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macrosomia相关的网络例句

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Local causes of uterine atony is the twins, fetal macrosomia, polyhydramnios, etc., uterine muscle fibers over-extended, over-expansion of the uterus, resulting in reduced post-partum resumption of non-performing.

宫缩乏力的局部原因就是双胎、巨大胎儿、羊水过多等,子宫肌纤维过度伸展,子宫过度膨胀,造成产后缩复不良。2、如果生第二胎,同样的事情未必会发生。

Poorly controlled gestational diabetes is associated with an increase in the incidence of preeclampsia, polyhydramnios, fetal macrosomia, birth trauma, operative delivery, and neonatal hypoglycemia.

控制不好的妊娠期糖尿病与先兆子痫,羊水过多,巨大儿,产伤,手术分娩,和新生儿低糖血症的发生率升高有关。

Poorly controlled gestational diabetes is associated with an increase in the incidence of preeclampsia,polyhydramnios,fetal macrosomia,birth trauma,operative delivery,and neonatal hypoglycemia.

控制不好的糖尿病期糖尿病与先兆子痫,羊水过多,巨大儿,产伤,手术分娩,和干细胞糖尿病足低糖血症的发生率升高有关。

Most studies report fourfold to fivefold higher rates of macrosomia in infants of mothers with type 2 diabetes.

多数研究报告称,患有2型糖尿病的孕妇产下巨大胎儿的几率比一般人群高4~5倍。

PIH disease pathogenesis, has not yet been clarified, according to the etiology of epidemic survey found that pregnancy causes a high incidence of disease may be related to the following factors:(1) the spirit of excessive tension or by stimulating the central nervous system dysfunction resulting in;(2) the cold season or temperature change is too large, especially at high pressure;(3) of young pregnant women or elderly primipara;(4) has chronic high blood pressure, nephritis, diabetes and other medical history of pregnant women;(5) malnutrition;(6) short body fat;(7) uterine hypertonicity, such as polyhydramnios, twins, diabetes, fetal macrosomia and the mole, etc.;(8) family history of hypertension, particularly pregnant women pregnant mother has history of hypertension.

妊高症的发病原因,至今还未阐明,根据流行病因学调查发现,妊高症的发病原因可能于以下因素有关:(1)精神过分紧张或受刺激致使中枢神经系统功能紊乱;(2)寒冷季节或气温变化过大特别是气压高时;(3)年轻孕妇或高龄初产妇;(4)有慢性高血压、肾炎、糖尿病等病史的孕妇;(5)营养不良;(6)体型矮胖;(7)子宫张力过高,如羊水过多、双胎、糖尿病巨大胎儿及葡萄胎等;(8)家族中有高血压史,尤其是孕妇之母有妊高症史者。

Results After 1 week diet therapy, the pregnant women's 2h PBG decreased significantly, and the difference was statistically significant (t=13. 685,P.01). Until delivery, 73 cases were treated by diet therapy only (86%), 12 cases were treated by diet and insulin therapy (14%), 2h PBG and HbA1C decreased significantly, and the difference was statistically significant (t=16.871, t=10.314, P.01). No ketonuria, pregnancy induced hypertension, fetal distress, premature delivery and fetal macrosomia were found.

结果 饮食治疗一周后,餐后2h血糖降低,差异有统计学意义(t=13.685, P.01);至分娩前,单纯饮食治疗73例(86%)、饮食治疗+胰岛素治疗12例(14%),餐后2h血糖、糖化血红蛋白均降低,差异有统计学意义(t=16.871、10.314, P.01),无酮症、妊娠期高血压征、胎儿宫内窘迫、早产、巨大儿发生。

C. Risk factors for macrosomia include maternal birth weight, prior macrosomia, preexisting diabetes, obesity, multiparity, advanced maternal age, and a prior shoulder dystocia.

C。 巨大儿的危险因素包括母亲出生时体重,巨大儿产史,糖尿病,肥胖,经产妇,高龄产妇和肩难产产史。

C. Risk factors for macrosomia include maternal birth weight, priormacrosomia, preexisting diabetes, obesity, multiparity, advancedmaternal age, and a prior shoulder dystocia.

C。巨大儿的危险因素包括母亲出生时体重,巨大儿产史,糖尿病,肥胖,经产妇,高龄产妇和肩难产产史。

B. preprandial blood glucose concentrations below 90 mg/dl and one-hour postprandial concentrations below 120 mg/dl minimize the incidence of macrosomia.

餐前血糖浓度小于90 mg/dl,餐后一小时浓度小于120 mg/dl使巨大儿的发生率降到最低。

Results Fetal Macrosomia group: the rate of obesity is 9.4%, obviously higher than that of the contrast group, 2.03%(χ2=7.4715,P.01), the rate of overweight is 19.4%, higher than that of the contrast group, 3.3%(χ2=18.9434,P.01);Fetal Macrosomia group: there is a remarkable difference in GLU, ALT, HDL, Insulin, and APN,(P﹤0.05~0.01),There were marked differences in the aspects of body mass index, the level of blood grease,HDL, ALT, HOMA-IR, adiponectin(P.05~0.01) Conclusion: In adolescence of Fetal Macrosomia weight index, GLU, Insulin and ALT are obviously higher than that of the contrast group; the HDL ,and APN which has great protective effect to angiosclerosis is also absolutely lower than the compare group.

结果:巨大儿组:肥胖率为9.4%,明显高于对照组2.03%(χ2=7.4715,P.01)、超重率为19.4%,高于对照组的3.3%(χ2=18.9434,P﹤0.01);巨大儿组:血糖、转氨酶、高密度脂蛋白、胰岛素、脂联素方面有显著差异,(P﹤0.05~0.01),结论:巨大儿在青少年期,其体重指数、血糖、胰岛素、转氨酶水平明显高于对照组,对动脉硬化有保护作用的高密度脂蛋白、脂联素明显低于对照组。

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