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inflammatory相关的网络例句

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Results(1) Group of electroacupuncture in Shu and dense wave can significantlyreduce the degree of ankle swelling(P<0.05, P<0.01), which shows thatelectroacupuncture has obvious anti-inflammatory and repercussive roles;(2) Theresults of HE staining showed that group of electroacupuncture in Shuand dense wave can significantly reduce inflammatory cell infiltration,inhibit synovial cell proliferation, and decrease proliferation of granulationtissue in rats joints;(3) group of electroacupuncture in dense-wave can increase the pain thresholdof experimental rats (P<0.05, P<0.01), reduce the content of peripheral painmediums, such as K~+, DA and 5-HT(P<0.05, P<0.01), which shows thatelectroacupuncture has significant analgesic effect;(4) In the local lesion of joints, electroacupuncture can significantly reducethe content of IL-1β, IL-8, TNF-a, increase IL-4 content, decrease theexpression of COX-2mRNA, and significantly promote the expression ofHSP-70 protein in synovial tissues of acute gouty arthritis rats (P<0.05,P<0.01),thus restrain the occurrence of synovial tissue inflammation.

结果:(1)电针疏密波组能显著减轻踝关节肿胀度(P<0.05,P<0.01),表明电针有明显的抗炎消肿作用;(2)HE染色显示:电针疏密波组可使大鼠关节炎症细胞浸润明显减轻,滑膜细胞增生明显被抑制,肉芽组织增生明显减少;(3)电针密波组能提高实验性大鼠痛阈(P<0.05,P<0.01),降低外周疼痛介质K~+、DA、5-HT的含量(P<0.05,P<0.01),表明电针有明显的镇痛作用;(4)在病变关节局部,电针能显著降低急性痛风性关节炎大鼠滑膜组织IL-1β、IL-8、TNF-α含量(P<0.05,P<0.01),升高IL-4的含量(P<0.05,P<0.01),并降低关节滑膜内COX-2mRNA的表达(P<0.05,P<0.01),可促使急性痛风性关节炎大鼠滑膜内HSP-70蛋白表达显著增加(P<0.05,P<0.01),从而抑止滑膜组织炎症发生。

PTX at tenuates the ALI induced by LPS or sepsis in rat. The therapeutic effects of PTX include:improve oxygenation, at tenuate pulmonary edema formation, enhance animal survival in sepsis-induced ALI. The mechanisms may related with the effects of PTX inhibiting the chemotaxis, migration, activation of inflammatory leukocytes, diminishing the production of active oxygen, and inhibiting inflammatory cytokine release partly.

4PTX对大鼠内毒素性ALI及脓毒症性ALI均有一定的防治作用,主要表现在改善动脉血氧合,减轻肺水肿形成,提高脓毒症性ALI大鼠的存活率等方面,其机理可能与抑制白细胞趋化与激活,减少肺内白细胞渗出及活性氧产生,一定程度上抑制炎性细胞因子生成等有关。

RESULTS: During 10 - 12 weeks, in cuntrol group: The defect area was repaired by white and soft tissue that had no resistance to press. The repaired tissue was still lower than the surrounding articular surface with clear boundary. By histological observation, it was found that the defect was repaired by the mechanism similar to inflammatory reaction and the defect is ultimately filled by the hyperplasia of hyaline degenerative fibrous tissues. In filling group: the defect was repaired by semi-transparent, smooth, textured tissues with polish that had resistance to press as well as elasticity. The repaired tissue was almost similar to the shape of the surrounding cartilage,difficult to be distinguished. After histological observation, it was found that there was no inflammatory reaction, but active hyperplasia of inner bonetissue and cartilage tissues; a lot of osteoid tissues and trabeculation were found. Newlborn cartilage was fused with surrounding cartilage tissue and connected with surrounding tissues.

