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macrophages相关的网络例句

查询词典 macrophages

与 macrophages 相关的网络例句 [注:此内容来源于网络,仅供参考]

We conclude that baicalein, wogonin and chrysin show a suppressive effect on iNOS gene expression in macrophages stimulated with LPS and the sequence of their suppressive effect intensity is chrysin, baicalein and wogonin seperately, and that it may be an important foundation for the combination therapy of TCM and Western medicine in the treatment of endotoxin-shock syndrome.

这些的发现,显示baicalein 、wogonin 和chrysin对脂多醣诱导小鼠巨噬细胞株生成一氧化氮有抑制的作用,而以。chrysin抑制作用最强,其次为baicalein 、wogonin,此结果可作为未来中西医整合治疗败血症之主要依据。

In contrast, treatment with baicalein (1OOμM), wogonin (1OOμM) and chrysin (1OμM) increased the formation of TNF-α in macrophages stimulated by LPS (0.1μg/ml).

但相对地,在以脂多醣(0.1μg/ml)诱导巨噬细胞中,以baicalein(100μM)、wogonin(100μM)和chrysin(10μM)处理后,却会增加TNF-α的生成。

AIM: To study the activation and the immune regulation of paecilomyces cicadidae total polysaccharides on macrophages in old rats.

目的:观察蝉拟青霉总多糖对老龄大鼠巨噬细胞的激活作用,探讨其对老龄大鼠的免疫调节作用。

2The distribution of HIF-1 a and COX-2 was unequal;.(3)These findings indicate that HIF-1a and COX-2 may be two important factors in pulpal lesions and there is a significantly hypoxia condiction in inflamed pulp .(4)The increase of HIF-1 a is a possible mechanism for hypoxia preconditioning .(5)Our results suggested that pulpal fibroblasts as well as macrophages might be involved in the pathogenesis of irreversible pulpitis.

和COX一2的表达均有不同程度的变化,在牙髓炎病程中可能发挥着重要的生理和病理作用,③在牙髓炎进程中和牙髓自身修复过程中存在着缺氧环境和缺氧耐受机制,④牙髓成纤维细胞有可能与单核巨噬细胞一同参与牙髓炎的病理进程,且具有修复和免疫功能。

Objective To investigate the role of osteopontin in the accumulation of macrophages in rat experimental crescentic glomerulonephritis.

目的 观察骨调素在大鼠实验性新月体肾炎肾脏组织中的表达及功能性抑制OPN的活性对巨噬细胞在肾脏局部浸润的影响。

Effect of Astragalus radix on proliferation and nitric oxide production of head kidney macrophages in Cyprinus carpio:an in vitro study .

黄芪对鲤头肾中巨噬细胞的增殖和一氧化氮产量的影响:离体研究。

The in vitro effect of Astragalus radix water extract onthe non-specific immune responses of macrophages isolated from the head kidney of Cyprinus carpio was evaluated.

用RPMI细胞培养液对分离的细胞进行原代培养,在细胞培养液中加入不同浓度的黄芪水提取液来研究黄芪对鲤头肾中免疫细胞非特异性免疫应答的影响。

Cytologically, there were rare neutrophils and macrophages at the margins of the specimen, which was consistent with a mild inflammatory reaction to the extruded allograft-collagen sponge complex.

