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ischemic相关的网络例句

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与 ischemic 相关的网络例句 [注:此内容来源于网络,仅供参考]

There was an obvious increase of insulin receptor gene and protein in the cerebral ischemia reperfusion tissues in rats. The positive cells such as neuron, neuroglia cells, vascular endothelium cells possibly participated in regulating the processes of energy metabolism of cells and neovascularization. It implied that the high expressions of insulin receptors in vascular endothelium cells in ischemic zones possibly participated in regulating the pathophysiological changes of the blood vessels in ischemic zones; The higher expressions of insulin receptors in different cells were possibly one of the self-protective machanisms in which the ischemic tissues confront the anoxemia injury.

脑缺血再灌注大鼠脑组织胰岛素受体基因和蛋白水平表达增加;半暗带区神经元、部分神经胶质细胞及血管内皮细胞等阳染细胞可能参与脑缺血后组织细胞的能量代谢、促新生细胞形成等病理生理调节过程;胰岛素受体在缺血区血管内皮细胞高表达提示其可能在缺血区血管的病理生理变化中起某种调节作用;胰岛素受体在不同细胞中表达增高可能是缺血脑组织对抗缺血缺氧损伤的自身保护机制之一。

Objective: To study the effect of elective cephalic hypothermia for treatment on newborn hypoxic ischemic encephalopathy.

目的:研究选择性头部亚低温治疗新生儿缺氧缺血性脑病( hypoxic ischemic encephalopathy,HIE)的疗效及安全性。

Department of Physiology, Institute of Basic Medicine,Hebei Medical University, Shijiazhuang 050017Abstract: The effects of ischemia-reperfusion and ischemic preconditioning on hemodynamics, epicardial electrography, myocardial infarct size, cardiomyocytic apoptosis and gene proteins involving apoptosis (Fas, Bcl-2 and Bax) were observed in aneasthetized rabbit myocardium.

在麻醉家兔心肌缺血-再灌注(ischemia-reperfusion,IR)模型上,观察IR和缺血预处理(ischemic pre~con~ditioning, IP)对血流动力学、心外膜电图、心肌梗塞范围、心肌细胞凋亡和凋亡相关调控基因蛋白(Fas、 Bcl-2、 Bax等)的影响。

Since the findings of Murry and Currie et al . that ischemic preconditioning and heat shock response could protect evidently myocardium against ischemia-reperfusion injury in the middle of 1980s, endogenous myocardial protection has drawn widespread attentions.

摘 要:自上世纪80年代中期Murry及Currie等分别发现缺血预适应(ischemic preconditioning, IPC)与热休克反应(heat shock response, HSR)具有明显的心肌保护作用以来,有关心肌内源性保护作用的研究引起了学术界的广泛关注。

Effects of acidic fibroblast growth factor on normal and ischemic myocardium.

酸性的纤维原细胞生长因素在上的作用正常并且ischemic心肌层。

Both brain ischemic pre-conditioning and PGE1 could induce cerebral ischemic tolerance through increasing Bcl-2 expression and reducing Bax expression. The effect of PGE1 in inducing cerebral ischemic tolerance was better than that of brain ischemic pre-conditioning.

预缺血及PGE1预处理后,使Bax蛋白表达减少,Bcl-2蛋白表达增高能够诱导脑缺血耐受,且PGE1的作用优于预缺血。

Methods: 18 healthy new-zealand rabbits were randomly divided into 3 groups (6 for each group): ischemic reperfusion group,ischemic preconditioning group and ischemic preconditioning combined with pyrrolidine dithiocarbamata group (group IPC+PDTC).

选择健康新西兰大白兔18只,随机分为缺血再灌注组(ischemic reperfusion, IR),缺血预处理组(ischemic preconditioning ,IPC)及缺血预处理联合PDTC组,每组6只。

INTERVENTIONS: 4 vessel occlusion(4VO) brain ischemic models in rats stained with thionine staining and GFAP immunohistochemistry staining. were used. Sixty-four rats, of which bilateral vertebral arteries were occluded permanently by electrocautery, were divided into the following 8groups: sham operation group, cerebral ischemic preconditioninggroup, ischemic insult group; BIT group; MTPG + sham operation group;MTPG+BIT group; MTPG+ischemia group and -4C3HPG+BIT coup. All the rats were killed 7 days after the operation or the final ischemic treatment. Cerebral sections were selected and stained with thionine staining and GFAP immunohistochemistry staining.

干预:采用大鼠四血管闭塞全脑缺血模型,应用硫堇染色和胶质纤维酸性蛋白免疫组化法。64只大鼠椎动脉凝闭后分为假手术组、单纯预处理组、单纯缺血组、脑缺血耐受组,MTPG+假手术组、MTPG+脑缺血耐受组、MTPG+缺血组和-4C3HPG+脑缺血耐受组,所有动物均在手术后或末次缺血后7 d处死取材,行脑组织切片,硫堇染色和胶质纤维酸性蛋白免疫组化染色。

AbstractAIM: To investigate the relationship between young eyes of central retinal artery and the changes in homodynamic and the occurrence of ischemic cerebrovascular disease.

目的:探讨中青年眼动脉(ophthalmic artery,OA)和视网膜中央动脉(central retinal artery,CRA)血流动力学的变化与发生缺血性脑血管病(ischemic cerebrovasctlar disease,ICD)的关系。

CTPI can evaluate the brain homodynamic of cerebral ischemia-reperfusion animal model in living state as a rapid, accurate, non-invasive method, show the cerebral infarction ischemic region's site, size, extent, and has important clinical value for the ultra-acute cerebral infarction to treatment and review.

第二部分CT灌注成像在超急性脑梗死实验中的诊断价值目的:应用CT灌注成像(CT perfusion imaging ,CTPI)技术,观察大鼠超急性脑梗死不同时间点脑血流动力学变化及半暗带(Ischemic penumbra ,IP)的演变,为临床早期诊断治疗提供依据和指导。

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