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lps相关的网络例句
与 lps 相关的网络例句 [注:此内容来源于网络,仅供参考]

Test of NO, TNFa, LPS in the serum and ascites of the patients with liver cirrhosis ascites can contribute to distinguishing between exudative ascites and transudate ascites and distinguishing between spontaneity peritonitis is infected in Gram-positivebacteria and Gram-negative-bacteria so as to instruct clinical use of drugs.

肝硬化腹水患者血清及腹水中测定NO、TNFa、LPS,有助於鉴别渗出性腹水和漏出性腹水;有助於鉴别自发性腹膜炎是革兰阴性菌或革兰阳性菌感染引起,指导临床用药。

Furthermore, we found that LPS could upregulate the expression of c-Src protein that in turn caused the enhancement of tyrosyl phosphorylation of Shc and ERK and promoted cellular growth.

此外,另一个Src的受质,FAK,其tyrosyl phosphorylation也因LPS促进Src整体活性提高而增加,而这可能与细胞的移动有关。

Results:The results showed that the apoptosis of neutrophiles was promoted by the treatment of 1 μg/ml LPS and 1 μm A23187,in 6 hours and 12 hours respectively.The apoptosis of neutrophil was markedly delayed by a PLA2 inhibitor,4-bromophanacyl bromid (p-BPB 0.01~1 mM).

结果:脂多糖(LPS 1μg/ml)和钙离子载体(A23187 1 μmol/L)等PLA2激动剂分别在共同培养6 h和12 h之内促进中性粒细胞凋亡,而PLA2阻断剂4-溴苯甲酰溴甲烷(p-BPB 0.01~1 mmol/L)可明显减弱上述激动剂的促凋亡作用。

Correlation analyses showed that the ITF content had a positive correlation to PCNA level but negative correlation to the levels of LPS and IMID.

相关分析显示,ITF含量同血浆LPS水平及肠粘膜损伤指数呈显著负相关,同肠粘膜PCNA值呈显著正相关。

PD antagonizes the damage effects of LPS and sera of burned rats by reversing the PKC activities, which may be one of the molecular mechanisms of PD antishock property.

PD对烧伤血清和LPS作用下的VSMC的保护作用可能是通过调节PKC的活性来发挥作用的,这可能是其抗休克的分子机制之一。

CONCLUSION摘要: PD antagonizes the damage effects of LPS and sera of burned rats by reversing the PKC activities, which may be one of the molecular mechanisms of PD antishock property.

结论摘要: PD对烧伤血清和LPS功能下的VSMC的保护功能可能是通过调节PKC的活性来发挥功能的,这可能是其抗休克的分子机制之一。

After maturation with the stimulation of LPS, apophyses became obviously increased by SEM scanning; the expression percentages of 33D1 and CD11c exceeded 80% by immunofluorescence staining and those of CD86 and MHCⅡwere 99.0% and 32.5%; MLR showed that mature BMDCs could better stimulate the proliferation of homologous T cells.

以LPS刺激成熟后,扫描电镜发现细胞突起更加明显,免疫荧光发现表达33D1和CD11c的细胞数均达到80%以上;流式细胞术检测CD86和MHCⅡ的细胞表达率分别为99.0%和32.5%;混合淋巴细胞反应提示成熟的BMDCs能明显刺激同源T细胞的增殖。

We conclude that NAC attenuates the increased expressionof CD11b in either LPS or IL-8-stimulated human whole blood.

我们认为 NAC 减弱 LPS 或 IL-8诱发的人全血中增高的 CD11b 表达。

Stable coating and minimal false positives were achieved by conjugating LPS to poly-L-lysine, which solved the desorption problem.

试验中采用LPS与多聚赖氨酸的结合,解决了包被过程中的解吸附作用,增强了包被的稳定性,同时也减少了假阳性反应,优化了包被过程。

AIM: To investigate the expression of human β-defensin-2 (hBD-2) mRNA and the changes of NF-κB activity induced by lipopolysaccharide in human primary epitheliums. METHODS: The pulmonary primary epitheliums from human were stimulated with LPS and the expression of hBD-2 mRNA was detected by reverse transcription polymerase chain reaction.

目的:观察脂多糖诱导人气道上皮细胞人β防御素-2(hBD-2)的表达及核转录因子κB活性的变化,探讨NF-κB在LPS诱导人气道上皮细胞hBD-2表达中的作用。

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