查询词典 infarct

Results: The permillage of apoptotic myocytes in infarct area increased at 1 h and achieved the peak at 4 h, and then decreased to ...

两种基因mRNA表达量的对数值之间呈负相关，心肌细胞凋亡数与Bac/Bcl 2基因mRNA比值呈正相关。

Results (1)The permillage of myocyte apoptosis in infarct area in elder patients with sudden death caused by AMI was significantly higher than that in control group(663.00‰±117.12‰ vs 34.30‰±20.68‰,P＜0 .01).However the permillage of myocyte apoptosis in 1-vessel,2-vessel,and 3 - vessel disease were 514.28±165.12‰,564.38‰±102.33‰ and 668.25‰±127. 19‰,respectiverly,significantly higher as compared to control group(All P＜ 0.01).But no significance was found among the three groups.(2)The size of DNA f ragment about 180～200 bp was found only in those patients with two and three ve ssels involoved.(3)The electron microscope showed the characteristics of myocyte apoptosis episodes,the others showed the characteristics of necrosis.

结果 TUNEL法发现，猝死者梗死区的心肌细胞凋亡千分数老年组(663.00‰±117.12‰)明显高于正常对照(34.30‰±20.68‰)(P＜0.001)；心肌细胞凋亡千分数在冠脉1支病变者(514.28‰±165.12‰)＜2支病变者(564.38‰±102.12‰)＜3支病变者(668.25‰±127.19‰)，虽然均明显高于正常对照(均P＜0.01)，但三组之间比较则无统计学上的差别(均P＞0.05)；冠脉2～3支病变者梗死区的心肌细胞DNA电泳可见相差约180～220 bp的阶梯片段；电镜发现猝死者梗死区内的心肌细胞核膜完整、染色质浓集、电子密度增加的凋亡特征，有的则出现核膜破裂、染色质溶解成碎屑的坏死现象。

Methods: The experimental myocardial infarction was induced by high level ligation of the anterior descending of the left coronary artery in rabbits. The protective effects were determined by the changes in ECG, the activity of serum creatin phosphorate kinase and infarct sizes.

采用结扎家兔冠状动脉前降支造成急性心肌梗塞的病理模型，用心电图、血清磷酸肌酸激酶活性和梗死面积评价TFGb对心肌缺血的保护作用。

At 6h after ischemia both MRI were found to indicate definite infarct foci in the caudate nucleus and the lentiform nucleus,mass effect was seen too.Histopathologic findings:At 2h after ischemia,electro microscope revealed the changes of ischemia and edema.At 4h after ischemia,the photomicroscope demonstrated the changes of ischemia edema and the electromicroscope indicated the damage to blood brain barrier.

结果 梗塞2h MRI T2加权可见尾状核头部、豆状核信号增高；6h MRI可见尾状核、豆状核形成明确梗塞，并出现占位征象。2h电镜下已有缺血水肿改变，4h光镜下出现脑缺血水肿改变，电镜下见到血脑屏障受损。

Methods The ischemic-reperfusion model of rat was used to comparing the effects of traditional pre-conditioning and preischemic NE-treatment.The incidence and duration of arrythmia,myocardial infarct size and myocardial enzyme were determined.

方法建立大鼠在体缺血再灌注模型，用去甲肾上腺素预处理，并与经典缺血预处理比较，观察心律失常发生率、心肌梗死范围和心肌酶的变化情况。

HRV is regarded as an independent and sensitive predicator of those patients who are at high risk of cardiac sudden death following myocardial infarct.

心率变异性（HeartRateVariability，HRV）分析可帮助判断多种心血管疾病预后，被认为是预测心梗后发生心性猝死危险性的独立而敏感的指标。

The microglial cells appeared the ramified and rod figuration in the early stages of cerebral ischemia and in the border zone of infarct.

采用侧脑室注射缝隙连接特异阻止剂甘珀酸，测定其对脑梗塞灶体积的影响及对即刻早期基因FOS和GFAP表达的影响，以了解缝隙连接在脑缺血损伤中的作用。

Methods assessment and comparison were made with mini-mental state examination and sandoz clinical assessment geriatric to 31 hospitalized patients with senile dementia (15 cases with alzheimer disease, ad, and 16 cases with multi-infarct dementia, md) before treatment and 4 weeks after treatment.results small dosage of olazapine could improve the psychotic symptoms of the patients with senile dementia; the total score of mmse after treatment increased obviously, and each factor score of scag showed out improvement.conclusions olazapine is effective in improving the psychotic symptoms of the patients with senile dementia.

对31例老年性痴呆（阿尔茨海默病15例和多发性脑梗死性痴呆16例）住院患者在治疗前、治疗后4周，采用简易智能状态检查及老年临床评定量表进行评定、比较。结果应用奥氮平治疗老年性痴呆患者的精神症状用量小，治疗后mmse总分明显增加。scag各项因子分明显改善。结论奥氮平治疗老年性痴呆的精神病性症状临床疗效肯定。

Brain-derived neurotrophic factor was shown to induce antiapoptotic mechanisms after stroke and to reduce infarct size and secondary neuronal cell death.

脑源性神经营养因子被显示了卒中后可诱导抗凋亡机制并减少梗塞面积以及继发神经细胞死亡。

The mechanism that acupuncture preconditioning induces cerebral ischemia tolerance and the reason of cerebral ischemia injuries have been studied from various angles and set up the theory of self-injurious mechanism and provide the theory foundation in preventing infarct, developing medicine and evaluating medical result.

关于针刺预处理后诱导脑缺血耐受机制及缺血性脑损伤的病因和发病机制，近年来，已从不同的角度进行了研究，并相应建立了自身破坏过程机制的假说，为防治脑梗死、开发新药和评价药物疗效提供了理论基础。

Neither the killing of Mr Zarqawi nor any breakthrough on the political front will stop the insurgency and the fratricidal murders in their tracks.

在对危险的南部地区访问时，他斥责什叶派民兵领导人对中央集权的挑衅行为。

In fact,I've got him on the satellite mobile right now.

实际上 我们已接通卫星可视电话了