查询词典 maco

The astrocyte swelling appeared as early as 30 mins and indented the neuron at 6h after MACO.

缺血30分钟后，〓ATP酶及〓-ATP酶反应产物在神经细胞中即减少到消失。

Sell Maco products to window manufacturers and developers

向门窗厂、开发商或承包商销售迈柯产品

The expression levels of VEGF and FLT1,FLK1 mRNA in equal area of penumbra were measured with situ hybridization in different times after MACO (1 h,3 h,6 h,12 h,24 h). The difference was compared between DMCI group and no diabetes cerebral infarction group.

建立DMCI大鼠模型，在脑缺血后1 h、3 h、6 h、12 h、24 h，采用原位杂交方法测定大鼠大脑缺血半暗带等值区VEGF、FLT1、FLK1 mRNA的表达水平；并与无糖尿病的脑梗死大鼠比较。

Our product s are saled to the whole world, including Taiwan Taipei, kaohsiung,maco,HongKong,Shenzhen ,Beijing,shanhai,tianjin,chongqin,qindao,xian,nanjing,guangzhou,and oversea Vietnam ,Singapore,malaysia, and so on.

我们的营销足迹，遍及从台北到高雄、香港、澳门，从深圳到北京、上海、天津、重庆、青岛、西安、南京、广州等中国大陆大部分大中城市甚至外销越南、新加坡、马来西亚等国家与地区。

In the experiment, depended on the traditional ischemia model MCAO, we both use the clot embolus model to study the edema of the neuron cell. We found that the hyperintense signal was seen in the DWI in few minutes by using the MACO models after the ischemia and reperfusion.

本实验在利用传统缺血模型MCAO基础上，结合与人类缺血卒中病程较接近的自体血栓脑栓塞模型，模拟急性脑卒中的过程，研究急性期缺血后神经细胞水肿发展的规律，了解神经元的生物代谢物质变化趋势，评价早期溶栓治疗的实验意义。

The expression level of FLK1 mRNA was lower (P.01) at 12 h after MACO in DMCI group than that in NDMCI group. Except for at 6 h, there was no significant difference between DMCI and NDMCI group in the expression level of FLT1 mRNA.

结果 DMCI组在脑缺血后各时间点脑缺血半暗带等值区VEGF mRNA的表达水平均显著低于NDMCI组（P.05～0.01），FLK1 mRNA 表达水平在脑缺血后12 h显著低于NDMCI组（P.01），FLT1 mRNA 表达水平在脑缺血后各时间点（除6 h）与NDMCI组差异无统计学意义。

Results The expression level of VEGF mRNA was lower in DMCI group than that of NDMCI group at every time point (P.05 ～ 0.01). The expression level of FLK1 mRNA was lower (P.01) at 12 h after MACO in DMCI group than that in NDMCI group. Except for at 6 h, there was no significant difference between DMCI and NDMCI group in the expression level of FLT1 mRNA.

结果 DMCI组在脑缺血后各时间点脑缺血半暗带等值区VEGF mRNA的表达水平均显著低于NDMCI组（P.05～0.01），FLK1 mRNA 表达水平在脑缺血后12 h显著低于NDMCI组（P.01），FLT1 mRNA 表达水平在脑缺血后各时间点（除6 h）与NDMCI组差异无统计学意义。

TSRP could decrease brain hydrated amount and markedly lower permeability of blood-brain barrier subjected to 2 h MACO followed by 24 h reperfusion, and this may be a mechanism of TSRP alleviating brain edema during I/R.

茅莓总皂苷各剂量组显著降低缺血2h再灌注6，24，48，72 h脑水肿和其对血脑屏障通透性，这可能是其减轻I/R时脑水肿的机制之一。

Methods: Thirty adult male Wistar rats were randomly divided into three groups: the operation group, the transplantation of MSCs group and the injection of saline group. Functional outcome measurements were performed based on the modified neurological severity score at day 1, 7, 14, 21 and 28 after MACO. The survival, migration, expression of Nestin, glial fibriliary acidic protein and neuronspecific enolase of 5bromo2deoxyuridinelabeled MSCs were detected by immunohistochemical double staining.

线栓法建立左侧大鼠MCAO模型，随机分为3组：手术组、MSCs 移植组、生理盐水组。5溴2脱氧尿核苷标记的MSCs移植后，采用改良神经功能损伤评分系统评价大鼠神经功能恢复情况；应用免疫组织化学双染技术检测MSCs的存活、迁移及其巢蛋白、胶质纤维酸性蛋白和神经元特异性烯醇化酶的表达。

Methods The high blood sugar animal model was induced by intraperitoneal injecting streptozotocin; the MACO model was made by the thread occlusions.

采用链脲佐菌素腹腔注射复制高血糖模型；线栓法制作大脑中动脉栓塞模型；应用免疫组化技术观察MMP-9蛋白表达，并测血脑屏障的通透性。

The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

这种做法不仅鼓励了更多的反应，而且减少跟风的可能性。