查询词典 infarction
- 与 infarction 相关的网络例句 [注:此内容来源于网络,仅供参考]
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Objective:To determine the risk factors for trigger effects of cerebral infarction, so as to supply basis for prevention of cerebral infarction paroxysm.
目的:探讨与脑梗死急性发作有关的触发危险因素,为有效防治突发脑梗死提供依据。
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Results The clinical manifestation of brain stem hemorrhage secondary to cerebral infarction included sudden onset,evelution abruptly,coma,quadriplegia,higher fever and respiratory failure. CT usually do not identify the high dense location of brain stem. Large infarction of cerebral hemisphere may lead to shift the brain laterally and downward to produce herniation with subsequent brain stem compression,shift and distortion. Secondary brain stem hemorrhage occured usually in midderline area of midbrain or pontine.
结果 脑梗塞继发脑干出血的临床特点是发病急、进展快、昏迷、四肢瘫、过高热及呼吸衰竭,CT一般不能发现脑干高密度病灶,病理结果均为半球大病灶脑梗塞,严重海马回疝致脑干严重受压、变形和移位,继发脑干出血以中脑为主,可累及桥脑,主要位于中线部位。
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Compared with CT or MRI,SPECT study can make out the location and size of infarction in the much earlier stage,and provide reliable evidence for the ultra-acute treatment of hemorrhage and infarction,or as MRI in the revealment of smaller lesion in and deep brain,so these three kinds of diagnoses method can not be substituted each other.
但对脑出血与脑梗死的鉴别不如CT对脑干及脑深部,较小病灶不如MRI显示清楚,因此三种检查方法不能互相取代。
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TGF-β1 in cortex infarction group was higher than that of subcortex group(P<0.05);the larger the infarction size was,the higher the TGF-β1 was(P<0.05);TGF-β1 in severe group was higher than that in mild group,but no statistical significance.
皮质梗死组TGF-β1高于皮质下组(P<0.05;梗死体积大则TGF-β1浓度高(P<0.05);重型组TGF-β1浓度高于轻型组,但无统计学意义。
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An association of ACE I/D polymorphism with myocardial infarction was found in a subpopulation younger than 60 years, which was strengthened in patients of myocardial infarction with obesity.
在研究的样本中,未发现ACE基因型与心梗相关;但在年龄60岁及以下的个体中,DD基因型与心梗显著相关,且这种相关在合并肥胖的个体中进一步加强。
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Therefore, it will of be of great clinical significance in preventing the transformation from TIA to cerebral infarction to intensify the study of the risk factor of high-risk TIA, to identify the risk factor for the transformation from TIA to cerebral infarction, and to make a individualized therapeutic regimen.
因此加强对高危TIA危险因素的研究,明确影响TIA发展至脑梗死的危险因素,有针对性的制定个体化治疗方案,对有效控制TIA向脑梗死转化有非常重要的临床意义。
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Patients had alien hand syndrome (11%), 17 patients had history of hypertention (54.83%) 16 patients had coronary artery heart disease (51.61%), 10 patients had diabetes mellitus (32.25%), 9 patients had history of cerebral infarction (29.03%), 17 patients had disorders of conciousness and mental (54.83%), 18 patients had language disorder (58.1%), 21 patients had disorders of limbs in different degrees (74.19%). MRI imaging of 22 patients showed previous cerebral infarction focuses (70.96%), 16 patients with leukoaraiosis (61%), 9 patients with angiostenosis (29%), stenosis was over 60% in 6 patients, and 7 patients showed cerebral microbleeding (22.58%). There were 34 focus of copus callusum infarctions in 31 patients.
异手综合征者3例(11%),高血压病史17例(54.83%),冠心病16例(51.61%),糖尿病10例(32.25%),脑梗死史者9例(29.03%),神志和精神异常的17例(54.83%),语言障碍的患者18例(58.1%),21例(74.19%)患者有不同程度肢体运动障碍。31例胼胝体梗死患者,共34处胼胝体梗死病灶,胼胝体梗死的患者合并其它部位陈旧性梗死灶22例(70.96%),伴有白质稀疏16例(61%),发现血管狭窄9例(29%,其中狭窄超过60%者6例),有7例(22.58%)伴有微出血。
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The results demonstrate that hypertensive cerebral arteriolosclerosis is the pathologic base which cerebral infarction is easy induced by hypotension, and thereis a close relationship between the occur of cerebral infarction and hypertensive degrees and duration in hypotension condition.
表明高血压性脑内小血管病变是低血压容易诱发脑梗塞的病理基础,说明低血压时,脑梗塞的产生与患者高血压程度的高低及持续时间的长短有密切的关系。
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Carotid atheromatous plaques is closely related to cerebral infarction.One important cause of cerebral infarction is from soft plaque broken in carotid atherosclerosis.
颈动脉粥样硬化与脑梗死的发生有密切关系,颈动脉粥样硬化的不稳定性斑块脱落是造成脑梗死的重要原因之一。
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Results Corpus callosal infarction was often coexisted with other area s infarction.
结果 单纯胼胝体梗死少见(8/78),多合并颅内其他部位梗死灶。
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