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ischemia相关的网络例句

查询词典 ischemia

与 ischemia 相关的网络例句 [注:此内容来源于网络,仅供参考]

Objective: To study the effects of Ischemia Preconditioning on the expression of HSP70 mRNA during ischemia reperfusion injury.

目的:探讨缺血预处理对心肌缺血再灌注过程中心肌细胞HSP70mRNA表达的影响。

AFGF shows greatpotential for clinical applications for therapy of a variety of diseases, such as Parkinson'sdisease, spinal cord contusion injury, neural regeneration in reimplantation of broken-offfinger, brain ischemia, renal ischemia, myocardial infarction, occlusive vascularitis, retinalischemia, gastric ulcer and nonhealing wound and so on. We expressed human acid fibroblast growth factor gene transiently in tobacco byAgro-bacterium mediated vacuum infiltration and studied the factors, which influenced theexpression level.

人类酸性成纤维细胞生长因子(aFGF,FGF-1)是成纤维细胞生长因子超家族成员,来自于三个胚层的多种细胞都可以表达aFGF.aFGF在治疗帕金森综合症、急性脊柱扭曲性损伤、断指中神经功能重建、脑缺血、肾缺血、心肌梗塞、闭塞性脉管炎、视网膜缺血、胃溃疡及难愈合性伤口等多种临床应用方面具有巨大潜力。

HISI treatment prolong the survival time of mice in toxic condition induced by sodium nitrite and duration of electrocardiograph in trachea closed mice .HISI could obviously improve the ischemic electrocardiogram T-wave induced by Pituitrin.ST segment drifts on electrocardiogram in acute myocardial ischemia rats induced by Isoprotereno were obviously opposed by administration of HISI. The myocardial infarction areas, releasement of lactic dehydrogenase 、 creatine phosphokinase significantly increased in myocardial ischemia dogs, HISI treatment prevented these effects .

实验结果表明:苦碟子氯化钠注射液能显著延长小鼠常压缺氧环境及亚硝酸中毒情况下的存活时间,延长小鼠气管夹闭后心电消失时间;可显著改善垂体后叶素诱发的大鼠急性心肌缺血心电图T波异常;明显对抗异丙肾上腺素诱导的急性心肌缺血大鼠心电图ST段的偏移;显著改善冠脉结扎所致急性心肌缺血犬的心外膜心电图缩小其梗死心肌面积,降低心肌耗氧量,减少心肌坏死导致的心肌酶如乳酸脱氢酶、肌酸磷酸激酶的漏出。

Both ischemia and ischemia/reperfusion injured the structures and functions of the membranes of sarcolemma and mitochondria.

两种损伤皆可导致大鼠心肌肌纤维膜及线粒体膜的结构和功能障碍。

The mechanism that acupuncture preconditioning induces cerebral ischemia tolerance and the reason of cerebral ischemia injuries have been studied from various angles and set up the theory of self-injurious mechanism and provide the theory foundation in preventing infarct, developing medicine and evaluating medical result.

关于针刺预处理后诱导脑缺血耐受机制及缺血性脑损伤的病因和发病机制,近年来,已从不同的角度进行了研究,并相应建立了自身破坏过程机制的假说,为防治脑梗死、开发新药和评价药物疗效提供了理论基础。

The recovery of blood circulation of ischemia semidarkness region, improvement of partly hypoxia state and prevention of more serious of impaired are key point in the stroke treatment. This experiment is designed to observe the blood vessel regeneration of ischemia injured region, and observe the variance of partly PCNA and VEGF expression. Combine with limbs functional recovery, we validate the therapeutical effect of acupuncture and investigate the possible mechanism of action.

尽早恢复缺血半暗区的血液循环,改善局部缺氧状态,防止损伤的进一步加重是脑卒中治疗过程中的重点,本实验从缺血损伤区血管再生为切入点,观察局部PCNA、VEGF表达的变化,结合肢体功能恢复情况,验证针刺的治疗作用,并探讨可能的作用机制。

Western blot was used to measure HSP70 of ischemia and non-ischemia myocardium and the expression of HSP70 was analyzed semiquantitatively.

采用蛋白质免疫印迹法检测各组缺血区和非缺血区心肌组织HSP70含量,并检测血清一氧化氯、丙二醛含量及总超氧化物歧化酶、肌酸激酶活性变化。

Methods: 2 hours of reversible focal ischemia in rats were produced by improved the method of Connolly' suture occlusion of MCA and the expression of SAMDC-mRNA in the cortex and subcortex was measured by RT-PCR after 2,4,8 and 24h reperfusion following 2h ischemia respectively.

在Connolly线栓法的基础上进行改良,复制大鼠MCA区2h脑缺血-再灌流2,4,8,24h动物模型,用逆转录多聚酶链反应测定缺血区皮质和皮质下组织不同时相SAMDC-mRNA的表达。

Brain ischemic preconditioning is that transient ischemia in sublethal dose will result in tolerance to the following ischemia in lethal dose for long time.

脑缺血预处理即给予短暂亚致死量缺血可对随后的长时间致死性缺血损伤产生耐受。

There were 613 breaks of ischemia symptomless in which 504 broke while at rest or sleeping, lasting for 3~5min and 88 breaks were symptomatic in which 53 broke while at rest and 25 broke in daily activities, lasting for 1~3 min and there were significant changes of ischemia on DCG and the ST segment descent for 0.1~0.3 mV.

结果:138例患者24h监测发现在所有ST段下移1mm以上的缺血发作701次,无症状发作613次,其中休息和睡眠时发作504次,持续时间为3~5min;有症状发作88次,其中休息和睡眠53次,日常活动时 25次,持续时间为 1~3 min,DCG检测中有明显的心肌缺血表现,ST段降低幅度0.1~0.3mV。

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