英语人>网络例句>macrophage 相关的网络例句
macrophage相关的网络例句

查询词典 macrophage

与 macrophage 相关的网络例句 [注:此内容来源于网络,仅供参考]

Methods DCs were prepared from peripherad blood mononuclear induce d with gra-nulocyt e/macrophage colony-stimulating factor and interleukin-4. Apoptosis of glioma cells were induced with γ-radiation. We design these experiment groups including (1) coculture of DCs and apoptotic glioma cells and T cells,(2) coculture of DCs and U937 cells and T cells,(3) coculture of DCs and cultured glioma cell and T cells,(4) coculture of Des and T cell.

用粒-巨噬细胞集落刺激因子加白介素-4(IL-4)从人外周血分化、诱导DCs、γ-射线在体外诱导培养的人脑胶质瘤细胞凋亡,将DCs、T淋巴细胞和凋亡胶质瘤细胞共培养,同时设计不同类型肿瘤细胞(U937及培养胶质瘤细胞)作对照,分离、富集DCs、T淋巴细胞进行免疫应答及肿瘤细胞杀伤试验。

Methods DCs were pre pared from peripherad blood mononuclear induce d with gra-nulocyt e/macrophage colony-stimulating factor and interleukin-4.Apoptosis of he patochlangioma cells were induced with γ-radiation.We design these experiment groups including(1)coculture of DCs and apoptotic cancer cells and T cells,(2)co culture of DCs and necrosis cancer cells and T cells,(3)coculture of DCs and cul tured cancer cell and T cells,(4)cocultu re of DCs and T cell,(5)cocuture of DCs and cultured cancer cell.

用粒-巨噬细胞集落刺激因子加白介素-4(IL-4)从人外周血分化、诱导DCs、γ-射线在体外诱导培养的人胆管癌细胞凋亡,将DCs、T淋巴细胞和凋亡胆管癌细胞共培养,同时设计不同类型肿瘤细胞(坏死胆管癌细胞及培养胆管癌细胞)作对照,7d后,分离、富集DCs、T 淋巴细胞进行免疫应答及肿瘤细胞杀伤试验。

The next most common milk leukocyte is the macrophage, which is phagocytic like neutrophils and performs a number of other protective functions.

另一个最常见的白细胞是巨噬细胞,它是象嗜中性白细胞一样的噬菌细胞,有着很多其他的防御功能。

The next most common milk leukocyte is the macrophage, which is phagocytic like neutrophils and performs a number of other protective functions.

另一个最常见的白细胞是巨噬细胞,它是象嗜中性白细胞一样的噬菌细胞,有着很多其他的防御机能。

Finally, the fluorescent intensity was measured by flow cytometry. Results: The E. coli was labeled well by FITC in carbonate buffer. Macrophage can phagocytize E.

结果:FITC能够有效标记大肠埃希菌;小鼠腹腔诱导的巨噬细胞能够吞噬大肠埃希菌,并且在30min时达到最大峰值。

Finally, the fluorescent intensity was measured by flow cytometry. Results: E. coli was labeled well by FITC in carbonate buffer. Macrophage can phagocytize E.

结果:FITC能够有效标记大肠埃希菌;小鼠腹腔诱导的巨噬细胞能够吞噬大肠埃希菌,并且在30min时达到最大峰值。

MethodsThe transplanted tumor model of the mouse S180 sarcoma was established.Fifty mice were randomly divided into five groups,the control group,the CTX group,the Gecko group.They were treated respectively with oral administration of saline,and intraperitoneal injection of CTX 100 mg/kg only one time,oral administration of Gecko in doses of 13.5,9,4.5 g/kg,one time a day.After 14 days,the anti-tumor activity was evaluated by tumor tissue weighing. The impact on immune organ was detected by accounting thymus index, spleen index and the number of peritoneal macrophage which phagocytose chicken red blood cells.The protein express of vascular endothelin growth factor and basic fibroblast growth factor were detected by SABC immunohistochemistry.

方法建立移植瘤小鼠S180肉瘤模型,将50只雌鼠随机分为对照组、环磷酰胺组、壁虎高组、中组、低组共5组,分别给于生理盐水灌胃1次/d,CTX(100 mg/kg)腹腔注射1次,壁虎高、中、低组(13.5,9,4.5 g/kg),每天灌胃1次。14 d后,称取荷瘤小鼠瘤重、胸腺重、脾脏重,计算抑瘤率、胸腺指数、脾脏指数,观察腹腔巨噬细胞吞噬鸡血红细胞实验指标;用SABC免疫组化法检测血管内皮生长因子、碱性成纤维细胞生长因子的蛋白表达,TUNEL方法检测细胞凋亡率。

Results:Demonstrated that the fat pad and synovium showed small vascular dilatation after injury, vascular congestion and small pieces of hemorrhage;interstitial edema of the fat,lymphoc yte,plasmocyte,mastocyte and macrophage infiltration;cellular and villus like pr oliferation on the synovium of fat pad surface.

结果:损伤后的脂肪垫及滑膜可见脂肪垫内小血管扩张、血管内充血、及小片状出血;脂肪间质水肿,淋巴细胞、浆细胞肥大细胞和巨噬细胞浸润;可见脂肪垫表面滑膜细胞增生,及滑膜绒毛状增生。

Among its many biological activities, the major function of GM-CSF is to stimulate hematogenie stem cells prolification from silent stage to S phage, so that the stem cells can differentiate into granulocyte-macrophage cells.

有4个外显子和3个内含子,基因定位于5号染色体长臂5q21-5q32区,大多数器官均能产生GM-CSF,GM-CSF有多种生物活性,作用于多种细胞谱系,其主要作用是刺激造血祖细胞增殖即从静止期进入S期,并分化为粒细胞---单核巨噬细胞。

Aim To investigate the effect and mechanism of retinoid X receptor activation on macrophage apoptosis induced by oxidized low density lipoprotein.

目的 探讨激活视黄醇类X受体对氧化型低密度脂蛋白诱导巨噬细胞凋亡的影响及其机制。

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