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insulin相关的网络例句

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与 insulin 相关的网络例句 [注:此内容来源于网络,仅供参考]

There was an obvious increase of insulin receptor gene and protein in the cerebral ischemia reperfusion tissues in rats. The positive cells such as neuron, neuroglia cells, vascular endothelium cells possibly participated in regulating the processes of energy metabolism of cells and neovascularization. It implied that the high expressions of insulin receptors in vascular endothelium cells in ischemic zones possibly participated in regulating the pathophysiological changes of the blood vessels in ischemic zones; The higher expressions of insulin receptors in different cells were possibly one of the self-protective machanisms in which the ischemic tissues confront the anoxemia injury.

脑缺血再灌注大鼠脑组织胰岛素受体基因和蛋白水平表达增加;半暗带区神经元、部分神经胶质细胞及血管内皮细胞等阳染细胞可能参与脑缺血后组织细胞的能量代谢、促新生细胞形成等病理生理调节过程;胰岛素受体在缺血区血管内皮细胞高表达提示其可能在缺血区血管的病理生理变化中起某种调节作用;胰岛素受体在不同细胞中表达增高可能是缺血脑组织对抗缺血缺氧损伤的自身保护机制之一。

Objective To observe the effects of berberine and taurine on PAI-1 mRNA expression in epiploon adipose tissue of high fat rats model of insulin resistance. and to study their effects on blood glucose, blood lipids, insulin levels, insulin sensitivity indexand body weight.

目的 观察黄连素、牛磺酸分别与联合应用对高脂饲料诱导的胰岛素抵抗大鼠脂肪组织 PAI-1 mRNA 表达的影响以及对大鼠空腹血糖、胰岛素、血脂、胰岛素敏感指数、大网膜脂肪细胞面积和体重的影响。

This heterogeneous phenotype (insulin resistance with respect to glucose metabolism and insulin sensitivity with respect to lipogenesis) seems to be a typical behavior of the liver able to explain simultaneously all the metabolic features of the insulin resistance syndrome.

肝脏的这种异质性表型(与葡萄糖代谢有关的胰岛素抵抗状态及与脂肪合成有关的胰岛素敏感状态)可能是胰岛素抵抗综合征所有代谢异常特点(葡萄糖代谢异常和脂肪代谢异常)的一种合理解释。

The insulin should be divided about 45 percent as NPH insulin, 30 percent before breakfast and 15 percent before bedtime, and about 55 percent as preprandial regular insulin, 22 percent before breakfast, 16.5 percent before lunch, and 16.5 percent before dinner.

胰岛素应该被分成45%低精蛋白锌胰岛素,其中30%早餐前,15%睡前,另55%为餐前普通胰岛素,22%早餐前,16.5%午餐前,16.5%晚餐前。

The insulin should be divided about 45 percent as nph insulin,30 percent before breakfast and 15 percent before bedtime,and about 55 percent as preprandial regular insulin,22 percent before breakfast,16.5 percent before lunch,and 16.5 percent before dinner.

上一篇:治疗糖尿病足的医院-什么样的水果有治疗糖尿病的疗效??下一篇:糖尿病足截肢|中国糖尿病治疗指南,糖尿病患者血糖应该降到多少

The insulin should be divided about 45 percent as NPH insulin, 30percent before breakfast and 15 percent before bedtime, and about55 percent as preprandial regular insulin, 22 percent beforebreakfast, 16.5 percent before lunch, and 16.5 percent beforedinner.

胰岛素应该被分成45%低精蛋白锌胰岛素,其中30%早餐前,15%睡前,另55%为餐前普通胰岛素,22%早餐前,16.5%午餐前,16.5%晚餐前。

Aims/hypothesis Ideally, a therapeutic insulin secretagogue should coordinately increase insulin production and insulin secretion to maintain islet beta cell secretory capacity.

目的/假设:理想情况下,治疗用的胰岛素促泌剂应该同时增加胰岛素的合成与分泌来保持胰岛β细胞分泌的能力。

Insulin secretin response to glucose challenge in vitro showed the mean value of insulin in the low-glucose medium was 39.74±2.73mM, while that of high-glucose medium was 128.94±8.72mM, the SI was 3.25±0.26, that means the islets functioned well. The releasing insulin levels of the nine groups responding to 16.7mM glucose at the end of 24h and 48h: At the end of the first 24h, the controlssecretion were similar in two phases, while the G group has increasing tendency;As for oleate groups, the base secretion have no difference,while the stimulated secretion was descending;As palmic acid as concerned,both base and sitimulated secretion was descending(p<0.01) accompany with the elevated glucose.

在培养48h后油酸组刺激后时相分泌量减少(p<0.05);软脂酸组两时向差别显著(p<0.01)各组细胞在16.7mM葡萄糖刺激5min时的胰岛素mRNA表达:在培养24h及48h后,葡萄糖对照组胰岛素mRNA表达组间无明显差别(p>0.05);油酸组培养24h胰岛素mRNA表达组间无明显差别(p>0.05),培养48h后mRNA表达组间比较存在统计学差异(p<0.05);软脂酸组培养24h时胰岛素mRNA表达量下降(p<0.05),培养48h后胰岛素mRNA表达量下降明显(p<0.01)。

Compared with these of fat-fed group, the body weight, blood glucose levels after oral glucose and the glucose area under curve, 40min and 90min blood glucose levels after insulin injection subcutaneously and the glucose area under curve in the insulin tolerance test, the fasting serum insulin, insulin resistance index, serum cholesterol, triglyceride and lower density lipoprotein cholesterol decreased, the fasting serum leptin, high density lipoprotein cholesterol and C18:2 were significantly increased in N-6 PUFAs group.

n-6脂肪酸组从第1~10周体重均较高脂组明显减轻,糖负荷后血糖和糖耐量试验中葡萄糖曲线下面积、皮下注射胰岛素后40min、90min血糖及胰岛素耐量试验中葡萄糖曲线下面积、血清胰岛素、胰岛素抵抗指数、血清胆固醇、甘油三酯和低密度脂蛋白含量均较高脂组明显降低;血清瘦素水平、高密度脂蛋白、18碳2烯酸较高脂组明显升高。

"Working with mice, we found that glucose levels were elevated and there was glucose production from a 'precursor', a source not normally metabolised."Other results in the study show that to some extent ARNT is regulated by insulin, so that insulin resistance in itself will contribute to a decrease in ARNT. If liver cells are treated with insulin, there will be a small increase in ARNT protein. The insulin will also help move the ARNT into the nucleus of the cell, where it does its job as a master regulator.

胰岛素绝对或相对不足时,肝脏内一种称之为 ARNT 的转录因子减少达90%,而 ARNT 与控制肝脏生产和释放葡萄糖的基因表达有关, ARNT 减少,肝脏生产和释放葡萄糖增加。2型糖尿病时,胰岛素相对不足不仅使肝脏内 ARNT 减少,也导致胰岛β细胞内 ARNT 减少,使得胰岛素生成减少和释放延迟。

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