查询词典 induced current

60Co-γray was a useful mutagen in induced mutation breeding .After treated with 60Co-γray ,free-living conchocelis of wild Porphyra haitanensis were induced and represented in offsprings. Purple mutant and excellent practical characters strains were selected from a lot of gametophytic blades. Single cell clone and enzymolysis were used to propagate fast in pure strains of induced mutations.

坛紫菜野生丝状体经一定剂量60Co-γ射线辐照后，子代叶状体发生了变异，经大量培养后筛选出紫色突变体（3号Ⅰ）和经济性状优良的个体（7号Ⅰ和7号Ⅱ），通过体细胞酶解和单克隆技术快速获得诱变选育纯系，并对其主要经济性状进行了研究。

Microarray technique was applied to look into the gene expression profiles. Ox-LDL induced HAEC apoptosis by inducing many redox- and apoptosis-related genes. Sal B prevented the ox-LDL induced endothelial damage and even induced endothelial cell proliferation.

本计划亦探讨丹参酚酸 B 抑制氧化低密度脂蛋白所造成内皮细胞的损伤与细胞附著分子表现的作用机转，亦引入微矩列技术以探讨基因表现差异之谱型，氧化低密度脂蛋白诱导许多氧化还原与细胞凋亡相关基因，丹参酚酸 B 可保护内皮细胞损伤甚至促进其增生。

For ADSCs at passage 3, the formation of red l ipid droplets within cells was observed 2 weeks after being induced towards adipocyte, red calcium nodules were evident 3 weeks after being induced towards osteoblast, and highly congregated cell mass positive for alcian blue staining appeared 2 weeks after being induced towards chondrocyte.

第3 代ADSCs 成脂诱导2 周，可见细胞内有红色脂滴形成；成骨诱导3 周，可见红色钙结节样改变；成软骨诱导2 周，可见高度聚集生长的细胞团，阿利辛蓝染色呈蓝色。

Based on these results and inferred to related reports from other labratories, it was possible to make some analyses and conclusions or inferrences:(1) CD〓AK with tumoricidal activity were induced and expanded in number througth costimulation of PBMC with anti-CD〓 McAb . and r IL-2 ;(2) CD〓AK induced and expanded in such manner did exibit more potent proliferation·ability and cytotoxicity which maintained for lonser time than those of LAK cells, thus CD〓AK was a new variety of antitumor effector cells worth to be explored;(3) CD〓AK could mediate MHC nonrestricted cytotoxicity and kill tumor target cells through inducing necrosis and apoptosis;(4) Normal mature lymphocytes of PBMC could be induced to proliferate and /or to die from apoptosis when they were costimulated by anti-CD〓McAb and rIL-2. Both proliferation and apoptosis were existing in the same cultivation system sugsesting that the presence of rIL- 2 might provide some accessary signals for apoptosis.

以这些结果为基础并参考其它有关文献可能做出如下分析与结论或推论：(1)用抗CD〓单抗和rIL-2共刺激外周血单个核细胞能诱生扩增出具有杀瘤活性的CD〓AK细胞，(2)与LAK相比，用这种方法诱生扩增的CD〓AK增殖能力强、细胞毒活性强而且维持时间长；CD〓AK是一类值得开发的抗瘤效应细胞；(3)CD〓AK能够介导MHC非限制性细胞毒活性，可以通过诱导靶细胞坏死和/或凋亡杀伤肿瘤细胞；(4)正常外周血单个核细胞中的成熟淋巴细胞在受到抗CD〓单抗和rIL-2共同刺激后既可诱导增殖也可诱导凋亡，两者并存于同一体系，推测rIL-2的存在可能为细胞凋亡提供一些辅助信号。

DST showed that the pretreatment with dexamethasone can not repress the total number of CRF immunoreactive neurons in the rat model induced by CUMS, by contrast with a normal repression in the number of CRF neurons in rat induced by acute stress. In brief, the rat model induced by CUMS represented decreased body weight, obvious depression-like behavior, hyperactivity in HPA axis and nonsuppression on the DST, which fits the criteria and neurobiological feature of depression.

简而言之，CUMS诱导的大鼠模型有典型的抑郁症样行为和表现、HPA轴活性升高、DST的失抑制，符合抑郁症模型的行为学标准和主要神经生物学特征，是抑郁症稳定可靠的模型，也是研究应激诱导的糖皮质激素升高所带来的神经生物学效应的良好模型。

objective to search the factors of liver injury via the anit-induced liver injury in rats.methods male sprague-dawley rats,were divided into 4 groups,data were analyzed using a completely randmized factorial anova and linear correlation.results after anit intoxication,the necrosis of hepatic cell and neutrophil infiltration im periportal region of the liver were observed in liver setions.jinshisan can reduce the alt,the tbil.pla2 correlated positively with alt、akp and tbil.conclusion the severity of anit-induced liver injury correlated positively with the pla2.jinshisan can protect against the rat anit-induced liver injury.

