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inflammatory相关的网络例句

查询词典 inflammatory

与 inflammatory 相关的网络例句 [注:此内容来源于网络,仅供参考]

Results The main histopathologic changes were:① experimental group: inflammatory cell infiltration (1W); inflammatory cell disappearance, fibroblast and capillary tachyauxesis, osteoid tissue and small amount neoformative bone tissue appearance (2 W); fibrous tissue decrease, neoformative bone tissue enlarging and maturity (1~3 M).

结果① 实验组:术后1周,骨缺损区周围有炎性细胞浸润,可见牙槽骨变性坏死;术后2周,炎性渗出明显吸收,成纤维细胞、毛细血管生长活跃,可见骨样组织和少量骨组织;术后1~3个月,新生骨组织范围扩大,钙化程度加强。

Control group: inflammatory cell infiltration (1W); inflammatory cell disappearance, fibroblast and capillary tachyauxesis (2W); osteoblast appearance around bone defect (1M); small amount neoformative bone tissue around bone defect.

对照组:术后1周,骨缺损区周围有大量炎性细胞浸润;术后2周,炎性渗出吸收,成纤维细胞、毛细血管生长活跃;术后1个月,骨缺损区周围有成骨细胞;术后3个月,骨缺损边缘有少量新骨沉积。

Control group: inflammatory cell infiltration (1W); inflammatory cell disappearance, fibroblast and capillary tachyauxesis (2W); osteoblast appearance around bone defect (1M); small amount neoformative bone tissue around bone defect. Conclusion TGF-β1 can induce alveolar bone regeneration.

对照组:术后1周,骨缺损区周围有大量炎性细胞浸润;术后2周,炎性渗出吸收,成纤维细胞、毛细血管生长活跃;术后1个月,骨缺损区周围有成骨细胞;术后3个月,骨缺损边缘有少量新骨沉积。

We previously demonstrated that intravenously administered arginine attenuates the inflammatory response, whereas the simultaneous administration of arginine and an NOS inhibitor, i.e., L-NMMA, might not improve hypermetabolic and inflammatory responses in peritonitic rats.

先前的实验证实,以静脉给予arginine可减缓腹膜炎大鼠的发炎反应,但同时给予arginine及NOS抑制剂未改善其高度代谢及发炎现象。

On the one hand, the inflammatory cells and inflammatory mediators phagocytize necrosis cells and impaired cell debris; on the other hand, they activate muscle satellite cells (locate among the basilar membrane and sarcolemma, and in dormancy state when quite) by secreting growth factor to proliferate to form new myotube cells which develop muscle fiber, repair impaired muscle fiber and accomplish muscle regenerative process.

炎症因子在损伤部位发挥着双重作用,一方面吞噬坏死细胞和损伤的细胞碎片,另一方面通过分泌生长因子激活肌卫星细胞(安静时处于休眠状态的,位于基底膜和肌膜之间),使其增殖形成新的肌管细胞进而发育成肌纤维,修复损伤的肌纤维,完成肌肉的再生过程。

Affect on the liver pathological state after XIEZHIPING treated: the golden hamster hepatic tissue morphous structure of normal control was normal, which had regular hepatic lobules and no lipid droplet;that of model control manifested diffuse heavy adipose degeneration and heavy ballooning degeneration with inflammatory cell infiltrate which mainly composed of lympholeukocyte;that of high dose group showed midrange ballooning degeneration with chronic inflammatory cell infiltrate which mainly composed of lympholeukocyte and plasmocyte;that of media dose group indicated slight focus ballooning degeneration with a small quantity normal hepatic cells in some locals;that of low dose group appeared midrange adipose degeneration with a few lympholeukocyte cell infiltrate in the parts of central veins and port area.

泻脂平治疗后对肝脏病理状况的影响:空白对照组金黄地鼠的肝组织形态结构正常,肝小叶规则,无脂滴发现;模型对照组肝脏弥漫性重度脂肪变性,发生重度气球样变,伴见以淋巴细胞为主的慢性炎性细胞浸润;泻脂平高剂量组肝细胞呈现中度气球样变,伴见以淋巴细胞、浆细胞为主的慢性炎性细胞浸润;中剂量组肝细胞轻度灶性气球样变,局部可见少量正常肝细胞;低剂量组肝细胞呈中度脂肪变性,可见中央静脉及汇管区些许淋巴细胞浸润。

Thus, in polyarthritic rat, a model of chronic inflammatory pain, we demonstrated that herpes simplex irus ector mediated oerexpression of proenkephalin A in primary sensory neurons at the lumbar leel elicited both antihyperalgesic and anti-inflammatory actiities.

在一个慢性炎症性疼痛模型-多关节炎鼠上,我们证明单纯疱疹病毒媒介介导的前脑啡肽在腰水平初级神经原的过度表达引起了抗痛觉高敏和抗炎症活动。

Result: Porosis and inflammatory cell infiltration in the spinal cord, rupture of muscle bundles, hypertrophia and over growth of muscle cells and collagens, and inflammatory cell infiltration in the bladder wall could been seen under HE dyeing. Nissal dyeing showed the decrease of neurons at the injured segment, upper and lower segments of the spinal cord. Result of immunohistochemistry showed there were caspase-3 positive expression in all the segments. The injured spinal cord and bladder expressed significantly more caspase-3 positive results (P.0l), upper and lower segments were lower than the injured segments (P.01), but higher than the control team (P.0l).

结果:HE染色发现脊髓损伤后脊髓内空洞形成、炎性细胞浸润等病理学变化,膀胱壁中可见到肌束断裂、肌细胞肥大增生、胶原增生、炎性细胞浸润等病理学改变;Nissal染色显示脊髓损伤后损伤节段及其上、下节段均出现神经元数量的减少;免疫组化结果表明3组均有不同程度的caspase-3表达,损伤组脊髓和膀胱内膜caspase-3表达较正常组显著增加(P.01),损伤上段及下段caspase-3表达较损伤节段为少(P.01),但均高于正常对照组(P.01)。

The body's inflammatory response to traumatic injury or infection is designed to protect it and return it to the preinjury state, but excessive inflammatory responses can seriously injure tissues and organs.

身体对于创伤或感染的炎症反应是为了保护身体,将其恢复到受伤前的状态,但过度的炎症反应会严重伤害组织和器官。

to use spinal lateral approach under the guidance of the c-arm x-ray machine, the needle position will be part of the disc, after successful percutaneous insert the plasma sword to melt herniated nucleus pulpous with the power, and then enter the moderate ozone to further expand the scope of ablation, and strengthen the inter-disk degradation and inhibition of the release of inflammatory mediators to eliminate protrusion of nerve roots caused by the oppression and inflammatory stimulation.

采用脊柱后外侧入路,在c型臂x线机引导下,将穿刺针定位于椎间盘部位,经皮穿刺成功后先插入等离子刀,通电后消融髓核使髓核皱缩,再打入适量臭氧,进一步扩大消融范围,加强降解间盘和抑制炎性介质的释放,来消除因间盘突出引起的对神经根的压迫和炎性刺激。

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