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metaplasia相关的网络例句

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与 metaplasia 相关的网络例句 [注:此内容来源于网络,仅供参考]

The histopathologic changes included lymphocyte and monocyte infiltration,hyperplasia of synovial cells and small vessels,interstitial fibrosis,hyaline degeneration and cartilaginous metaplasia.The immunohistochemical observations showed that the high expressions of IL-1 and IL-6 were significantly different between the pathologic plicae and the control groups(P<0.01).The positive expressions of IL-1 and IL-6 were the synovial cells and monocyt-lymph cells in the pathologic synovial plicae.The positive expressions of MMP-1 and TIMP-1 have significant difference between the experiment and control group(P<0.01,P<0.05).The expression of MMP-1 was positive in synovial lining cell,monocyte,fibroblast,endothelial cell in small vessel and chondrocyte.The TIMP-1 expression was detected in the synovial lining cells and a small quantity fibroblast.

结果 正常滑膜皱襞和病理性滑膜皱襞在滑膜细胞增生及小血管增生、间质纤维化及玻璃样变、软骨化生组织学改变方面,差异均有显著性(P<0.01);IL-1、IL-6在病理性滑膜皱襞内的增生滑膜细胞、单核及淋巴细胞和在正常滑膜皱襞内的表达差异均有显著性(P<0.01); MMP-1、TIMP-1在病理性滑膜皱襞和正常皱襞内的阳性表达,差异具有显著性(P<0.01,P<0.05),MMP-1在增生滑膜衬里层细胞、单核和纤维母细胞、血管内皮细胞和软骨化生的软骨细胞呈阳性表达;TIMP-1只在滑膜衬里层细胞和少量纤维母细胞有表达。

Administration of LTE4 to the airways of sensitized mice potentiates eosinophilia, goblet cell metaplasia, and expression of interleukin-13 in response to low-dose aerosolized allergen.

对于低剂量变应原雾化吸入致敏的小鼠,经气道给予LTE4可导致嗜酸性粒细胞增多,杯状细胞化生以及IL-13生成。

Objective To observe expressions of Survivin and basic fibroblast growth factor in tissues of gastric carcinoma and intestinal metaplasia and their relationship with angiogenesis of gastric carcinoma so as to provide experimental and theoretical basis for diagnosis, treatment and predicating prognosis of gastric cancer.

目的 探讨Survivin和bFGF在胃癌及肠化生组织中的表达及其与胃癌血管生成的关系,为胃癌的诊断、治疗及预后判断提供依据。

Objective To observe expressions of Survivin and basic fibroblast growth factor in tissues of gastric carcinoma and intestinal metaplasia and their relationship with angiogenesis of gastric carcinoma so as to provide experimental and theoretical basis for diagnosis, treatment and predicating prognosis of gastric cancer.

作者:姬永忠罗梅张煦赵宪龙目的探讨Survivin和bFGF在胃癌及肠化生组织中的表达及其与胃癌血管生成的关系,为胃癌的诊断、治疗及预后判断提供依据。

Gastric cancer usually arises in chronic stomach inflammation and where atrophy and intestinal metaplasia have supervened.

胃癌发生的过程通常是长期的慢性发炎,慢慢导致细胞的萎缩和化生,然后走向癌化。

In both human and animals, ozone is known to induce inflammatory responses and mucous cell metaplasia in nasal cavity, which thought to be related to pathogenesis of nasal airway hyperreactivity.

在过去人体及动物实验中,已知臭氧会引发呼吸道产生发炎反应及鼻腔的黏膜细胞变形,这些被认为与呼吸道过度反应的病理生成有关。

Degradable biomedical membrane is a biological barrier membrane, whose mechanism is to segregate the inflamed or trauma from the tissue and prevent the fibroblast inrushing the traumatic surface. And the controllable degradation time can assure the trauma of insulating until metaplasia. The membrane can effectively avoid the adhesion.

生物可降解医用膜作为生物屏障膜可有效防止术后粘连,其作用机理是将炎症或损伤部位与周围组织隔离开来,阻止周围组织中成纤维细胞对创面的入侵,而其降解时间的可控性能保证有效隔离直到损伤部位再上皮化,从而达到预防粘连形成的目的。

Squamous metaplasia present with some mitoses which is acceptible at the lowr portion of the epithelium for reactive/reparative changes.

上皮底层反应性/修复性改变所致的有部分有丝分裂活性的鳞化是可以接受的。

It is reported that COX-2 plays important roles in it, through xenobiotic metabolism, inhibiting apoptosis, development of inflammation-stimulated hyperplasia and metaplasia, suppressing host immune responses, inducing angiogenesis and promoting infiltration and metastasis.

食管癌具有高发病率和死亡率,近年来认为环氧合酶-2通过异型生物质代谢,抑制凋亡,参与慢性炎症、免疫抑制,上调血管生成因子的表达,以及促进肿瘤的浸润和转移在其致癌中起重要作用。

The experimental results show that: DNA of normal gastric mucosa has stable phosphate backbone; Since the peak intensity at 1090 cm^(-1) is lower than that at 1050 cm^(-1), phosphate backbone in DNA of intestinal metaplasia becomes unstable, while the rest spectra peaks are similar to those in normal gastric mucosa; In terms of adenocarcinoma DNA, double peaks at 1090 cm^(-1) whose intensity are higher than 1050 cm^(-1), are observed, which indicates that DNA chain is broken and re-forms a double stabilized phosphate backbones. In addition, peaks at 950 cm^(-1), 1010 cm^(-1), 1100~1600 cm^(-1) also exhibit obvious difference compared with normal gastric mucosa, which shows that deoxyribose and bases are changed due to broken of DNA.

实验结果表明,胃粘膜正常组织DNA中有稳定的磷酸骨架;肠化组织基因组DNA拉曼特征峰在1090 cm^(-1)处谱峰强度低于1050 cm^(-1)处谱峰强度,表明其磷酸骨架变得不稳定,其它位置的谱峰与正常组织相似;腺癌组织基因组DNA拉曼特征峰在1090 cm^(-1)附近出现双峰,其相对于1050 cm^(-1)处谱峰强度更强,提示DNA有可能出现断链并再次形成了稳定的磷酸骨架,在950 cm^(-1)、1010 cm^(-1)、1100~1600 cm^(-1)波段的特征谱峰与正常组织DNA相比变化也较大,说明脱氧核糖和碱基可能由于DNA断链也随之改变。

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