查询词典 insulin-shock treatment

The peroxisome proliferator-activated receptors play an important role in adipocyte differentiation and fatty acid metabolism, activation of PPAR may induce the differentiation of adipocyte,influence the expression of genes involved in lipogenesis and fatty acid oxidation,eventually lessen ectopic fat accumulation and improve the impaired insulin signaling,so the medicine which targets to the control point of FFA metabolism would be hopeful in the treatment of a cluster of metabolic abnormalities resulted from insulin resistance.

过氧化物酶体增殖物激活受体在脂肪细胞分化、脂肪酸代谢中起重要作用，PPAR及其所调控基因的激活可促进脂肪细胞分化、减少脂质产生、增加脂肪酸氧化，从而减少脂质的异位沉积，进一步改善损伤的胰岛素信号转导通路，逆转胰岛素抵抗。针对脂肪酸合成及氧化的关键调控点的药物可能是今后治疗胰岛素抵抗引发的一系列代谢异常的新的靶点。关键词过氧化物酶体增殖物激活受体；脂肪酸代谢；胰岛素抵抗

By imitating physical secretion model of insulin, CS Ⅱ treatment has better and prompter effect on metabolic disorders and hyperglycemia.

CSⅡ模拟胰岛素的生理分泌模式，能更快、更有效地纠正代谢紊乱、控制高血糖。

Insulin or insulin secretagogue treatment of diabetes mellitus is the most common cause of hypoglycemia.

应用胰岛素和促胰岛素分泌治疗是引起低血糖的最常见原因。

In the drug-only group, those getting insulin sensitization treatment did slightly better than those who got insulin therapy.

药物治疗组中使用胰岛素增敏治疗的患者较接受胰岛素治疗的患者略好。

Through binding GLP-1 receptor , it can arouse insulin secretion, increase sensitivity of insulin, restrain glucagon secretion and slower the velocity of stomachic emptying. More recent studies showed that GLP-1 is effective for both type 1 and type 2 diabetes mellitus. Treatment of type 2 diabetes patients with GLP-1 does not lead to hypoglycemia.

此外，GLP-1通过激发其受体产生生理功能，能明显刺激胰岛素分泌、增加胰岛素敏感性、抑制胰升血糖素水平和减慢胃的排空，对2型糖尿病和1型糖尿病均有效，而且将其用于治疗不会产生低血糖。

Science treatment: Patients with diabetes are aware of this sentence, diabetes is not terrible, terrible is complications in some patients with drugs and the effects of insulin injections, blood glucose to normal, but complications lights up not mistaken, this is because the endocrine and metabolic disorder is a major cause of complications, Xiaoke special hypoglycemic contain trace elements necessary for the human body, the glucose tolerance factor in effective complement to eliminate insulin resistance, protection and restoration of pancreatic islet cells, a scientific and rational hypoglycemic and with the prevention and reversal The emergence of.

科学治疗：糖尿病患者都知道这样一句话，糖尿病不可怕，可怕的是并发症，有的患者在药物和注射胰岛素的作用下，血糖正常，但并发症照出不误，这是因为内分泌及代谢紊乱也是发生并发症一个重要原因，消渴特降糖含有人体所必需的微量元素，有效补充糖耐量因子含量，消除胰岛素抵抗，保护和修复胰岛细胞，科学合理降糖和预防及逆转并发症的出现。

METHODS:The techniques of RT- PCR and Northern Blot were adopted to test the gene expression of OB- R and the radioimmunoassay was used to test the serum a nd hypothalamus leptin and insulin in order to observe the changes of serum lept in, insulin and lipide in simple obesity patients and the changes of weight,Lee' s index,somatic fat,the contents of serum and hypothalamus leptin and insulin as well as the expression of hypothalamus OB- R gene in obesity rats before and a fter acupuncture treatment.

采用RT-PCR和NorthernBlot技术测定瘦素受体基因表达水平，放射免疫分析测定血清和下丘脑瘦素和胰岛素的含量。

METHODS: After all the right tibiae were broken, 30 μg/kg IGF-1 or 6-8 U insulin was used in the IGF-1 and insulin groups respectively. There was no treatment in the model control and normal control groups.

4组大鼠右胫骨上段骨折后，胰岛素样生长因子1组用胰岛素样生长因子1治疗，30μg/kg，胰岛素组用胰岛素治疗，6~8U，模型对照组及正常对照组未进行治疗。

Results Insulin was employed in 35.6% type 2 diabetic patients after failure of tablets treatment. Their psychological health was similar to that of non insulin treated patients. However, their life quality was significantly lower than the latter resulting from unsatisfaction with control of the illness, hypoglycemia anxiety and interference in daily life.

结果 2型糖尿病患者口服降糖药物治疗失效后使用胰岛素治疗者占35.6%，其心理健康情况与未使用胰岛素组相似，但生活质量显著差于后者，主要包括对病情控制不满意、担心低血糖事件和感觉日常生活受影响。

Results In 3T3-L1 adipocytes, isoproterenol at the concentration of 10 μmol/L reduced the adiponectin secretion and insulin at the concentration of 5 nmol/L could block the inhibitory effect of isoproterenol. In 313-Li adipocytes expressed myrAkt, adiponectin secretion decreased significantly, isoproerenol treatment increased adiponectin and insulin couldn't block the effect of isoproterenol.

结果 异丙肾上腺素可明显降低3T3-L1脂肪细胞脂联素的蛋白质表达，胰岛素可纠正这一变化；表达myrAkt的3T3-L1脂肪细胞中脂联素明显减少，异丙肾上腺素可增加脂联素蛋白的表达，胰岛素不能纠正这一变化。

The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

这种做法不仅鼓励了更多的反应，而且减少跟风的可能性。