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miocardial infarction相关的网络例句

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Objective To examinate the expression of GFAP and S - 100 protein between cerebral infarction and diabetes combined cerebral infarction by immunohistochemistry method. To detect the expression of GFAP and S -100 protein in diabetes.

目的 通过免疫组化方法,检测单纯脑梗塞、糖尿病合并脑梗塞时GFAP及S-100蛋白的表达特点以及糖尿病对GFAP及S-100蛋白表达的影响。

Abstract] objective to identify the relationship between serum homocysteine and carotid artery atherosclerosis in patients with cerebral infarction and effects of foliate,methylcobalamin.methods the concentrations of hcy were determined by means of fluorescence polarization immunoassay and carotid arteries was examined with color doppler ultrasound in 165 patients with cerebral infarction.the subjects were divided into 4 groups according to the severity of lesion of carotid artery.the concentrations of hcy were compared in different groups.the patients with hyperhomocysteinemia received foliate and methylcobalamin.results when lesion of carotid artery became severer,serum hcy was higher.the intima medial thicknesses of carotid artery were significantly decreased in patients with hyperhomocysteinemia who received foliate and methylcobalamin for two years.conclusion hyperhomocysteinemia may play an important role in development of carotid artery atherosclerosis.the intervention of foliate and methylcobalamin may reduce carotid artery atherosclerosis.

摘要] 目的探讨脑梗死患者颈动脉病变与血清同型半胱氨酸水平的关系及叶酸、甲钴胺长期干预对其影响。方法对165例急性脑梗死患者进行血清hcy的测定及颈动脉超声检查,并按动脉硬化的程度分组,比较不同程度颈动脉粥样硬化患者hcy水平,部分高hcy血症患者给予叶酸、甲钴胺干预,随访2年。结果随着颈动脉病变程度的加重,血清hcy浓度呈上升趋势,高hcy血症干预者动脉内中膜厚度明显减小。结论高hcy血症对颈动脉粥样硬化的形成起重要作用,长期给予叶酸、甲钴胺干预可能减轻颈动脉粥样硬化。

In the died patients there were2for myocarˉdial infarction,2for cerebral infarction,2for innutrition,and16for insufficient PD.

死亡原因:心梗2例,脑梗2例,营养不良2例,PD不充分16例。

OBJECTIVE: To investigate the relationship between the incidence of MAU in patients with acute cerebral infarction and the risk factor of stroke and the recurrence of cerebral infarction.

目的:研究急性脑梗死患者微量白蛋白尿的发生率、与脑卒中危险因素及脑梗死复发的关系。

Acute myocardial infarction is a common cause for sudden and unexpected death. However, it is difficult to detect the myocardial changes in medico-legal practice with traditional macroscopic examination or routine histological stains when the sudden death occurs in the hyper-acute phase of infarction (<3h).

急性心肌梗死是引起猝死的常见原因,然而,在法医实践中,对于心肌梗死的超急性期(<3小时)发生死亡的心脏用传统的肉眼观察或常规组织学检查的方法很难发现心肌的形态学改变从而提供客观的诊断依据。

Objective:To determine the risk factors for trigger effects of cerebral infarction, so as to supply basis for prevention of cerebral infarction paroxysm.

目的:探讨与脑梗死急性发作有关的触发危险因素,为有效防治突发脑梗死提供依据。

Results The clinical manifestation of brain stem hemorrhage secondary to cerebral infarction included sudden onset,evelution abruptly,coma,quadriplegia,higher fever and respiratory failure. CT usually do not identify the high dense location of brain stem. Large infarction of cerebral hemisphere may lead to shift the brain laterally and downward to produce herniation with subsequent brain stem compression,shift and distortion. Secondary brain stem hemorrhage occured usually in midderline area of midbrain or pontine.

结果 脑梗塞继发脑干出血的临床特点是发病急、进展快、昏迷、四肢瘫、过高热及呼吸衰竭,CT一般不能发现脑干高密度病灶,病理结果均为半球大病灶脑梗塞,严重海马回疝致脑干严重受压、变形和移位,继发脑干出血以中脑为主,可累及桥脑,主要位于中线部位。

Compared with CT or MRI,SPECT study can make out the location and size of infarction in the much earlier stage,and provide reliable evidence for the ultra-acute treatment of hemorrhage and infarction,or as MRI in the revealment of smaller lesion in and deep brain,so these three kinds of diagnoses method can not be substituted each other.

但对脑出血与脑梗死的鉴别不如CT对脑干及脑深部,较小病灶不如MRI显示清楚,因此三种检查方法不能互相取代。

TGF-β1 in cortex infarction group was higher than that of subcortex group(P<0.05);the larger the infarction size was,the higher the TGF-β1 was(P<0.05);TGF-β1 in severe group was higher than that in mild group,but no statistical significance.

皮质梗死组TGF-β1高于皮质下组(P<0.05;梗死体积大则TGF-β1浓度高(P<0.05);重型组TGF-β1浓度高于轻型组,但无统计学意义。

An association of ACE I/D polymorphism with myocardial infarction was found in a subpopulation younger than 60 years, which was strengthened in patients of myocardial infarction with obesity.

在研究的样本中,未发现ACE基因型与心梗相关;但在年龄60岁及以下的个体中,DD基因型与心梗显著相关,且这种相关在合并肥胖的个体中进一步加强。

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