目的 拟通过对α－萘异硫氰酸酯诱导大鼠肝损伤发病机理的探讨，对引起肝损伤的因素进行系统的研究。方法选择s-d大鼠，雄性，随机分4组，检测数据以平均值±标准差表示，采用方差分析，相关分析进行统计学处理。结果s-d大鼠经anit染毒后，病理切片显示肝损伤明显，汇管区有大量中性粒细胞浸润。经金石散及地塞米松治疗后，生化指标均有所下降，中药金石散优于地塞米松，血生化指标与pla2活性相关分析显示，呈正相关。结论 anit诱导的肝毒性损伤程度与肝组织匀浆pla2活力呈正相关，应用抑制剂降低pla2活力，肝损伤程度也减轻，中药金石散具有降低pla2活性，减轻肝毒性损伤。

On the normotensive rat, bilateral microinjection of L-arginine (16 n mol/0.1 ul/site), a precursor of nitric oxide into the rVLM, caused a decrease in BP, HR and the pressor response induced by the dPAG stimulation, while microinjection of NG-methyl-Larginine (L-NMMA, 16 n mol/0.1 ul/site), a blockade for NO to form, into the rVLM bilaterally, caused an increase in BP, HR and the pressor response induced by the dPAG stimulation. There was no effect of both drugs mentioned above on the tachycardiac response induced by the dPAG stimulation.

五、在正常血压大鼠，于双侧rVLM微量注射一氧化氮前体(16nmol/0.1μl/site)可引起BP降低、HR减慢、以及电刺激dPAG诱发的防御性升压反应减弱；而于同区微量注射生成一氧化氮的拮抗剂N〓-甲基-L-精氨酸(16nmol/0.1μl/site)则可引起BP升高、HR加快、以及电刺激dPAG诱发的防御性升压反应增强；而上述两药对电刺激dPAG诱发的防御性加快心率反应均无明显作用。

Motilin and gastrin induced contraction of isolated gastric smooth muscle cells in a dose-dependent manner.(2) Motilin- and gastrin-induced antral smooth muscle cell contraction was inhibited by antibodies against Gαi-3. Motilin and gastrin caused an increase of Guanosine-5′-(3-［35S］thio)triphosphate(［35S］GTPγS) binding of Gαi-3.(3) Contraction of antral muscle cells induced by motilin and gastrin was inhibited by the phospholipase C inhibitor U-73122 and the IP3 receptor antagonist heparin.

结果显示：(1)胃动素和胃泌素对胃窦平滑肌细胞均有收缩作用；(2) Gαi-3抗体可抑制胃动素和胃泌素加强胃窦平滑肌细胞的收缩，胃动素、胃泌素明显增加Gαi-3抗体与［35S］GTPγS的结合；(3)磷脂酶C抑制剂U-73122、三磷酸肌醇受体拮抗剂肝素可抑制胃动素和胃泌素引起的胃窦平滑肌细胞的收缩。

Further, we found that phenytoin couldn't inhibit the LPS-induced IkappaB-alpha degradation suggesting that this drug might not affect LPS-induced activation of NF-kappaB by abrogating the LPS-induced IkappaB-alpha?ndegradation.

在机转方面，我们证实phenytoin并不能有效抑制经LPS活化所造成ΙκB-α的降解，因此，我们推论phenytoin的抑制作用可能不是经由NF-kappaB这条路径。

We approach the problem based on the perception that electric field induced microstructure evolution at the crack tip will lead to fracture and fatigue. It follows that a micrograph of intensified electric field induced domain switching at the crack tip is obtained, a theoretical model of domain switching induced fracture is proposed, and the failure mechanisms of electric fracture, electric fatigue and crack kinking are revealed.

论文从电场驱动的裂尖微结构演化导致电致失效这一基本思路出发，获得了裂尖集中电场诱发铁电畴变的实验图像，提出了电致畴变断裂的理论模型，阐明了铁电陶瓷电致断裂，疲劳裂纹扩展和裂纹偏折的失效机理。

Now she was hungry and angry.She began to smoulder.

现在她又饿又气，她开始流露难以抑制的怒火。

You have placed our iniquities before You, Our secret sins in the light of Your presence.

诗90：8 你将我们的罪孽摆在你面前、将我们的隐恶摆在你面光之中。