查询词典 lactate

Methods Rabbit pulmonary alveolar macrophages were cultured in vitro. The cell death rate, the malandialdehyde, lactate dehydrogenate and superoxide dismitase contents in the cultured cells were determined at different incubation time and different dose of chrysotile asbestos for the cytotoxicity of the material.

采用体外细胞培养技术，以兔肺泡巨噬细胞死亡率、乳酸脱氢酶、超氧化物歧化酶活力及丙二醛含量变化来表示不同剂量、不同培养时间的纤状蛇纹石石棉对巨噬细胞的毒性。

SMART analysis suggested that the ORF encoding protein contained a Ldh_1_N and a Ldh_1_C domain. Psite analysis showed that there was lactate dehydrogenate active site in the protein, which localized in cytoplasm according to PSORT program analysis.

SMART分析表明ORF编码的蛋白质具有Ldh_1_Ndomatn和Ldh_1_Cdomain，Psite分析表明有L-乳酸脱氢酶活性位点，PSORT程序分析将其定位于细胞质中。

Cultured ECV304 were either not treated, treatedwith 10 M of hydrogen peroxide (H2O2), treated with TNF-(40 ng/mL) alone, TNF- in the presence of 10 M of H2O2, or propofol plus H2O2 for 24 h. Cell viability was measuredby lactate dehydrogenate assay.

本研究取人血管内皮细胞 ECV304经培养后分为以下几组：对照组、过氧化氢预处理组、 TNF-预处理组， TNF-复合 H2O2预处理组、丙泊酚复合 H2O2预处理组。

A first demonstrator of the nanopotentiostat for lactate measurement has already been produced

第一个测量乳酸的nanopotentiostat已经产生。

After developing 48 hours, hepatocytes enters 2.0 mM t-butyl hydroperxide or 2.0 mM diamide to induced cell blebbing. The samples collected at 0, 15, 30, 60 minutes to analyze cell about membrane protein thiol deletion; the leakage of lactate dehydrodenase; the change of glutathione; the lipid peroxidation; the variation of actin and tubulin, and use Confocal fluorescent microscope to observe the change of actin and tubulin in rat hepatocytes.

肝细胞在48小时培养后，分别加入2.0mM t-buty1 hydroperoxide或2.0 mM diamide诱发细胞膜小泡生成，并分别於0、15、30、60分钟取样，分析细胞膜硫醇流失程度；乳酸去氢□渗漏；□胱甘□浓度变化；脂质过氧化；肌动蛋白与管蛋白变化；并利用雷射共轭焦萤光显微镜观察肝细胞中肌动蛋白及管蛋白的变化。

The result indicates the t-BHP or diamide both cause the mass loss about the membrane protein thiol in hepatocytes, the fast oxidization about glutathione in hepatocytes, the leakage of lactate dehydrodenase, but the level of t-BHP is larger than of diamide. In the aspect of lipid peroxidation, only the t-BHP produces apparent change. As to actin and tubulin, they both find fall-off and present time-dependency effects. The result in Confocal fluorescent microscope also show these two cytoskeletal proteins are related to cell blebbing, but they don''t play the same role during the porcess. Tubulin may be more important than actin. If the study predispose dithiothreitol before adding t-BHP or diamide, then the damage by the diamide is repressed, but just partly protection for the damage by t-BHP.

结果显示t-BHP或diamide均可造成细胞膜蛋白硫醇大量流失与胞内GSH快速氧化，亦显著导致乳酸去氢□渗漏，但t-BHP诱发的渗漏程度大於diamide；在脂质过氧化方面，则仅t-BHP处理组产生明显的脂质氧化情形；至於肌动蛋白与管蛋白单体均可见减少且呈现时间依赖效应；在雷射共轭焦萤光显微镜观察中，亦显现这两种架构蛋白与细胞膜起泡有关，但两者在小泡生成的过程中扮演的角色并不完成相同，可能管蛋白较肌动蛋白重要；如果细胞在t-BHP或diamide加入前预先以dithiothreitol处理，则diamide 所造成的伤害显著的受到抑制，但DTT对t-BHP所诱发的伤害则仅有部分保护效果。

Objective To evaluate the significance of plasma D-lactate,diamine oxidase and endotoxin activities in the patients with liver cirrhosis.

内毒素血症和肠粘膜屏障功能衰竭在肝硬化患者病情恶化中的作用，日益受到人们的重视。

Objective To evaluate the significance of plasma D-lactate,diamine oxidase and endotoxin activities in the patients of hepatic cirrhosis.

目的探讨D-乳酸、二胺氧化酶及内毒素在肝硬化患者血浆中的变化与临床意义。

Objective To explore the effect of total parenteral nutrition supplemented with glutamine on the activity of plasma diamine oxidase and D-lactate content in blood of patients with multiple organ dysfunction syndrome.

目的 探讨添加谷氨酰胺的全胃肠外营养对多器官功能障碍综合征患者血浆二胺氧化酶、D-乳酸的影响。

The weight ratio of catalysis system and ammonium lactate being 0.005-5％, and the magnetism solid acid catalyst and tin dichloride being 20∶1-1∶20; c.

催化体系总的加入量为乳酸铵重量的0.005-5％，磁性固体酸催化剂与二氯化锡的重量比为20∶1-1∶20；c。

The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

这种做法不仅鼓励了更多的反应，而且减少跟风的可能性。