查询词典 ischemic

Methods:108 cases of 7-day-old sprague-dawley rat were divided at random into 3 teams : hypoxic-ischemic-team (hi team,36 cases),false-oprational control team(control team 36 cases), hypoxic-ischemic-gm1（gm1 team,36 cases）.deligation of cephalic artery and breathing in low-oxygen gas mixture was applied to prepare hibd mode.

选择新生7日龄sprague-dawley大鼠108只，随机分为三组：缺氧缺血组(hi组，36只)，假手术对照组(control组，36只)，缺氧缺血＋神经节苷脂组(gm1组，36只)。

Results According the MRI characteristric of hypoxic-ischemic encephalopath sequela:(1)The most common findings were white matter injury in 47 cases:①Periventricular leukomalacia in 23 cases;②Selective cerebral neuronal necrosis of basilar node lentiform nucleus and optic colliculus in 9 cases;③Corpus callosum damaged in 15 cases;(2)The most damaged of grey matter in 18 cases;(3)Focal or multifocal ischemic crerbral intenerate in 9 cases;(4)Diffuse crerbral injury in 5 cases.

结果 根据儿童缺血缺氧性脑病后遗症的MRI影像特点，将其分为：(1)脑白质损伤为主：47例；①脑室周围脑白质脱髓鞘、软化：23例；②基底节豆状核、视丘神经元选择性损害：9例；③胼胝体受损变薄：15例；(2)脑灰质损害为主：18例；(3)局部或多处脑软化：9例；(4)广泛脑损害：5例。

OBJECTIVE: To observe dynamic changes in flowing quality and field in cerebral ischemic area in rats so as to qualitatively and quantitatively analyze the microvascular state in pia matral encephali ischemic area and the changes of associated leukocytic flowing velocity and flowing morphology.

目的：动态观察针刺后大鼠脑缺血区流质、流场的变化，定性、定量地分析软脑膜缺血区微血管的状态及其所伴随的血细胞的流速及流态变化。设计：随机对照实验。

Outcome:(1) Acute ischemic damage may induce the expression of c-fos protein in Cerebral Cortex quickly. Also, the expression increases by the ischemia time delay. It may hint that ischemia injury can rise the ability of nerve cells to tolerate ischemia and anoxia. And the expression level of c-fos can be up-regulated by putting blood of 12 hand Jing Points. So we think that treated by this therapy can rise the irritability of brain to resist the development of ischemia and enhance the ability to plerosis.(2) Acute ischemic damage may induce the expression of HspVO in Cerebral Cortex quickly. Also, the expression increases by the ischemia time delay. It may hint that ischemia injury can rise the ability of nerve cells to tolerate ischemia and anoxia.

三 结果(1)急性缺血性脑损伤可快速诱导c-fos蛋白在皮层缺血区脑组织的表达，其表达有随缺血时间延长而逐渐增多的趋势，提示脑的缺血性损伤可应激性地增强神经细胞对缺血、缺氧的耐受和适应能力，而应用井穴放血的干预治疗可明显上调缺血区皮层脑组织c-fos的表达水平，进一步增强脑组织对抗缺血性损伤的应激能力，抵抗缺血性脑损害的进一步发展。

86 hypertensive patients with ischemic stroke . 85 hypertensive patients without ischemic stroke and 90 healthy blood donors were randomly enrolled in this trial . p-fibrinogen gene -455G/A polymorphy was analyzed from all subjects by polymerase chain reaction with restrictive enzyme HaeIII(PCR-RFLP method).

随机抽取住院高血压脑梗死病人86例，高血压非脑梗死病人85例，健康老年人90例，β-纤维蛋白原基因启动子区域-455G／A的多态性分析用多聚酶链反应加HaeⅢ内切酶检测。

Objective To use an ischemical reperfusion injury rat model by pulsinelli-4-VO andobserve the changes of the expression of Glu, N-methy-D-asparagic acidreceptor subunits NR_1 and NR_(2B) and ability of learning and memory after procerebrum ischemic reperfusion injury, and approach the pathogenesy of learning andmemory disorder induced by ischemic reperfusion injury, and the intervention ofginsenoside Rg_2 on the learning and memory disorder after procerebrumischemic reperfusion injury.

目的利用pulsinelli四血管法体建立脑缺血／再灌注损伤模型，观察前脑缺血损伤后谷氨酸、N-甲基-D-天门冬氨酸受体亚单位NR_1、NR_(2B)表达的变化及学习记忆的改变，从兴奋性氨基酸的兴奋毒作用尤其是其受体的表达方面探讨脑缺血／再灌注损伤引起的学习记忆功能障碍的发生机制，并观察人参皂苷Rg_2对脑缺血缺氧损伤引起的学习记忆障碍的干预机制。

Batroxobin can decrease PAF level in plasm after cerebral ischemic reperfusion and may have some effects on PAF-R mRNA expression in semidark band cortex after cerebral ischemic reperfusion, which provides experimental data for prophylactic intervention.

巴曲酶可降低脑缺血再灌注后血浆中PAF水平，并且可能对脑缺血再灌注缺血半暗带皮质组织PAF-RmRNA表达有影响，以期为预防性干预提供试验数据。

Objective To study the MR findings and its relationship with clinical manifestions of cerebral palsy caused by hypoxic-ischemic brain damage.The pathological mechanisms of MR findings were also explored.Methods The MR findings of14patients with cerebral palsy caused by hypoxic-ischemic damage were studied retrospectively and correlated with clinical findings.All those patients underwent MRI.Cerebral cortex and subcortical white matter,deep white matter,basal regions and thalami,and ventricles and subarachnoid space were observed on T1WI and T2WI.

目的 通过分析14例缺氧缺血性脑损伤所致脑瘫的MR成像特点及临床表现，讨论了该病的MRI表现与临床表现的相关性，以及MRI表现的病理损伤机制方法对14例缺氧缺血性脑损伤所致脑瘫患儿行MR检查，在T1WI及T2WI上对脑皮层及皮层下白质、深部白质、基底节、丘脑、以及脑室和蛛网膜下腔等部位进行观察。

CNP is a endothelium independent vasodilating factor of coronary artery. Because it has a certain prevention on ischemic myocardium, CNP may has a potential therapeutic value to ischemic heart disease.

CNP是冠脉的一个非内皮依赖性舒张因子，其对缺血心肌具有一定的保护作用，因而对缺血性心脏病有潜在的治疗价值。

These results suggest that nimodipine in addition to its cerebral vasodilating effect may protect brain from ischemic neuronal damage through modifying iNOS expression in the infarct area, and imply that 〓 may be involved in the iNOS expression in ischemic brain tissue which contributes to the recruitment of penumbra into infarction after transient focal cerebral ischemia.

上述结果表明，除了扩张脑血管外，尼莫地平可能还通过抑制iNOS在缺血性脑组织的表达阻止NO过量产生而起到脑缺血损伤的保护作用。鉴于尼莫地平是典型的钙拮抗剂，本结果还提示〓可能涉及缺血脑组织的iNOS表达，从而介入半暗区向梗死区转变的病理过程。

I didn't watch TV last night, because it .

昨晚我没有看电视，因为电视机坏了。

Since this year, in a lot of villages of Beijing, TV of elevator liquid crystal was removed.

今年以来，在北京的很多小区里，电梯液晶电视被撤了下来。