细胞学方面,标本的边缘见少量中性粒细胞和巨噬细胞。与所取骨-胶原海绵复合物有中度炎性反应相一致。组织培养未见感染。

Results: Tyroserleutide can significantly increase the life span of H22 tumor-bearing mice by 50-70% in dosages of 20ug/kg/d-80ug/kg/d,specially the high dosage of 80ug/ml can significantly increase the life span by 69.24%; Tyroserleutide can inhibit the growth of transplanted hepatocellular tumor BEL-7402 in nude mice,the rate of tumor inhibition was25-50% in dosages of 40-320ug/ml ,the inhibition rate of 160ng/ml was 44.03%; Tyroserleutide could inhibit the growth of H22 and BEL-7402 tumor in a dose-dependent manner. Simultaneously, tumoricidal activity of tyroserleutide against BEL-7402 cell line in vitro was observed hinger when compared with the control group(P.05).The inhibition effect of 72hrs was higher than 24hrs,48hrs,96hrs.And specially the high dosage of 160ug/ml can significantly inhibit growth of tumor cell by 19.36%. Tyroserleutide can activated PEM and marked enhance cytotoxicity andphagocytosis functions in vitro and in vivo. The OD values of cytotoxicity were observed hinger when compared with the control group(P.05).The cytotoxicity of macrophages activated by tyroserleutide against BEL-7402 and B16-F10 was 35.58%,61.2% in vitro and21.39%,47.63% in vivo. The cytotoxicity rate of nude mice PEM was 32.86%,73.07% in vivo. Furthermore, tyroserleutide alone could stimulated the production of IL-1B TNF- a and NO by M . Tyroserleutide and LPS could synergistically activated M producing more cytotoxicity effectors. Conclusion: Tyroserleutide had inhibition functions against hepatoma carcinoma .Its possible mechanisms were related to the affect that Tyroserleutide could inhibit tumor cell directively and induce tumor cells apoptosis or death effectively.

结果:酪丝亮肽能显著延长腹水型肝癌H_(22)小鼠的生存时间,给药剂量为80μg/kg/d时疗效最显著,达到69.24%,在20μg/kg/d-80μg/kg/d剂量范围内生命延长率为50-70%,给药剂量与荷瘤鼠生存时间呈现一定量效关系;酪丝亮肽能显著抑制人肝癌BEL-7402移植瘤裸鼠的肿瘤生长,给药剂量为160μg/kg/d时疗效最显著,抑制率为44.03%,并且在40-320μg/kg/d剂量范围内抑制率为25-50%,给药剂量与肿瘤抑制率呈现一定量效关系;酪丝亮肽体外对人肝癌BEL-7402细胞生长有一定的抑制作用,在作用72hrs时各浓度酪丝亮肽对肿瘤细胞的抑制作用较24hrs、48hrs、96hrs明显,其中浓度为100μg/ml时抑制率达19.36%;酪丝亮肽体内外均能增强小鼠腹腔巨噬细胞对肿瘤细胞的杀伤:体外作用中巨噬细胞对BEL-7402、B16-F10的杀伤功能明显增强,与效应细胞对照组相比有显著性差异(P<0.05)杀伤率分别达到35.58%、61.2%;体内作用中巨噬细胞对BEL-7402、B16-F10的杀伤功能明显增强,与生理盐水对照组相比有显著性差异(P 。05),杀伤率分别达到21.39%、47.63%;裸鼠腹腔巨噬细胞经酪丝亮肤作用后对BEL一7402、B 16一F10杀伤功能明显增强,与生理盐水对照组相比有显著性差异(P.05),最高杀伤率分别达到32.86%、73.07%;酪丝亮肤能增强单核巨噬细胞系统的吞噬功能,吞噬指数与生理盐水组比较有显著性差异(P.05);酪丝亮肤体外作用能促进小鼠腹腔巨噬细胞分泌合成细胞毒效应分子IL一lp、TNF一Q和NO,与效应细胞对照组相比有显著性差异(P.05);酪丝亮肤体内作用能促进小鼠腹腔巨噬细胞分泌合成细胞毒效应分子IL一lp、TNF一Q和NO,与生理盐水对照组相比有显著性差异(P.05);酪丝亮肤能促进鼠巨噬细胞株R戌W264.7分泌合成IL一1p和NO,IL一1日、NO水平分别在酪丝亮肤作用24hrs、12hrs时达到高峰,酪丝亮肤单独应用能提高巨噬细胞的分泌合成功能,而且酪丝亮肤能与LPS协同作用刺激巨噬细胞的细胞毒效应分子分泌合成。

However, another investigation12 using the immunolocalization of MMP1, MMP2, MMP9, and MMP14 (MMP14 is a membrane-bound MMP) showed elevated levels of MMP2 and MMP14 in macrophages, as well as in the type II cells and fibroblasts, in emphysematous, compared to nonemphysematous, tissue.

但是在另一项对MMP 1,MMP 2和MMP 14(MMP 14 为有膜MMP)的免疫定位的研究中发现,气肿性组织较非气肿性组织而言,巨噬细胞中的MMP 2与MMP 14水平有升高,II型细胞和层纤维细胞亦如此现象。

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