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These help a mitochondrial enzyme called carnitine acetyl transferase to do its job.

这能有助于一种叫做肉碱乙酰转移酶的线粒体酶的活动。

Evidences show that excitotoxcities of excited amino acid and free radical, Ca2+ overload, environmental alteration of neurons, mitochondrial dysfunction and activation of some apoptotic genes may be involved in it.

缺血性脑卒中确切的神经元凋亡机制尚不清楚，目前认为可能与兴奋性氨基酸、Ca2+超载、自由基的毒性作用、神经元生存环境的改变、线粒体功能障碍及凋亡调控基因的激活等因素有关。

They all contain 13 protein coding gene,2 rRNA gene,22 tRNA gene and 1 D-Loop.The base composition for the four nucleotides is A-32.0%,C-27.6%,G-14.7%,T-25.8%,And it is A-32.5%,C-26.9%,G-14.1%,T-26.5%for Yellow-throated Marten.But there are some definite differences in base composition,the using of Initiation codon and Stop codon,and the mode of repeat sequences in control region.The codon usages of Manes have bias,and the ttiird locations of codon of protein-coding genes have the higher frequency in using A and C.There may be some relativity with the content of A and C in D-loop,namely,it has relation with the mode of repeat sequences.The complete mitochondrial genome of the Sable and Yellow-throated Marten were submitted to GenBank,and the accession number are FJ429093 and FJ719367 respectively.3、The complete mitochondrial genome of 6 other species of Mustelidae from GenBank and some sequences of D-loop from the 6 species were aligned.

分析紫貂大兴安岭亚种、长白山亚种、阿尔泰亚种和北欧亚种间的基因流及进化历史得知：大兴安岭种群与新疆种群和长白山种群间的基因流水平最高(Nm=0.1260和0.1427)，新疆与长白山种群间最低Nm=0.0053紫貂种群在进化过程中可能发生过种群膨胀，经历过种群增长过程。2、对紫貂和黄喉貂的线粒体全基因组结构进行分析发现：全长分别为16 523bp和16549bp，均包含13个蛋白质编码基因、2个rRNA基因、22个tRNA基因和1个非编码序列区(D-Loop区，紫貂全序列中碱基组成为A-32.0%，C-27.6%，G-14.7%，T-25.8%，黄喉貂为A-32.5%，C-26.9%，G-14.1%，T-26.5%；基因排列顺序与日本貂和貂熊的一致，但碱基组成、起始密码子和终止密码子的使用及控制区中串联重复序列模式等均存在一定差异。

Our results indicate that 1 NSC606985, at nanomolar level, can effectively induce apoptosis in AML cells NB4 and U937 and significantly inhibit the proliferation without cell death in breakpoint cluster region–Abelson murine leukemia kinase-carrying leukemic K562 cells; 2 At such low concentrations, this agent also significantly inhibits the clonogenic activity of hematopoietic progenitors from patients with AML; 3 For apoptosis induction, NSC606985 rapidly induces the proteolytic activation of protein kinase Cδ with loss of mitochondrial transmembrane potential and caspase-3 activation; 4 Co-treatment with rottlerin, a PKCδ-specific inhibitor, completely blocks NSC606985-induced mitochondrial m loss and caspase-3 activation, while the

结果显示：(1)纳摩尔浓度的NSC606985即可有效诱导AML细胞系NB4和U937细胞凋亡并显著抑制含有bcr-abl融合蛋白激酶的K562细胞的增殖；(2)低浓度的NSC606985也显著抑制来自AML病人骨髓的新鲜白血病细胞的克隆形成能力；(3)除了迅速诱导线粒体跨膜电位的崩塌及Caspase-3的活化外，NSC60698也导致蛋白激酶Cδ的水解激活；(4)PKCδ特异的抑制剂rottlerin能够完全抑制NSC606985诱导的线粒体跨膜电位的崩塌和Caspase-3的活化，而Caspase-3特异的抑制剂z-DEVD-fmk仅能部分削弱PKCδ的活化及细胞的凋亡；(5)以移植PML-RARα转基因小鼠产生的白血病细胞建立的模型研究了该化合物对白血病可能的治疗作用。

METHODS HepG2 cells were pretreated with specific inhibitors of mitochondrial respiratory chain complexes and a mitochondrial oxidative phosphorylation uncoupler, then exposed to But.

线粒体呼吸链复合物特异性抑制剂及线粒体氧化磷酸化解偶联剂预处理HepG2细胞后，染毒But。

In this experiment,nad4 and cox1 genetic variation of cestodes were firstly studied.Genetic variation and DNA polymorphism of mitochondria were carried out from mitochondrial gene.So the effective genetic markers were found which studied molecular taxonomy and Phylogenetic relationship of cestodes.This study have filled in the blank space of research that was related to China\'s cestodes mitochondrial genome research.

本试验首次对绦虫nad4和cox1基因的遗传变异情况进行研究，从线粒体基因的角度，开展绦虫线粒体遗传变异和DNA多态性方面的研究，找到了研究绦虫分子分类学、种群遗传关系有效的遗传标记，填补了我国寄生绦虫线粒体全基因组相关研究的空白。

After mitochondrial DNA transfer, the oxygen free radical increased in mitochondrial respiratory chain in cybrids of RTT children.

因此我们应用细胞融合技术，分析RTT患儿线粒体损伤在其发病中的作用。

The OXPHOS electron transfer chain (mitochondrial respiration chain) is located on the inner membrane of mitochondrion and comprising of four large trans-membrane protein complexes (mitochondrial respiratory Complex I, II, III and IV) as well as ubiquinone between Complex I/II and III and cytochrome c between Complex III and IV.

摘 要：线粒体作为真核细胞的重要&能量工厂&，是细胞进行呼吸作用的场所，呼吸作用包括柠檬酸循环和氧化磷酸化两个过程，其中氧化磷酸化过程的电子传递链位于线粒体内膜上，由四个相对分子质量很大的跨膜蛋白复合体(I、II、III、和IV)、介于I/II 与III之间的泛醌以及介于III 与IV之间的细胞色素c共同组成。

We suggest that by adopting other suitable MTSs and addition of a 3' untranslated region (3'-UTR) in the recoded mitochondrial gene may improve ND3 mitochondrial import.

我们建议在重新编码的基因上加上其他适合的粒线体标的序列和加入3'端未转译区域(3'-UTR)，或许能够增进ND3的正确运送。

This paper summarizes the general properties of mitochondrial ribosomal RNA,transfer RNA,protein coding genes and noncoding regions,and the phylogenetic usefulness of individual mitochondrial genes for insect taxa.

通过对昆虫遗传物质的研究，探讨不同分类水平上的昆虫类群之间的系统发育、进化机制和种群遗传变异及分化，从生命的本质上探寻昆虫各类群之间的系统关系[1，2 ] 。

I didn't watch TV last night, because it .

昨晚我没有看电视，因为电视机坏了。

Since this year, in a lot of villages of Beijing, TV of elevator liquid crystal was removed.

今年以来，在北京的很多小区里，电梯液晶电视被撤了下来。