooth muscle cell to enhance content of intracellar cGMP and AMP. 3. Wenxin capsule can inhibit releasing of platelet activiting factor 〓 which inhibit platelet aggregation and have antithrombosis function. 4. Wenxin capsule can promote activity of t-PA, reduce activity of PAI-1, strengthen fibrinolytic function, therefore inhibit fibrin disposition. 5. Wenxin capsule can inhibit synthesis of smooth muscle cellular DNA. Its function don't kill it directly but prevent transformation from period Go to peried S. 6. Wenxin capsule is an effective prescription of anti-coronary atherosclerosis atherosis. The results of image pattern analysis tastifies that area of lipid plaque in treating group reduce 54% and 58% in contrast with that in control group. The results of light, electro- microscopy show that structure of endolethial cell and smooth muscle cell in treating group is more integral than that in control group.

其作用机制之一是由于该药具有良好的调脂降脂作用,其不仅能明显降低血清LDL-c及升高HDL-c含量,且能有效阻止TC沉积于血管壁,从而保护内皮细胞结构完整;此外,温心胶囊尚具有明显的抗氧化作用,该药不仅能明显降低血浆LPO含量,且能提高SOD活性,同时其对血红素在细胞内代谢的调节作用也参与了抗氧化过程。2 温心胶囊能明显促进内皮细胞释放CO及合成〓,提高HO活性,通过激活血管平滑肌细胞内鸟苷酸环化酶及腺苷酸环化酶引起细胞内cGMP、AMP浓度增加而引起血管舒张。3 温心胶囊能明显抑制血小板激活因子〓的释放,从而抑制血小板聚集,抗血栓形成。4 温心胶囊能明显提高t-PA活性,降低PAI-1活性,增强纤溶功能,从而抑制纤维蛋白沉积。5 温心胶囊能明显抑制平滑肌细胞DNA合成,其抑制作用不是直接杀伤抑制,而是阻止细胞由Go期向S期转化。6 温心胶囊是抗冠状动脉粥样硬化的有效制剂,图象分析结果证实,该药治疗组脂斑面积比对照组缩小了54%和58%,光电镜结果显示内皮细胞及平滑肌细胞结构较为完整。

Cell apoptosis is an important factor in the cell necrose following the skeletal muscle damage. The apoptosis relative genes such as Bcl-2, Bax are crucial in skeletal muscle cell spoptosis. Adaptation and damage are two dichotomal facets in daily physical exercise and military training.

细胞凋亡是骨骼肌损伤后细胞发生坏死过程中的重要作用因素,如凋亡相关基因:Bcl-2、Bax等在骨骼肌细胞凋亡过程中起着非常重要的作用。

Leucocytic differentiation antigen 40 sign-al access participates in regulation of inflammatory reaction accommodation of major cell component (vascular endothelial cell、vascular smooth muscle cell and macrophage), it is playing critical role in AS process, participates ather-osclerotic occurrence and development ,reside in atherosclerotic plaque cell thro-ugh CD40-CD40L interaction, induce activate by itself , express and secrete profit to happen immune response、inflammatory reaction、hemagglutination t-hrombogenic proteo-cytokine.

白细胞分化抗原40 (CD40)信号通路参与了动脉粥样硬化斑块内主要细胞成分(血管内皮细胞、血管平滑肌细胞以及巨噬细胞)炎症反应的调节,其在AS进程中扮演着关键性的角色,参与了动脉粥样硬化的发生和发展;存在于AS斑块内的细胞通过CD40-CD40L相互作用,导致自身活化,表达和分泌了有利于免疫应答发生、炎症反应、血凝和血栓形成的蛋白质细胞因子,如激活在AS斑块中的关键性细胞成分如黏附分子、炎症因子、基质金属蛋白酶等的产生,导致斑块的不稳定和破裂,而最终导致粥样硬化斑块病变的恶化。

Motilin and gastrin induced contraction of isolated gastric smooth muscle cells in a dose-dependent manner.(2) Motilin- and gastrin-induced antral smooth muscle cell contraction was inhibited by antibodies against Gαi-3. Motilin and gastrin caused an increase of Guanosine-5′-(3-[35S]thio)triphosphate([35S]GTPγS) binding of Gαi-3.(3) Contraction of antral muscle cells induced by motilin and gastrin was inhibited by the phospholipase C inhibitor U-73122 and the IP3 receptor antagonist heparin.

结果显示:(1)胃动素和胃泌素对胃窦平滑肌细胞均有收缩作用;(2) Gαi-3抗体可抑制胃动素和胃泌素加强胃窦平滑肌细胞的收缩,胃动素、胃泌素明显增加Gαi-3抗体与[35S]GTPγS的结合;(3)磷脂酶C抑制剂U-73122、三磷酸肌醇受体拮抗剂肝素可抑制胃动素和胃泌素引起的胃窦平滑肌细胞的收缩。

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