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Under favorable condition, it multiplies as a unicellular organism. Upon starvation, a pathway involving aggregation, mound, slug, culmination stages induces the formation of a fruiting body consisting of a head of spores supported on a stalk of vacuolated cells.

在营养丰富条件下，以单细胞形式存在；一旦食物匮乏，单个细胞聚集成多细胞体，细胞历经细胞丘、蛞蝓体、拔顶阶段完成发育和分化过程，最终形成由孢子细胞和柄细胞组成的子实体。

Objective To investigate the mechanism by which the dorsal vagal complex ghrelin administration induces a hyperphagic response.

目的 探讨脑干迷走神经复合体区微量注射ghrelin对摄食的影响及其可能机制。

Variant angina, also called as vasospastic angina, expands very quickly, and induces miocardial infarction, so it is very important to cure this disease reasonably.

变异型心绞痛(variant angina； VA)也称血管痉挛性心绞痛(vasospastic angina； VSA)，病情发展迅速，容易发展成心肌梗塞，合理治疗十分重要。

The expression of GFAP in ILN was significant at 60 mm and it decreased obviously at 120 min after animal model induced. Conclusion Behavior changes in rats are mediated by visceralgia, which induces acute colitis. The change of GFAP in gliocyte, which is closely related with pain, emerged early and remarkably. The change of ILN, which is intensely related with emotion, appears lain.

大鼠痛行为学的变化是由于急性炎症引起的内脏痛而导致，与疼痛变化密切相关的胶质细胞中GFAP在脊髓内反应早且明显，而在与情绪变化密切相关的丘脑内的板内核群中变化相对较晚。

Natural torpor in this species is normally triggered by winterlike photoperiods and low ambient temperatures. Intracerebroventricular injection of neuropeptide Y reliably induces torporlike hypothermia that resembles natural torpor.

当在老鼠脑中注射NPY可以触发其在冬眠期间所产生的低体温，而这种低体温的现象也可以在脑中被注射NPY Y1受器的促进剂时被发现，可是在脑中被注射NPY Y5受器促进剂的老鼠身上则没有这种情形。

Background: Oesophageal acid infusion induces enhanced pain hypersensitiity in non-acid exposed upper oesophagus in patients with non-cardiac chest pain, thus suggesting central sensitisation contributes to isceral pain hypersensitiity in functional gut disorders.

背景：非心源性的胸痛患者，在非酸暴露的食管上段因食管酸灌注可导致疼痛的敏感性增加，这提示在功能性肠道紊乱中，中枢促感觉剂可导致内脏疼痛的过敏。

Background: Oesophageal acid infusion induces enhanced pain hypersensitiity in non-acid exposed upper oesophagus in patients with non-cardiac chest pain, thus suggesting central sensitisation contributes to isceral pain hypersensitiity in functional gut disorders.

背景：非心源性胸痛的患者，在无酸暴露的上段食管因食管酸灌注可诱导痛觉高敏感，这提示在功能性肠道机能紊乱中，中枢致敏可导致内脏痛觉高敏感。

This looks weak and induces further bluffs.

这看起来是示弱，能引诱对手接着诈唬。

Itamin D from the skin and diet is metabolized in the lier to 25-hydroxyitamin D (Figure 1), which is used to determine a patient's itamin D status1,2,3,4; 25-hydroxyitamin D is metabolized in the kidneys by the enzyme 25-hydroxyitamin D-1-hydroxylase (CYP27B1) to its actie form, 1,25-dihydroxyitamin D.1,2,3,4 The renal production of 1,25-dihydroxyitamin D is tightly regulated by plasma parathyroid hormone leels and serum calcium and phosphorus leels.1,2,3,4 Fibroblast growth factor 23, secreted from the bone, causes the sodium–phosphate cotransporter to be internalized by the cells of the kidney and small intestine and also suppresses 1,25-dihydroxyitamin D synthesis.5 The efficiency of the absorption of renal calcium and of intestinal calcium and phosphorus is increased in the presence of 1,25-dihydroxyitamin D (Figure 1).2,3,6 It also induces the expression of the enzyme 25-hydroxyitamin D-24-hydroxylase (CYP24), which catabolizes both 25-hydroxyitamin D and 1,25-dihydroxyitamin D into biologically inactie, water-soluble calcitroic acid.2,3,4

从皮肤和食物来的维生素D在肝中代谢为25-羟基维生素D（图1），被用来决定病人体内维生素D情况的1，2，3，4；25-羟基维生素D在肾中被25-羟基维生素D1羟化酶（CYP27B1）转变为有活性的1，25-二羟基维生素D 。1，2，3，4由肾产生1，25-二羟基维生素D是被血浆甲状旁腺激素和血清钙，磷水平紧密调节。1，2，3，4由骨分泌的成纤维细胞生长因子23使钠磷协同转运蛋白被肾和小肠细胞内化及抑制1，25-二羟维生素D合成。5 在1，25-二羟基维生素D作用下肾和小肠吸收钙及磷的效率增高(图1)。2，3，6 它也包括25-羟四- 24 -羟化酶的表达(CYP24)，且将1，25二羟基维生素D和25羟基维生素D异化成无生物活性，水溶性的维生素D3-23羧酸。2，3，4

One who coerces or induces another person, or instigates a person under the age of eighteen to violate the security administration

有较严重后果的；胁迫、诱骗他人或者教唆不满十八岁的人违反治安管理的

DL: A job? He's ten years old, and you let him go?!

一份工作？他才十岁，而你就让他被带走了？！

In the research, the dependent variable is compliment response strategy, and the independent variables are gender and age of the respondents, and social status of the interlocutors.

本研究所涉及的因变量为恭维语应答策略，自变量为恭维语应答者的社会地位，性别和年龄。