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lymphocyte相关的网络例句

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与 lymphocyte 相关的网络例句 [注:此内容来源于网络,仅供参考]

Deficiency of selenium in chicken decreases immunity of erythrocyte and the regulation of erythrocyte to the specific immunity, which includes the decrease of the phagocytic ability of phagocyte, the eliminating ability of circulatory immune complex, the ability of identifying organism, carrying antigen and the immune regulation effects in the activation course of T lymphocyte, B lymphocyte, NK cell alexin, which decreases immunity of organism.

雏鸡硒缺乏降低了红细胞免疫力;同时降低了红细胞对特异性免疫的调节作用,主要表现为降低了吞噬细胞的吞噬能力、清除循环免疫复合物能力、机体识别和携带抗原能力以及对T、B淋巴细胞、NK细胞补体的活化过程的免疫调节作用,结果使机体免疫力下降。

Lymphocyte blastogenesis and T lymphocyte population were analysized 1 d after the LPS administration,gene expression of IL-2 and IFN-γ was quantified by RT-PCR technology.

注射LPS使肉仔鸡发生了以细胞免疫为主的应激反应,皮质酮和T3水平、T4/T3比值及细胞因子mRNA表达量均能作为衡量免疫应激的敏感指标;免疫应激是相对于正常状态而言的,就其本身来说,尚无法衡量程度的高与低。

The immunosuppressive effect was determined with ratio of lymphocyte blastogenesis,phagocytotic function of macrophage,percentage of peripheral T-lymphocyte,delayed type hypersensitivity and antibody forming cell assay.

免疫抑制作用:测定小鼠脾淋巴细胞转化率;巨噬细胞的吞噬功能;外周血T淋巴细胞的百分率;迟发性的变态反应;血清溶血素和抗体形成细胞功能测定。

Results interleukin2activity,interleukin2receptor expression of lymphocyte membrane and lymphocyte blastogenesis in peripheral blood were inhibited at and above30PPM concentration of fluorine ion.

结果 当氟离子浓度达30PPM时,即可抑制白细胞介素2活性、淋巴细胞膜白细胞介素2受体表达及外周血淋巴细胞转化功能。结论提示氟化钠具有免疫毒性。

Methods The inter-leukin2activity was observed by MTT colormietric assay,the interleukin2receptor expression of lymphocyte mem-brane was examined by immunofluoresence technique and lymphocyte blastogenesis in peripheral blood was measured by morphologic observation.

通过体外实验,运用MTT比色法、免疫荧光技术、淋巴细胞转化试验等实验方法,检测白细胞介素2活性、淋巴细胞膜白细胞介素2受体含量及外周血淋巴细胞转化功能。

The following methods were used in the study, such as viral inoculation of animal, pathological method, flow cytometry, RT-PCR, in situ hybridization, immunohistochemistry and terminal deoxy nucleotidyl transferase mediated dUTP nick end labling . Morphological changes of the infected chickling, dynamic changes of T, B cells and T subsets, changes of adhesion molecules on the lymphocyte surface, necrosis and apoptosis of lymphocyte and neurons, changes of MIP-1β and IL-8 producing cells in the brain, cell types of perivascular tuffing in cerebral tissue were systematically studied.

在研究过程中,采用了病毒接种技术、普通病理学研究方法、流式细胞仪技术、RT-PCR技术、原位杂交技术、免疫组化技术、凋亡细胞末端标记技术,系统研究了不同日龄的SPF雏鸡人工感染AEV-NH937株后的病理变化,T和B淋巴细胞、T淋巴细胞亚群的动态变化规律,淋巴细胞表面某些粘附分子的变化,淋巴细胞和神经元的坏死与凋亡,雏鸡脑组织中产生趋化因子MIP-1β和IL-8细胞的变化规律,脑组织中围官性细胞浸润的细胞类型。

Results:Berberine inhibited HUVEC adhesion for lymphocyte when pretreated HUVEC and antagonised IL-1 and TNF-induced upregulation of HUVEC adhesion for lymphocyte.Meanwhile,IL-1-stimulated lymphocyte adhesion to HUVEC was diminished in the presence of berberine.The main molecular mechanism of berberines function may be downregulating the expression of ICAM-1 on the HUVEC .

结果:小檗碱能抑制静息的及IL-1、TNF激活的内皮细胞与淋巴细胞间的粘附,其主要分子机制为下调内皮细胞表面粘附分子ICAM-1的表达;小檗碱能抑制IL-1激活的淋巴细胞与内皮细胞的粘附,对淋巴细胞表面粘附分子CD18的表达无影响。

Using a model of the muscle pouch of bone graft, the effects of the systemic and local administration of IL-4 plus IL-10 on bone xenograft rejection were studied through the determination of lymphocyte responses from the mixed lymphocyte culture , subsets of lymphocyte, cytokines and histology in 1, 2, 4, and 6 weeks after implantation.

分别于术后1、2、4、6周取材,进行淋巴细胞刺激增殖实验、淋巴细胞亚群分析、细胞因子检测及组织学检查,了解移植免疫特点,并观察经腹腔和局部注射IL-4和IL-10后产生的免疫抑制作用。

DCs was impulsed with pure HBsAg before DCs maturation and cocultured with self-blood T lymphocyte, while DCs without pure HBsAg stimulated group, T lymphocyte group and only T lymphocyte group were prepared as control group.

体外实验中HBsAg刺激的人DCs能有效的呈递抗原并诱导HB特异性CTL的产生,能有效的抑制2.2.15细胞培养上清中HBeAg的表达。

Results:Berberine inhibited HUVEC adhesion for lymphocyte when pretreated HUVEC and antagonised IL-1 and TNF-induced upregulation of HUVEC adhesion for lymphocyte.Meanwhile,IL-1-stimulated lymphocyte adhesion to HUVEC was diminished in the presence of berberine.The main molecular mechanism of berberine's function may be downregulating the expression of ICAM-1 on the HUVEC .

结果:小檗碱能抑制静息的及IL-1、TNF激活的内皮细胞与淋巴细胞间的粘附,其主要分子机制为下调内皮细胞表面粘附分子ICAM-1的表达;小檗碱能抑制IL-1激活的淋巴细胞与内皮细胞的粘附,对淋巴细胞表面粘附分子CD18的表达无影响。

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