结果:10~12周,对照组:缺损区由白色、质软、按压无阻抗的组织修复,修复组织仍低于周围关节面,边界仍清晰可辨,组织学以类似炎症反应的机制修复缺损,最终以透明变性的纤维组织的增生来填补缺损部位;填充组:缺损区由半透明状、质韧光滑有光泽,按压有阻抗并有弹性的组织修复,修复组织与周围软骨外形上已基本相似,不易区分,组织学未见有炎症反应的过程,内骨组织和软骨组织增生活跃,并可见大量类骨组织和骨小梁形成,新生软骨和周围软骨组织融合,并与周围组织连接。

Background and Objictive:Crohns disease is a main type of inflammatory bowel disease,and is a segmental transmural inflammatory disease of gastrointestinal tract.

背景、目的克罗恩病是炎症性肠病的一个主要类型,是全胃肠道节段性全层炎症性病变。

Both usnic acid material and SMEDDS had certain antibacterials activity and good anti-inflammatory activity. The anti-inflammatory activity of SMEDDS is better than material.

松萝酸原料药和自微乳制剂均有一定抗菌作用,并显示出较强抗炎效应,其中制剂作用更为明显,抗炎效应明确。

However, higher concentration of CRP may increase the expression of protein and mRNA of inflammatory factor in endothelial cells; 2 Higher concentration of CRP may participate in the development of inflammatory process in endothelium by the mechanism that CRP induce cells apoptosis and activate signal pathway involved in cell proliferation. 3 CRP is the member of pentraxins family, in which two faces of pentamer form a special bi-phase structure: one face is ligand recognition phase; the other face is effector phase.

总结: 1CRP是一种保守蛋白,在正常生理水平下对内皮细胞没有明显的诱导炎症发生的作用,仅在高浓度条件下有可能诱导细胞炎症相关蛋白及mRNA的表达; 2高于生理水平的CRP对内皮细胞的致炎作用主要通过诱导细胞凋亡及激活细胞增殖相关蛋白信号通路的表达; 3CRP是一种双面五聚体蛋白,结构具有特殊的二相性,能够分别结合内皮细胞和E-LDL。

Abstract]ObjectiveTo study the diagnostic value of the color Doppler flow imaging for the inflammatory mass in epididymides. MethodsThe inflammatory mass of the epididymides in 126 patients were examined with CDFI.

摘要]目的探讨彩色多普勒血流显像(color Doppler folw imaging,CDFI)对附睾炎性结节的诊断价值。

Furthermore, the association of distinct inflammatory myopathies with certain breeds of dogs point to important genetic components of inflammatory myopathy that can now be studied using dogs as both parallel disorders and animal models.

此外, 这种关联同某些特定种类犬的明显炎性肌病指向了炎性肌病的重要的遗传组成,可以借此进行平行研究并可以作为疾病的动物模型。

This preliminary case control study reconfirms that TWHf is a very potent herbal remedy to suppress mononuclear expressions of many inflammatory cytokines and pro-inflammatory molecules.

尽管只有少数的研究对象,本篇研究提供了在免疫致病机转上的可能证据来支持雷公藤对於僵直性脊椎炎患者周边血液单核球细胞内的许多发炎性细胞激素及其它和发炎反应有关的分子的基因表现具有强烈的抑制作用的说法。

Thalidomide has been shown to selectively inhibit TNF-α, and this anti-inflammatory property was utilized in treating some TNF-αrelated diseases, like multiple myeloma, erythema nodosum leprosum, chronic graft-versus-host-disease, rheumatoid arthritis, and a variety of other inflammatory diseases and immunomodulation disorders.

沙利度胺(thaliodmide,Thal)又称反应停,能够选择性抑制TNF-α的水平,临床应用Thal治疗与TNF-α相关的多种疾病,如多发性骨髓瘤、麻风结节性红斑、慢性移植物抗宿主疾病、风湿性关节炎以及一些免疫调节紊乱和炎症有关的疾病,取得良好疗效,提示Thal有可能通过抑制TNF-α治疗人工关节无菌性松动。

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