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ischemia相关的网络例句

查询词典 ischemia

与 ischemia 相关的网络例句 [注:此内容来源于网络,仅供参考]

The overexpression of HO-1 induced by CoPP can inhibit death of the myocardial cells and subsequently alleviate myocardial ischemia/reperfusion injury,whose major mechanism may be related with anti-oxygen free radicals.

CoPP诱导的HO-1过表达可以抑制心肌缺血再灌注损伤后的细胞坏死,从而减轻心肌的再灌注损伤,其主要机制与抗氧自由基有关。

The upregulation of HO1 expression induced by CoPP can inhibit the necrosis of the myocardial cells and subsequently alleviate the myocardial ischemia/reperfusion injury. One of its main mechanisms may be related with antioxygen free radicals.

CoPP诱导的HO1过表达可以抑制心肌缺血-再灌注损伤后的细胞坏死,从而减轻心肌的再灌注损伤,其主要机制与抗氧自由基有关。

Result HO-1 expression was upregulated following pretreatment of CoPP before reperfusion,resulting in both an increase in SOD activity and a decrease in MDA content and thus in a reduction in death of the myocardial cells after ischemia/reperfusion injury.

结果:再灌注前使用CoPP进行预处理,可以诱导HO-1蛋白的表达上调;HO-1蛋白表达上调可以减少缺血再灌注后的心肌细胞坏死,提高心肌组织中SOD含量并降低MDA的含量。

Objective To observe the effect of Cornu Cervi on the change of VEGF after the acute damage of myocardium in animal model of myocardial ischemia, and inquired into the mechanism of the protective effect on myocardium cell.

目的观察鹿茸提取物对心肌缺血动物模型心肌损伤急性期后血管内皮生长因子变化的的影响,并探讨其对心肌细胞保护作用的机制。

ABSTRACT Objective: To observe the protective effects of Corydalis ambailis migo total alkaloids on cerebral ischemia/reperfusion injury in rats and to investigate its mechanism.

摘要] 目的:观察夏天无总碱(Corydalis ambailis migo total alkaloids, COAMTA)对脑缺血/再灌注损伤的保护作用并对其机制作初步探讨。

KEY WORDS Corydalis decumbens; brain ischemia; neurologic manifestations; nitric oxide synthase; superoxide dismutase; malondialdehyde

关键词]夏天无;脑缺血;神经病学表现;一氧化氮合酶;超氧化物歧化酶;丙二醛

SHEN-FU Injection can accelerate the resume of myocardial function during ischemia-reperfusion, countermine arrhythmias after reperfusion, reduce the myocardial infarct size.

可以促进缺血再灌注心肌收缩和舒张功能的恢复,降低再灌注心律失常的发生率,缩小心肌梗死范围,减轻心肌组织的超微结构损伤性改变。

AIM To study apoptotic gene expression of the endothelial cells in cremaster ischemia-reperfusion of the rat.

目的 研究大白鼠提睾肌缺血再灌后血管内皮细胞的凋亡基因表达。

Ischemia-reperfusion of the cremaster could induce the apoptosis of the endothelial cells and might play an important role in microcirculation disorder.

大白鼠提睾肌缺血再灌注可引起血管内皮细胞凋亡,并可能在微循环障碍中起重要作用。

METHODS Expression of Bcl-2,Bax and P53in endothelial cells of cremaster ischemia-reperfusion of the rat was investi-gated by means of immunohistochemical SP method.

以免疫组织化学SP法对大白鼠提睾肌缺血再灌注后血管内皮细胞Bcl-2,Bax及p53的表达进行观察。

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