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ischemic相关的网络例句

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与 ischemic 相关的网络例句 [注:此内容来源于网络,仅供参考]

Approximately one-quarter of ischemic stroke occurred in vertebrobasilar artery system.

近1/4的缺血性卒中发生在椎基底动脉系统。

59 cases with vertebrobasilar ischemic vertigo were examined by DSA during 2 years.

对两年来收治的57例椎基底动脉供血不足性眩晕患者行DSA检查,并进行综合分析。

objective: To investigate the manifest and clinical significance of DSA on vascular structural abnormalities in patients with vertebrobasilar ischemic vertigo.

目的:研究椎基底动脉供血不足性眩晕患者血管结构异常的DSA表现及临床意义。

Methods and Results— A case-crossoer study design was used to analyze ischemic eents in 12 865 patients who lied on the Wasatch Front in Utah.

方法和结果:一病例交叉研究纳入了居住在犹他州Wasatch 前线的12865例病人,以分析他们的缺血事件。

Methods and Results— A case-crossoer study design was used to analyze ischemic eents in 12 865 patients who lied on the Wasatch Front in Utah.

方法和结果:采用交互式病例研究分析了居住在Wasatch Front in Utah的12865个病人的缺血性事件。

Hypoxic-ischemic encephalopathy experimental grouping: Sixteen successful C57/BL6J mice models, aged 7 days, were randomized into 2 groups: experimental group (n =8), following ligation of right common carotid artery, mice were placed in the closed container containing 0.08 volume fraction of oxygen and 0.92 volume fraction of Nitrogen gas, and taken out 1.5 hours later; 3 μL ESCs were injected into the right cerebral ventricle at about 1 week, and control group (n =8), in which, the same amount of PBS was injected into the right cerebral ventricle.③ At 12 weeks after transplantation, the survival and migration of induced ESCs labeled by Lac-Z in the spinal cord and brain were observed by zymologic method.

缺氧缺血性脑病实验分组:将造模成功的鼠龄7 d的C57/BL6J16只小鼠随机分为2组:实验组8只,结扎右侧颈总动脉后将小鼠置入含体积分数0.08氧气和体积分数0.92氮气的密闭容器内,1.5 h后取出;1周后在右侧脑室内注入3 μL诱导胚胎干细胞。

The study of ischemic mechanisms has been vastly abetted by the use of transgenic and knockout mouse mutants.

转基因和被击昏的老鼠突变体的使用极大地支持了对缺血机械的研究。

Objective To evaluate the efficacy of combined transplantation of neurotrophin-3 and neural stem ceils in treatment of hypoxic-ischemic brain injury in rats, and study the possible mechanism.

目的研究神经营养素3(NT-3)及神经干细胞联合移植对缺氧缺血性脑损伤大鼠神经功能恢复及神经干细胞分化为神经元的比例的影响,并探讨其可能机制。

Results All patients were followed up for 18 months to 5 years, the function of articulatio coxae and degree of bony union, inclusion area to head of femur and ischemic necrosis of femoral head were observed through X-ray.

术后观察髋关节功能,X线片显示的截骨愈合程度、髋臼对股骨头包容程度及有无股骨头坏死等。

Mitochondria cristae and membrane in a great measure were anastomosis or vacuolization.Conclusion: All these results suggest that effects of H2S on papillary muscles are due to an increase in potassium efflux through opening the KATP channels at the early phase of ischemia, and acceleration in potassium reuptake through improving the Na+-K+-ATPase at the late phase of ischemia. Endogenous H2S may act as important regulator in electrophysiological characters in ischemic papillary muscles.

外源性H2S前处理可能通过兴奋KATP通道促进缺血早期K+外流,并且由于对Na+-K+-ATP酶的保护作用改善了缺血晚期对K+的重摄取,进而影响缺血环境下豚鼠乳头状肌细胞的电生理效应;乳头状肌细胞内源性产生的H2S可能在心肌缺血损伤时发挥着重要的电生理作用。

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